The oxygen - containing molecules the body uses to produce energy can be highly reactive and can inadvertently
cause damage to the mitochondria and even the cells themselves.
The scientists revealed that these very commonly used antibiotics caused
profound damage to the mitochondria, and this led to a dramatic increase in the production of damaging chemicals called free radicals.
Often, the best way to reduce mitochondrial dysfunction is to take preventative steps to reduce exposure to free radicals, which may cause
further damage to the mitochondria.
Research has shown that either alpha - lipoic acid or acetyl - L - carnitine can help to
reduce damage to mitochondria and improve normal blood vessel tone, allowing blood vessels to relax.
Any damage to mitochondria could compromise the smooth running of the cell.
As Miria Ricchetti, joint last author of the paper and a scientist at the Institut Pasteur, explains, «
the damage to mitochondria caused by H. pylori bacteria is temporary and disappears once the infection has been eliminated.
Using cutting - edge research techniques, the Martin laboratory, funded by Science Foundation Ireland, found that
damage to mitochondria (which function as «cellular battery packs») activates the Parkin protein, which results in one of two different outcomes — either self - destruction or a repair mode.
And it's suspected that many common medications, like acetaminophen and antibiotics, can cause
damage to the mitochondria.
These researchers determined,
damage to your mitochondria may be RESPONSIBLE for muscle atrophy, weakness and functional impairment.
Unless we have enough antioxidants in our diet or we make enough in our body, we can't protect ourselves from
the damage to our mitochondria.