When sugar levels increase in the blood, the pancreas secretes insulin to
decrease glucose production from the liver to maintain balance.
Not exact matches
Some of these hormones have other characteristics, such as anti-insulin properties, that
decrease your body's sensitivity to insulin, increase
glucose production, and can cause diabetes.»
Disturbances in maternal
glucose metabolism, such as increased insulin resistance or
decreased insulin
production, may be a key factor in the observed relations between older maternal age, larger birth weight, obesity, and delayed OL.
The scientists found that both the activity of the genes, as well as the
production of the cholera toxin itself were increased when the bacterium was fed with
glucose, but they were considerably
decreased when it was fed with starch from rice.
The action of TZDs on adipose tissue macrophages may
decrease local
production and concentrations of proinflammatory factors and thereby contribute to their physiologically beneficial effects on
glucose and lipid metabolism.
min, in 4 AL, 18 AL, and 18 CR, respectively (P < 0.01 between all groups), and in 18 CR rats infused with insulin at similar rates as in the 18 AL (1.4 mU / kg / min) hepatic
glucose production was
decreased by 32 % (P < 0.
When the adrenal gland produces adrenaline and the adrenaline (beta - adrenergic) receptor communicates with the G - protein and its signal cascade, the parts of the body are alerted to the need for action; the heart beats faster, the blood flow to the gut
decreases while the blood flow to the muscles increases and the
production of
glucose is stimulated.
Now, when your brain's
production of insulin
decreases, your brain literally begins to starve, as it's deprived of the
glucose - converted energy it needs to function normally.This is what happens to Alzheimer's patients — portions of their brain start to atrophy, or starve, leading to impaired functioning and eventual loss of memory, speech, movement, and personality.
These include insoluble extracellular plaques made of beta - amyloid peptide (Aβ); intracellular neurofibrillary tangles (NFTs) resulting from the hyperphosphorylation of tau (a microtubule - associated protein); loss of hippocampal neurons; a
decrease in
production of brain acetylcholine; and a marked decline in
glucose usage in regions of the brain associated with memory and learning.5,11,20 - 22 All of these changes can be logically explained as the sequelae resulting from long - term dysregulation of insulin signaling and
glucose metabolism.
«During ketosis, there is a
decrease in the
production and use of
glucose and as a consequence in the breakdown of proteins (found in muscles) are used as energy,» says the nutritionist of celebrities such as Jessica Alba: Kelly Leveque about this diet.
(Insulin's job is the help process
glucose out of the bloodstream, and
decrease the liver's
glucose production.)
the primary mechanism for maternal adaptation to short - term fasting is
decreased maternal
glucose use by the early development of ketosis and fatty acidemia, thus maintaining a
glucose supply for milk
production and a constant milk supply for the infant.
It may also help us
decrease our body's
production of
glucose and help us increase our body's ability to store starch in the form of glycogen.
Metformin works to
decrease your body's
production of
glucose.
Those with PCOS may find relief by utilizing this medical aid due to its ability to improve
glucose metabolization,
decrease insulin
production, and regulate testosterone.
In vitro, IF pancreatic islets had increased insulin secretion,
glucose metabolism and net reactive oxygen species
production, while
decreased their mass.
Ketosis occurs when insulin and blood
glucose levels
decrease to an extent that allows for increased fat oxidation — which is followed by ketone
production.
Allick et al concluded that it was remarkable that, in the context of diabetes risk, 2 aspects of
glucose homeostasis actually improved after consumption of the high - fat, low - carbohydrate diet: basal endogenous
glucose production decreased, and insulin - stimulated nonoxidative
glucose disposal increased.
At the end of the study the researchers found that weight, liver fat content, C - peptide levels (an indicator of insulin
production), and fasting plasma
glucose had
decreased in both groups, however to a greater extent in the group that ate the two - meal plan.
Inadequate amounts of insulin cause improper metabolism of both
glucose and fats, leading to
decreases in energy
production and, too often, diabetes.
For example, KBs were recently reported to act as neuroprotective agents by raising ATP levels and reducing the
production of reactive oxygen species in neurological tissues, 80 together with increased mitochondrial biogenesis, which may help to enhance the regulation of synaptic function.80 Moreover, the increased synthesis of polyunsaturated fatty acids stimulated by a KD may have a role in the regulation of neuronal membrane excitability: it has been demonstrated, for example, that polyunsaturated fatty acids modulate the excitability of neurons by blocking voltage-gated sodium channels.81 Another possibility is that by reducing
glucose metabolism, ketogenic diets may activate anticonvulsant mechanisms, as has been reported in a rat model.82 In addition, caloric restriction per se has been suggested to exert neuroprotective effects, including improved mitochondrial function,
decreased oxidative stress and apoptosis, and inhibition of proinflammatory mediators, such as the cytokines tumour necrosis factor - α and interleukins.83 Although promising data have been collected (see below), at the present time the real clinical benefits of ketogenic diets in most neurological diseases remain largely speculative and uncertain, with the significant exception of its use in the treatment of convulsion diseases.
This study shows that green tea
decrease fat
production, boost fat breakdowns, and increase the utilization of
glucose.
As this occurs there is a
decrease in
glucose production and utilization.
Daily intake of 20 g of fructooligosaccharides significantly
decreased basal hepatic
glucose production (Luo et al., 1996).
In Type I diabetes,
glucose concentrations are high because of a
decrease in the
production of insulin.