Photodynamic therapy (PDT), involves the use of chemicals called photosensitizers that are activated by exposure to specific wavelengths of light to release reactive molecules that can
damage nearby cells.
They found that even the briefest of pulses in the nanosecond to femtosecond range produced a microscopic explosion called a cavitation bubble powerful enough to
damage nearby cells.
Not exact matches
However, implanting them can further
damage hearing, and electrical currents can spread easily through the neural tissue to stimulate
nearby cells, which the patient hears as noise.
In a bid to progress beyond the shotgun approach to fighting cancer — blasting malignant
cells with toxic chemicals or radiation, which kills surrounding healthy
cells in the process — researchers at the Harvard - MIT Division of Health Sciences and Technology (HST) are using nanotechnology to develop seek - and - destroy models to zero in on and dismantle tumors without
damaging nearby normal tissue.
Arising from the Schwann
cells of the vestibular (balance) nerve, these tumors can grow to the point of
damaging nearby structures — and can lead to death by compressing the brainstem.
Once inside, researchers found that the antibodies zeroed in on their targets — viral antigens — and the radioisotopes destroyed
nearby cancer
cells without
damaging surrounding healthy tissue.
These
cells are now thought to serve as the immune system's principal sentinels in the skin — when they detect
damage signals from
nearby wounded skin
cells, they summon other, non-skin-resident immune
cells to the site of the wound.
«Both the immune system and
nearby healthy
cells send signals instructing
damaged and unhealthy
cells to undergo apoptosis, which is like programmed «
cell suicide».
When they are activated, they release those compounds and sometimes cause collateral
damage to
nearby cells.
This paradigm of harnessing
cells in
nearby areas of
damage to replenish lost tissue is not just applicable to the heart; it may be useful in many other organs in the body that have support
cells like fibroblasts.
As days pass, M2s essentially become ineffective, and the enduring inflammation prompted by their counterparts begins to
damage healthy or slightly injured
cells nearby, which is believed to increase potential deficits caused by the TBI.