Emotional sounds, such as crying and laughter also had a similar pattern of activity, with an area near the primary auditory
cortex lighting up in dogs and humans.
As a subject imagined hearing words, his auditory
cortex lit up the screen in a characteristic pattern of reds and greens.
And their medial prefrontal
cortex lit up, particularly at images of overweight women.
When he asked them to read braille with their reading fingers, their visual
cortex lit up.
Dr. Berns found the dogs»
cortex lit up when their owner's worn clothing was placed near their nose, proof that they remembered the owner even when that person wasn't present.
Not exact matches
Just around the time that the nature of the dilemma crossed the border from impersonal to personal, I would see your amygdala and related brain circuits — your medial orbitofrontal
cortex, for example —
light up like a pinball machine.
When the mouse activated a specific neuron, the one chosen for neuroprosthetic control, we simultaneously applied stimulation proportional to this activity to the sensory
cortex using blue
light.»
Indeed, neurons of the sensory
cortex were rendered photosensitive to this
light, allowing them to be activated by a series of optical flashes and thus integrate the artificial sensory feedback signal.
Focusing on the neural pathway from the brain's prefrontal
cortex to the amygdala, they combined optogenetics — a technique that uses
light to control the activity of neurons in living tissue — with behavioral testing, a methodology that allows researchers to study functional connections between different regions of the brain.
At the same time, transcranial magnetic stimulation was used to excite the brain's visual
cortex, priming the volunteers to see illusory spots of
light called phosphenes.
The findings shed new
light on the role of the mammalian
cortex in orchestrating eye movement, according to Scanziani.
Genetically programmed molecules guide embryonic nerve fibers from the
light - sensitive retina toward the visual
cortex, where images are perceived.
The best microscopes currently available can detect
light from 3 to 4 millimetres into the brain, enough to see
light signals coming from the
cortex of a small animal, but not enough to see deep - seated structures such as the hippocampus.
In one of his team's first experiments, a
light - sensing gene coded to «turn on» the right side of the motor
cortex was put into mice.
I don't know, but in
light of what we found in the motor
cortex, it's a very interesting quotation.
But Amedi and his colleagues found that the area of the visual
cortex responsible for recognizing body shapes in sighted people — called the extrastriate body area —
lit up with activity in the study participants when they were interpreting the human silhouettes.
Intriguingly, the new research could help explain how our own early - developing retinal fovea, which contains the densest concentration of
light receptors and produces the sharpest vision, claims so much
cortex.
To do this, they inserted genes into neurons of the prefrontal
cortex that would turn the neurons on when exposed to
light.
When anorexic and bulimic women see images of overweight women, an area of the brain, the medial prefrontal
cortex,
lights up in a functional MRI.
«We might compare these regions of the prefrontal
cortex with a traffic
light,» says Stefanie Hardung.
For this research brain activity is also being recorded using a technique called functional near infrared spectroscopy (fNIRS), which uses
light to measure changes in blood concentration in the prefrontal
cortex, it is a non-invasive and highly portable technique meaning it can be easily interchanged between car and simulator.
Scientists have been studying how visual space is mapped in the cerebral
cortex for many decades under the assumption that the map is equal for
lights and darks.
As a consequence of the greater spatial mapping of darks, a 0.5 x 0.5 mm cube of visual
cortex can represent the same position of a dark spot but different positions of
light spots that appear to rotate around a dark anchor in visual space.
The rhesus monkeys were trained to move their hands to a
light cue on a touch screen (for example from the center of the screen to the left), while at the same time the activity of neurons in their posterior parietal
cortex was recorded.
The team found significant changes in gene expression after
light exposure in all cell types in the visual
cortex — both neurons and, unexpectedly, nonneuronal cells such as astrocytes, macrophages and muscle cells that line blood vessels in the brain.
Into the cerebral
cortex of mice with these
light - sensitive proteins, the team implanted cancer cells from a human pediatric cortical glioblastoma.
Using novel technologies developed at HMS, the team looked at how a single sensory experience affects gene expression in the brain by analyzing more than 114,000 individual cells in the mouse visual
cortex before and after exposure to
light.
To investigate just how this change affects an animal's sense of novelty, Burwell and her colleagues infected brain cells in rats» perirhinal
cortex with a virus containing a
light - activated channel.
They then inserted a fiber - optic probe into the prefrontal
cortex to record the flashes of
light.
Researchers from the MRC CDN, led by Professor Oscar Marín, have shed
light on this problem by discovering that some neurons in the cerebral
cortex can adapt their properties in response to changes in network activity — such as those observed during learning of a motor task.
Unlike the visual
cortex, say, whose coding will be influenced by
light falling onto the retina, the entorhinal
cortex creates the hexagonal pattern entirely internally, by integrating whatever information about the environment is received by other areas of the brain.
Artificial retinas,
light - sensitive chips that mimic the eye's signal - processing ability and stimulate the optical nerve or visual
cortex, have been tested in a handful of blind subjects who usually «see» nothing more than phosphenes, or flashes of
light.
The
light is processed by an area of the brain called the occipital
cortex.
A recent generation of studies of postmortem brain tissue from people with schizophrenia, particularly from the laboratory of Professor David Lewis and his colleagues at the University of Pittsburgh, have shed
light on schizophrenia - related abnormalities in the interplay of the main excitatory neurons, pyramidal neurons, and a specific class of inhibitory nerve cells, called chandelier cells, in the prefrontal
cortex.
Some of the most riveting sections are those that deal with the activity of the brain during thinking or remembering, with different areas of the
cortex «
lighting up» to carry out the job.
Someone whose striate
cortex (primary visual
cortex) has been damaged or destroyed can sometimes identify the position and even the shape of
light stimuli without the aid of any conscious visual experience.
They sacrificed some of the engineered mice and isolated a slice of the
light - sensitive somatosensory
cortex, which remained viable for several hours.
The possibility of such cells has been debated at least since the 1950s, when researchers found single neurons in the visual
cortex of cats and other animals that respond to simple stimuli, such as lines oriented at a certain angle or moving in a specific direction, or
light of a particular wavelength.
Bao's team then partnered with Stanford colleagues, led by Karl Deisseroth, to genetically engineer somatosensory
cortex tissue of mice to absorb blue
light and fire in response.
Parts of the orbital frontal
cortex, which is implicated in decision making, also
light up.
Karl Deisseroth, who pioneered optogenetics with Boyden, gave an early demonstration of its power when he flashed a blue
light on a mouse's brain — the right motor
cortex to be precise — and found that the animal ran in circles, anticlockwise.
The medial prefrontal
cortex, associated with self - awareness and emotional regulation, quickly
lit up in angry brooders.
In this way, they were able to use pulses of
light on mice to stimulate theta - brainwave - like activity around the anterior cingulate
cortex.
The researchers tagged engram cells in the
cortex and then activated them with
light, causing the mice to freeze in environments in which they had never been shocked.
These manipulations, belonging to optogenetics — a technique extensively studied in Yizhar's lab — enabled the researchers to activate only those amygdala neurons that interact with the
cortex, and then to map out the cortical neurons that receive input from these
light - sensitive neurons.
He found that the premotor
cortex — a brain region that integrates vision and touch —
lights up, signaling the source of the illusion.
But only a «yes» answer generates a response intense enough to stimulate the visual
cortex and cause the inquirer to see a flash of
light known as a «phosphene.»
(Left) An unlabeled phase - contrast microscope transmitted -
light image of rat
cortex — the center image from the z - stack (vertical stack) of unlabeled images.
The research that was carried out on the mice showed a dramatic reduction of amyloid protein in the visual
cortices of the animals after just one hour of being under the
light.
In the late 1970s, Shatz followed up on the Nobel Prize - winning work of Hubel and Wiesel showing that soon after birth, in monkeys and cats,
light stimulation of the eye promotes the self - organisation of the visual
cortex — the part of the brain responsible for vision.