LPS binding to Toll - like receptor 4 (TLR4) causes activation of NF - κB that requires two downstream pathways, each of which when isolated exhibits
damped oscillatory behavior.
The activation dynamics of the transcription factor NF - κB exhibit
damped oscillatory behavior when cells are stimulated by tumor necrosis factor — α (TNFα) but stable behavior when stimulated by lipopolysaccharide (LPS).
Not exact matches
A few months after my sawtooth model of the AMO (i.e. mid-2013) I concluded that the mechanism I suggested in column 4 of that poster, namely seismic interference at the core - mantle boundary, CMB, would be too strongly
damped to yield any sort of reliably
oscillatory behavior, and my 2013 talk at AGU FM makes no mention of any oscillation except the 20 - year magnetic Hale cycle, which can be seen clearly even in CET back to the end of the Maunder minimum.
The consequence is that in the natural world all
oscillatory behavior is
damped, and without an external driver the oscillations would die out — it is unlikely that these processes began in 1880, so why are they still there without forcing?