Sentences with phrase «depression at baseline»

Preschoolers with depression at baseline were more likely to have later depression rather than other psychiatric disorders.

Parental Bonding Inventory (PBI) scores at baseline were investigated as predictors of depression on the Edinburgh Postnatal Depression Scale (EPDS) at 4, 14 and 21 months after childbirth in mothers without depression at baseline.

This indicates that participants with higher levels of depression at baseline who received the intervention showed larger reductions in depression symptoms over the course of the study.

The clinic group had more indicators of severe depression at baseline than the community group (see http://www.ebmentalhealth.com/supplemental for table).

The findings reported herein suggest that remission of maternal depression over 3 months is statistically significantly associated with reduction in children's current symptoms and diagnoses after controlling for the child's age and sex, baseline symptoms, socioeconomic status (annual household income), as well as severity of maternal depression at baseline, mother's treatment setting, and the child's treatment status over the 3 - month follow - up.

In the clinical interview, 27 % of the group were found to have depression at baseline.

In our analysis, the association of sugar intake and recurrent depression was attenuated by measures of body fatness in participants without doctor diagnosis of depression at baseline supporting the hypothesis of an indirect effect mediated by adiposity26, 27,28 driving the association of sugar intake and recurrent depression.

In sensitivity analyses, main analyses were repeated by: (a) excluding participants with unknown or reported doctor diagnosis of depression at each baseline (at phases 3 / 5/7 / 9: 166 / 156/193 / 209 individuals) and: (b) excluding participants with extreme values of sugar intake (> 7 SD) at phases 3 / 5/7 / 9: 5 / 3/4 / 4 individuals.

Excluding participants who reported a doctor diagnosis of depression at each baseline strengthened the association (Model 4 for CMD after 5 years, Person observations = 10944; highest vs. lowest tertile OR; 1.25; 95 % CI 1.03, 1.50; P for trend = 0.02, Supplementary Table S2) and exclusion of person observations with extremely high sugar intakes did not affect the results.

In a study of 1002 cases and controls followed up for 12 years, those with gut disorders had elevated levels of anxiety and depression at baseline, but also those with higher levels of anxiety and depression were more likely to have gut disorders at follow - up (17).

Not exact matches

At baseline, patients age 80 and older had a higher prevalence of hypertension, heart disease, osteoporosis and joint problems, but a lower BMI, and a lower prevalence of depression and smoking.

A growing number of human studies suggest that a low plasma Aβ42 / Aβ40 ratio is a risk factor for major depression, 45, 46 dementia47 and higher mortality.48 The Framingham Study also showed that increased plasma Aβ42: Aβ40 ratios are associated with decreased risk of AD and dementia.13 Thus, the higher Aβ42 / Aβ40 ratio observed in regular meditators at baseline and the increase in this ratio from pre - to post intervention in the novice meditator and vacation groups may be salutary to brain health.

Various biological factors at baseline appear to predict response to rTMS, including levels of certain molecular factors, blood flow in brain regions implicated in depression, electrophysiological findings, and specific genetic polymorphisms.

The data came from roughly 70,000 women, none of whom suffered from depression at the study's start, who had baseline measurements taken between 1994 and 1998, and then again after a three - year follow - up.

Sensitivity analyses excluding extreme values of sugar intake and excluding person - observations with self - reported doctor diagnosis at baseline attenuated the association of sugar intake from sweet food / beverages and recurrent depression slightly (before P for tertile trend 0.003 after 0.022 and 0.010, respectively).

Further adjustments for central obesity and physical health (not shown), exclusion of 709 person - observations (377 in CES - D analysis) with reported doctor diagnosis of depression and person - observations with extreme values of sugar intake at baseline did not change the results.

About 44 % of participants who were CMD cases at baseline of each cycle remained recurrent CMD cases, 47 % became recurrent depression cases and 58 % recurrent clinical depression cases.

This group may tend to report higher consumption at study baseline even in the absence of depression at the time of the questionnaire, while having an increased risk of future depression compared to other participants14, 15.

To minimize this bias, we excluded at baseline 10 280 women with severe depressive symptoms and we computed the cumulative mean of caffeinated and noncaffeinated beverages with at least a 2 - year latency; yet, we can not exclude the possibility that mild depressive symptoms were the common reason for low caffeine consumption and incident depression.

This incidence is not directly comparable to that observed in unselected populations because to minimize reverse causation, we excluded women with severe depressive symptoms at baseline, thus eliminating a group at higher risk of depression.

On the whole, depression levels decreased between baseline and follow - up, with 14 % of participants found to have depression diagnosed through clinical interview at follow - up (an average of two years later).

Youth baseline and follow - up interviews assessed mental health — related quality of life using the Mental Health Summary Score (MCS - 12)(range of possible scores, 0 - 100), 48,49 overall mental health using the Mental Health Inventory 5 (MHI - 5)(range of possible scores, 5 - 30), 50 service use during the previous 6 months using the Service Assessment for Children and Adolescents51 adapted to incorporate items assessing mental health treatment by primary care clinicians, 52 and satisfaction with mental health care using a 5 - point scale ranging from very dissatisfied (1) to very satisfied (5).53 CIDI diagnoses of major depression and dysthymia were evaluated at baseline and follow - up.

At both baseline and follow - up there was a high rate of depressive symptoms with one third of the group scoring 14 or more on the Beck Depression Inventory (a questionnaire designed to measure severity of depressive symptoms).

The pre — post effect size (d) was 0.95, and pre — follow - up was 1.08, comparable to effect sizes published investigating face - to - face mindfulness interventions for depressive symptoms in those with diabetes, PTSD and cancer15, 56, 57 and online cognitive therapy interventions for depressive symptoms in a moderately depressed sample.27, 36 The change in PHQ - 9 is higher than effect sizes found for IAPT depression and anxiety treatment where follow - up was at 4 and 8 months (0.46 and 0.63, respectively) 3 where the IAPT sample started with higher baseline depression scores.

Categorical outcomes for depression (50 % decrease in depression scores on symptom checklist and major depression by structured clinical interview for DSM - IV) since baseline assessment at three and six month blinded outcome assessments in patients receiving usual care (n = 196), feedback only (n = 221), and care management (n = 196)

At 12 weeks, the intervention group adjusted mean score for depressive symptoms on the BDI - II was significantly lower than the control group by 5.8 points (95 % CI − 11.1 to − 0.5) after adjusting for baseline depression scores, anxiety, sociodemographics, psychotropic medication use and clustering by practice.

All children of mothers whose depression remitted after treatment and who themselves had no baseline diagnosis for depression remained free of psychiatric diagnoses at 3 months, whereas 17 % of the children whose mothers remained depressed acquired a diagnosis.

Anxiety was measured using the Zung Self - rating Anxiety Scale, 16 and depression was assessed using the Zung Self - rating Depression Scale17 at baseline as well as at fodepression was assessed using the Zung Self - rating Depression Scale17 at baseline as well as at foDepression Scale17 at baseline as well as at follow - up.

Of the children with a diagnosis at baseline, remission was reported in 33 % of those whose mothers» depression remitted compared with only a 12 % remission rate among children of mothers whose depression did not remit.

Depression rates had not fallen lower than those at baseline by 9 months, although the ethnic difference remained (figure 1C, D).

Formal tests to determine if the above rates of changes in children's diagnoses varied with mothers» remission status were statistically significant (P =.02), and remained significant after further adjusting for maternal depression severity at baseline, maternal treatment setting, annual household income, and child treatment status during the 3 - month follow - up interval (P =.01).

In the comparison of behavioural therapy versus CBT or cognitive therapy, effect size varied according to depression severity at baseline, though this was not the case with behavioural therapy versus control.

The elevated 30 - month Center for Epidemiological Studies Depression Scale scores in the intervention group were driven by a higher prevalence of depressive symptoms among the PP+HS group (a difference not present at baseline).

Percentage of the Young - HUNT cohort (n = 7497) in receipt of long - term medical benefits at different ages during follow - up according to self - reported anxiety and depression symptom level at baseline.

This result was likely to be attributable to unexplained high depression scores in the intervention group parents at baseline.

For example, variations in the still - face effect have been associated with mothers» baseline sensitivity and interactive style, and the infants» later attachment classification at age 1, internalizing (e.g. depression, anxiety) and externalizing (e.g. aggression, impulsivity) behaviors at 18 months, and behavior problems at age 3.

At baseline, severe depression (BDI score ≥ 10) was present in 27 men (14.4 per cent); mild depressive symptoms (BDI scores 5 — 9) in 73 (38.8 per cent); and no depression (BDI score < 5) in 88 men (46.8 per cent).

It is unclear how dementia and depression were assessed at baseline.

Maternal depression was measured with the abbreviated Center for Epidemiologic Studies Depression Scale at baseline, preschool, and kindepression was measured with the abbreviated Center for Epidemiologic Studies Depression Scale at baseline, preschool, and kinDepression Scale at baseline, preschool, and kindergarten.

It was hypothesized that preschool depression would show homotypic continuity over the course of 24 months, evidenced by a greater likelihood of subsequent depression when compared with rates observed in those with other psychiatric disorders and those without disorders at baseline.

At baseline (start of study) and 2 and 5 years after completion of the rehabilitation program all participants were evaluated in terms of sick leave, pain rating (Visual Analogue Scale), the Disability Rating Index, Hospital Anxiety and Depression Scale, and Stress Test.

At baseline, the Diagnostic Interview Schedule (DIS - III - R) and the Beck Depression Inventory (BDI) were administered.

Mean (95 % confidence interval) scores on hospital anxiety and depression scale and sleep problem questionnaire at baseline and follow up at three, six, and 12 months

Analyses controlled for baseline scores on each outcome measure, as well as for both the youth's self - reported depression and the severity score on a self - report screening measure for adolescent substance abuse, at baseline.

Nevertheless, many families with known and measured risks for behaviour problems were recruited, with 39 % of the sample at baseline reporting the risk factors of maternal depression, anxiety, substance misuse, partner conflict, social isolation and / or financial problems.5

Degree of phobic avoidance, bodily sensations, anxiety cognitions, and depression were assessed at pretreatment, baseline, and end of treatment and at follow - up after 3 and 12 months.

In several prospective studies, our group at Massachusetts General Hospital examined rates of depression in children with ADHD.45 In a 4 - year follow - up, lifetime rates of comorbid depression in children with ADHD increased from 29 % at baseline to 45 % at average age 15.

They were assessed at baseline using the Hamilton Rating Scale for Depression (HAM - D) and the Beck Depression Inventory.

Measures administered to children at baseline post-treatment, 6 - and 12 - month follow - ups included the Kiddie Schedule for Affective Disorders for School - age Children - Present and Lifetime Version (K - SADS - PL - PTSD) PTSD subscale, Children's Depression Inventory (CDI), State - Trait Anxiety Inventory for Children (STAIC), and the Children's Attributions and Perceptions Scale (CAPS).

Where participants were not diagnosed with anxiety and depression symptoms at baseline, scores were compared against...