As such, atherogenic and inflammatory risk may arise through a combination of dietary lipoprotein patterns and an increase in
circulating endotoxin, exacerbated by feeding patterns (26,27).
«Because the human gut is host to 100 trillion commensal organisms, which together contribute to an enteric reservoir of 1 g LPS (8), we hypothesized that most of
the circulating endotoxin may derive from the gut and that a small amount of commensally derived] LPS maycotransit with dietary fat from the gut after a high - fat meal, which thereby increases plasma endotoxin concentrations postprandially»
A friend sent me a study suggesting that
circulating endotoxin levels spike in diabetics who have eaten a high fat meal (I post this piece of science at the end of this message).
In fact, the five meal per day protocol has even led to an increased level of systemic inflammation and
circulating endotoxins.
Not exact matches
GWI exposures alter the microbiome (i.e., bacterial content in the gut), and the affected microbiota then produce
endotoxins, which pass through a thinned lining of the gut (i.e., leaky gut) and into the blood where they
circulate throughout the body.
Further research showed that a bacterial toxin called
endotoxin was
circulating in the bloodstream following this meal, causing inflammation and injury in the heart and lungs.
Interestingly, the low - grade inflammation correlates with
circulating levels of a plasma
endotoxin known as lipopolysaccharide (LPS).