Sentences with phrase «cholesterol particles in»

Soluble fiber can reduce both «bad» LDL and overall cholesterol, perhaps by binding with cholesterol particles in your digestive system and moving them out of the body before they're absorbed.

We run the NMR lipid test which looks at the size and number of the cholesterol particles in your blood, rather than just the amount of cholesterol.

Not exact matches

(5) Research has shown that in most cases eating up to 3 eggs per day can actually improve your good cholesterol (HDL) and your bad cholesterol (LDL) will stay the same or change from being small dense LDL particles to large LDL particles.

To mimic this versatile substance, Joke Bouwstra and Robert Rissman at Leiden University in the Netherlands mixed a range of fatty compounds including lanolin, fatty acids, ceramides and cholesterol with particles made of a water - storing hydrogel (International Journal of Pharmaceutics, DOI: 10.1016 / j.ijpharm.2009.01.013).

In individuals with high HDL - cholesterol levels in the circulation, the lipid composition of the HDL particle was more beneficial regarding heart disease risIn individuals with high HDL - cholesterol levels in the circulation, the lipid composition of the HDL particle was more beneficial regarding heart disease risin the circulation, the lipid composition of the HDL particle was more beneficial regarding heart disease risk.

In individuals, whose HDL - cholesterol levels were low, the quality of HDL particles was also impaired; they contained smaller amounts of lipid molecules which are known to be antioxidant and thus protective to arteries.

Another area focuses on the HDL particle (carrier of good cholesterol in the blood).

Now a team of scientists, led by Mary Jo LaDu in the department of pathology at the University of Chicago Medical Center, has shown that the apoE - containing fat transporters found in brain cells are very different from the apoE particles found elsewhere in the body and contain most of their cholesterol in a different form.

In the blood, fats and cholesterol are packaged, with key protein components, into particles called lipoproteins.

The differences between the lipoproteins secreted by astrocytes and those found in the CSF suggest that the astrocytes produce particles that help the brain rid itself of excess cholesterol in addition to helping deliver membrane components to the nerve cells.

I saw «The Last Heart Attack» and I was wondering what you thought about what Dr. Arthur Agatston said in the show, as he's the inventor of the South Beach Diet, I'm wondering how credible his idea that the size of cholesterol particles and your cholesterol number doesn't matter.

It also transports cholesterol from extra-hepatic sites, including the arterial wall, to the liver for excretion via reverse cholesterol transport.77 Results from IER (60 - 85 % ER / alternate days) trials have thus far been inconsistent, with some reporting decreases 40, and others increases 37, 39, but with the majority showing no effects on HDL cholesterol levels.38, 42, 43, 45, 49, 51, 52Inconsistencies may have arisen due to the biphasic response of HDL documented by studies of ER induced weight - loss, whereby levels typically decrease during active weight - loss then either return to baseline or (less commonly) rise following attainment of weight stability.78 Complementary increases in HDL levels and particle size have been shown when endurance exercise have been combined with IER 42, 46, 47 however further discussion goes beyond the scope of this review.

So, for those you listening in, just to kinda bring this full circle here briefly, high cortisol, in the case of high cortisol, one of the things that Dr. Bryan recommended was to look at things like your cholesterol particles and your HDL, to look at things like your testosterone, to look at things like the ACTH that we talked about, and some of these other variables that can affect ACTH, and then of course, something like an oxidative stress panel.

In multiple human studies, the low - fat diet has actually made some important risk factors worse, raising triglycerides, lowering HDL (the good) cholesterol and making the LDL particles smaller (10, 11, 12, 13).

It seems fairly clear that those on keto and very - low - carb diets who get most of their energy from fat — including their own fat stores — tend to have more LDL particles circulating in their bloodstream, delivering triglycerides to cells and transferring cholesterol to other lipoproteins.

Increased enterohepatic circulation on high fat means that cholesterol is kept «in play» - bounced back into the bloodstream in ApoB particles - while low enterohepatic circulation, in people with with higher synthesis rates, during weight loss - when cholesterol is being dumped by shrinking cells - means that cholesterol can pile up in the gall bladder faster than it can be conjugated to bile salts and bile acids and faster than it can be extracted by the weak stimulus of low fat food.

In a 2010 study published in the American Journal of Clinical Nutrition, the researchers found that when you replace saturated fat with a higher carbohydrate intake, you increase insulin resistance and obesity, and increase your chances of getting heart disease by increasing triglycerides and small LDL particles, and reducing beneficial HDL cholesteroIn a 2010 study published in the American Journal of Clinical Nutrition, the researchers found that when you replace saturated fat with a higher carbohydrate intake, you increase insulin resistance and obesity, and increase your chances of getting heart disease by increasing triglycerides and small LDL particles, and reducing beneficial HDL cholesteroin the American Journal of Clinical Nutrition, the researchers found that when you replace saturated fat with a higher carbohydrate intake, you increase insulin resistance and obesity, and increase your chances of getting heart disease by increasing triglycerides and small LDL particles, and reducing beneficial HDL cholesterol.

We are just starting to understand how the body carries cholesterol to and from cells in the form of LDL, VDL, and HDL particles.

The type of cholesterol (LDL particle size) is the more important factor in regards to cholesterol consumption.

They can increase LDL cholesterol and in particular the small dense LDL particles that damage our arteries.

LDL or «bad cholesterol» measures the amount of cholesterol in the LDL particles but not the number or size of the particles.

Increased glycated ApoE particles have been detected in the cerebrospinal fluid (CSF) of AD patients.34, 35 The physiological insult of glycated ApoE is that ApoE helps transport LDL particles (and their critical cholesterol and fatty acid passengers) across the blood brain barrier.

Yet the reduction in LDL cholesterol from reducing saturated fat intake seems to be specific to large, buoyant (type A) LDL particles, when in factit is the small, dense (type B) particles (responsive to carbohydrate intake) that are implicated in cardiovascular disease.Indeed, recent prospective cohort studies have not supported any significant association between saturated fat intake and cardiovascular risk Instead, saturated fat has been found to be protective.»

Vegetable oils oxidize when heated, and when oxidized cholesterol and trans fat enter into your LDL particles, they become destructive, contributing to arterial plaque buildup in your brain

Glucose / HBA1C Free - ranging glucose molecules in your bloodstream can adhere to cholesterol particles and cause those particles to remain in the bloodstream for long periods of time, since your liver can't properly process cholesterol when it has a glucose molecule attached to it.

In fact, the diet should be approximately 70 % of calories from unadulturated fats like low carb nuts (pecans and macadamias are great, almonds ok and peanuts and cashews are considered higher carb on the nut scale), avocado, grass fed butter, coconut oil, olive oil; and the remainng 15/15 for protein and non-starchy vegetable carbs, especially nutrient dense leafy greens It is carbohydrates or high protein leading to gluconeogenesis in the diet that make concurrent consumption of fats a cardiovascular risk, but in a properly carb - restricted and moderate protein diet, and in the absence of systemic inflammation (hsCRP, ESR), one should not worry about increases in cholesterol, but focus on the size of the cholesterol particles (bigger is better) Dr. Peter Attia explains this complex topic welIn fact, the diet should be approximately 70 % of calories from unadulturated fats like low carb nuts (pecans and macadamias are great, almonds ok and peanuts and cashews are considered higher carb on the nut scale), avocado, grass fed butter, coconut oil, olive oil; and the remainng 15/15 for protein and non-starchy vegetable carbs, especially nutrient dense leafy greens It is carbohydrates or high protein leading to gluconeogenesis in the diet that make concurrent consumption of fats a cardiovascular risk, but in a properly carb - restricted and moderate protein diet, and in the absence of systemic inflammation (hsCRP, ESR), one should not worry about increases in cholesterol, but focus on the size of the cholesterol particles (bigger is better) Dr. Peter Attia explains this complex topic welin the diet that make concurrent consumption of fats a cardiovascular risk, but in a properly carb - restricted and moderate protein diet, and in the absence of systemic inflammation (hsCRP, ESR), one should not worry about increases in cholesterol, but focus on the size of the cholesterol particles (bigger is better) Dr. Peter Attia explains this complex topic welin a properly carb - restricted and moderate protein diet, and in the absence of systemic inflammation (hsCRP, ESR), one should not worry about increases in cholesterol, but focus on the size of the cholesterol particles (bigger is better) Dr. Peter Attia explains this complex topic welin the absence of systemic inflammation (hsCRP, ESR), one should not worry about increases in cholesterol, but focus on the size of the cholesterol particles (bigger is better) Dr. Peter Attia explains this complex topic welin cholesterol, but focus on the size of the cholesterol particles (bigger is better) Dr. Peter Attia explains this complex topic well.

As reported by The New York Times: 8 «The problem, [Dr. Kummerow] says, is not LDL, the «bad cholesterol»... What matters is whether the cholesterol and fat residing in those LDL particles have been oxidized...

Siri Tarino stated, «However, replacement of saturated fat by carbohydrates, particularly refined carbohydrates and added sugars, increases levels of triglyceride and small LDL particles and reduces high - density lipoprotein cholesterol, effects that are of particular concern in the context of the increased prevalence of obesity and insulin resistance.

What matters is whether the cholesterol and fat residing in those LDL particles have been oxidized... «Cholesterol has nothing to do with heart disease, except if it's oxidized,» Dr. Kummerow said... [He] contends that the high temperatures used in commercial frying cause inherently unstable polyunsaturated oils to oxidize, and that these oxidized fatty acids become a destructive part of LDLcholesterol and fat residing in those LDL particles have been oxidized... «Cholesterol has nothing to do with heart disease, except if it's oxidized,» Dr. Kummerow said... [He] contends that the high temperatures used in commercial frying cause inherently unstable polyunsaturated oils to oxidize, and that these oxidized fatty acids become a destructive part of LDLCholesterol has nothing to do with heart disease, except if it's oxidized,» Dr. Kummerow said... [He] contends that the high temperatures used in commercial frying cause inherently unstable polyunsaturated oils to oxidize, and that these oxidized fatty acids become a destructive part of LDL particles.

This is important because cholesterol particles can be found in bile acid.

We are now learning that its carbs, high processed carbs that are in reality causing high trigs, which also lead to small dense cholesterol particles (the kind that penetrate artery walls and cause unstable plaques).

Without VLDL particles, fats and cholesterol are trapped in the liver and never reach the blood and adipose cells.

Studies have linked them to improvements in LDL cholesterol and particle levels, lower blood pressure, and reduction of inflammatory markers (23, 24, 25, 26, 27).

Furthermore, positive associations between TSH and LDL as well as total cholesterol levels have been found in cross-sectional studies in euthyroid healthy subjects, and the strength of these associations seems to depend on an individual's insulin sensitivity.We therefore hypothesize that the KD has diminished the production of T3 from T4, thereby reducing the number of LDL receptors and thus reducing LDL particle clearance which might be further impaired due to the missing stimulating effect of insulin on LDL uptake into cells.

According to research found in Atherosclerosis, Thrombosis, and Vascular Biology, large LDL particles were not found to be associated with an increased risk of ischemic heart disease in men, and that the cardiovascular risk LDL cholesterol does pose is related to levels of its small particles.

What we've learned since then, when it comes to cholesterol health and heart disease risk, is that the relative size and number of LDL particles in the bloodstream matter.

As they circulate in the bloodstream they release little particles of fat and cholesterol that are utilized by the cells or stored as body fat.

DHA supplementation led to changes in triglycerides, apoB and LDL - cholesterol, which may suggest an increase in LDL particle size (a good change).

The results from this trial clearly indicate that the replacement of refined carbohydrates with EPRO and UFA increased insulin sensitivity, LDL peak particle size, and lowered fasting TG and VLDL cholesterol concentrations in men and women with an elevated TG concentrations.

Some say its a numbers game, the more LDL particles crashing the walls of the arteries... some say the LDL is only a sign of vascular damage, but if my LDL goes down on a low fat diet, then that is bs, some say that it may be a problem metabolizing LDL, intake / production is outpacing LDL receptor activity, some say its not LDL but LDL that stays in the blood too long and oxidizes, some say about 20 % carbs (I was less than 5 %) will produce just enough insulin to help metabolize cholesterol, but the hard core low carb guys, say the whole cholesterol thing is a scam and cholesterol under 500, without insulin resistance is nothing to worry about.

Moreover, all LDL cholesterol is not harmful — only tiny LDL particles, which increase in those with carbohydrate resistance.

, a particle in your blood that carries cholesterol, fats and proteins.

1) A genetic Condition called Familial Hypercholesterolemia which affects 1 in 500 people, where a person's liver expresses much less LDL receptors and is therefore unable to clear «used» cholesterol in LDL particles from the blood

But at the same time, cholesterol may go up on a ketogenic diet but if it does, I see the HTL go up in conjunction wih the LDL so the ratio is not getting worst and the trigs will also go down and the particles side shifts so your LDL A would go up, B would go down and your LDL will also improve too.

Another significant contributor is the high carbohydrate, lowfat diet, which leads to excess glucose in the blood stream, which glycates LDL particles and renders them ineffective in delivering cholesterol to the tissues.

The study also reported improvements in other cardiovascular and metabolic variables such as triglycerides, LDL - cholesterol particle size, and C - reactive protein, but it did not correct for multiple comparisons (a common scientific design problem of many fasting studies)(36).

After seven weeks, both groups had a big reduction in heart disease risk (reduced LDL cholesterol, triglycerides and increases in LDL particle size — good).

Both diets also lowered levels of harmful LDL («bad» cholesterol) and triglycerides, the most abundant fat - carrying particle in the bloodstream.

In some cases it did raise LDL cholesterol levels but this was a transition from smaller to bigger, more effective, LDL particles which is a good thing.

Not only does it help with cholesterol but L. reuteri 30242 has been shown to safely support healthy CRP (a marker for inflammation), fibrinogen (involved in clot formation), apoB - 100 (a marker for LDL particle size, a known cardiovascular risk factor), and vitamin D levels (important for cardiovascular health) for those within normal range.3, 4,5