Chemical genetics unveils a key role of mitochondrial dynamics,
cytochrome c release and IP3R activity in muscular dystrophy.
Not exact matches
More of both support cell suicide as now sick or injured cells signal mitochondria to
release factors such as
cytochrome c into the cell cytoplasm.
Release of
cytochrome c, Bax migration, Bid cleavage, and activation of caspases 2, 3, 6, 7, 8, and 9 during endothelial cell apoptosis.
Kostmann syndrome: severe congenital neutropenia associated with defective expression of Bcl - 2, constitutive mitochondrial
release of
cytochrome c, and excessive apoptosis of myeloid progenitor cells.