«Second, greater anterior
cingulate cortical thickness could be the result of both the continuous coping efforts and emotion regulation attempts of MDD and SAD patients.»
Not exact matches
Structural MRI revealed disease - related reductions in
cortical thickness in the same regions: the inferior frontal cortex, anterior
cingulate cortex, and superior frontal gyrus.
It is still unclear exactly what the relationship is between the clinical manifestations of MDD and SAD and
cortical thickening in brain regions like the anterior
cingulate cortex, a part of the brain associated with emotion, Dr. Zhao said.
In this work, we show that chronic treatment with L. rhamnosus induced region - dependent alterations in GABA (B1b) mRNA in the brain with increases in
cortical regions (
cingulate and prelimbic) and concomitant reductions in expression in the hippocampus, amygdala, and locus coeruleus, in comparison with control - fed mice.
The relationship between
cortical surface area and cognitive control was found in the right caudal anterior
cingulate, and post hoc analyses showed a similar but weaker relationship in the opposite hemisphere.
We hypothesized that whole - brain and region - of - interest analyses would identify differences in
cortical thickness in prefrontal (especially anterior
cingulate, middle frontal gyrus and orbitofrontal cortex) brain regions in female adolescents with early onset SUD, compared to controls.
This finding also yields support from a previous study of adults and the elderly, where executive control was related to anterior
cingulate thickness, and it was suggested that
cortical maturation could be the driving causal factor (52).
Two recent studies of normal children found that parent - reported hyperactivity, impulsivity (11), and conduct problems (32) were related to
cortical thickness in several areas, including the anterior
cingulate.