Sentences with phrase «depressive symptoms at»

Namely, in adolescents who scored high in theses schema domains, the level of depressive symptoms at T1 and T2 was higher when bullying victimization was high than when it was low.

Depressive symptoms in adolescence were predicted by gender, children's depressive symptoms at age 8, maternal depressive symptoms in adolescence, and maternal depressive symptoms in infancy.

Peer nomination measures at Time 1 assessed both types of crush experiences and mutual friendship involvement, and participants completed self - report measures of loneliness and depressive symptoms at Times 1 and 2.

Specifically, we found that smaller hippocampus volumes and greater responses to sad faces in emotion reactivity regions predict increased depressive symptoms at the time of scan, whereas larger amygdala volumes, smaller insula volumes, and greater responses in emotion reactivity regions predict decreased emotion regulation skills.

Two interaction effects also revealed that crush status was a risk factor for depressive symptoms at low levels of anxious - withdrawal but a protective factor at high levels.

The results from regression analyses revealed significant associations between having an other - sex crush and depressive symptoms at Time 1 and loneliness at Time 2, after accounting for the effects of mutual friendship.

Loneliness was found to be a correlate of depressive symptoms at the cross-sectional level, independent of gender, other demographic factors, multiple psychosocial variables, and social desirability.

Disorganization of attachment in infancy and maternal hostility were independent predictors of depressive symptoms at age 8 and did not mediate the relation between maternal and child depressive symptoms.

Participants included 410 early adolescents (53 % female; 51 % African American; Mean age = 12.84 years) who completed measures of social anxiety and depressive symptoms at three time points (Times 1 — 3), as well as measures of general interpersonal stressors, peer victimization, and emotional maltreatment at Time 2.

Over three - quarters of mothers (78.7 %) provided retrospective reports of clinically significant depressive symptoms regarding the week following their child's ASD diagnosis, with some 37.3 % continuing to report clinically significant levels of depressive symptoms at follow - up.

Maternal depressive symptoms during infancy contributed to the prediction of child depressive symptoms at age 8, after controlling for concurrent maternal depressive symptoms, clinical risk in infancy, and gender.

The total indirect effect was β = −.04, p <.01, and the total effect of self - esteem on depressive symptoms at T5 was β = −.08, p =.01.

The association between timing of menarche and depressive symptoms at 10.5, 13 and 14 years was examined within a structural equation model.

1Maternal reports of partner's alcohol consumption; 2Univariable linear regression models; 3Models adjusted for maternal age at delivery, parity, social economic position, maternal education, maternal smoking during first trimester in pregnancy, housing tenure, income, and maternal depressive symptoms at 32 weeks gestation.

As we have observed here it is possible to come to different conclusions about whether depressive symptoms at age 12 increase risk of PEs at age 18 independently of the effects of persisting depression when using standard regression techniques compared to using an SEM approach.

We hypothesize that clique isolation at age 11 to 13 years would predict self - reported depressive symptoms at age 14 years, even if initial levels of depressive symptoms at age 11 years and other problems in the peer relations domain (i.e., peer rejection and friendlessness at age 11) would be controlled for.

Self - esteem at T1 was significantly associated with depressive symptoms at T5 (β = −.08, p <.01) when controlling for depressive symptoms at T1 (β =.23, p <.001).

Non-significant direct paths from self - esteem tot the social factors and associations from the control variable depressive symptoms at T1 are not depicted.

1Maternal reports of partner's alcohol consumption; Model 1 adjusted for maternal age at delivery, parity, social economic position, maternal education, maternal smoking during first trimester in pregnancy, housing tenure, income, and maternal depressive symptoms at 32 weeks gestation; Model 2 further adjusted for maternal alcohol use at 18 weeks gestation.

2Models adjusted for maternal age at delivery, parity, social economic position, maternal education, maternal smoking during first trimester in pregnancy, housing tenure, income, and maternal depressive symptoms at 32 weeks gestation.

Forty - eight percent of women had depressive symptoms at 1 or 2 time points (ever symptoms) and 12 % had depressive symptoms at all points (persistent symptoms).

This study used participant's reports of marital conflict at time 2, depressive symptoms at time 3, and physical health at time 4.

Accounting for children's initial level of depressive symptoms at age 11 years and for other problematic peer experiences (i.e., age 11 peer rejection and friendlessness), the results showed that the probability of being isolated from cliques from age 11 to 13 years predicted an increase in depressive symptoms from age 11 to age 14 years.

Note: 1Maternal reports of partner's alcohol consumption; 2Univariable multinomial logistic regression models; 3Multinomial logistic regression models adjusted for maternal age at delivery, parity, Social economic position, maternal education, maternal smoking during first trimester in pregnancy, housing tenure, income, and maternal depressive symptoms at 32 weeks gestation; CL: childhood limited, AO: adolescent onset, EOP: early onset persistent, the Low conduct problems class was used as the reference group.

NCS related to depressive symptoms at the level of a trend in middle to late adolescent boys reporting many (mean +1 SD) stressors (β =.81, p =.10), whereas this relationship was not significant in middle to late adolescent boys reporting few (mean − 1 SD) stressors (β =.30, p =.37).

While accounting for initial levels of depressive symptoms, peer rejection, and friendlessness at age 11 years, a high probability of being isolated from cliques from age 11 to 13 years predicted depressive symptoms at age 14 years.

There was a bidirectional association between the use of negative coping strategies and depressive symptoms, such that using more (as measured by a count and a mean) negative coping strategies at Time 1 was associated with higher depressive symptoms at Time 2, and depressive symptoms at Time 1 were positively associated with more engagement in negative coping strategies at Time 2.

Results showed that among the five classes of value affiliation, two classes had a greater likelihood of experiencing depressive symptoms at the beginning of the assessment (age 15).

Only the absence of positive self / other perceptions, and negative peer and mother expectations at age 8 predicted depressive symptoms at age 10 (after controlling for depressive symptoms at age 8).

We also observed a similarly strong relationship between depressive symptoms at age 12 and PEs at age 18.

However, perceived social acceptance did not predict depressive symptoms at age 14 years above and beyond the association between clique isolation and depressive symptoms.

Simple slope tests with a cutoff of ± 2 SD indicated that in the condition of very high negative parenting (b = 0.00, t = 0.01, p >.05) or very low negative parenting (b = − 0.00, t = − 0.11, p >.05), A1 allele was also not related to depressive symptoms at T3.

However, when using an SEM approach we found little evidence that those who had high depressive symptom scores at 12 were more likely to experience PEs at 18 if their depressive symptoms had resolved by this age, whereas those with PEs at 12 were slightly more likely to experience depressive symptoms at 18 even if their PEs had resolved by 18 years.

Adolescents with an approach - oriented coping style reported the fewest depressive symptoms at Time 3 and Time 4, whereas avoidant copers reported the most at both times.

With standard regression modelling we found strong evidence that PEs at age 12 were associated with depressive symptoms at age 18 even after adjusting for concurrent depressive symptoms at age 12 and persistence of PEs at age 18.

Additional analyses indicated that pubertal timing moderated the association between synchrony and depressive symptoms at age 20, such that girls who exhibited asynchronous development had the highest levels of depressive symptoms when they matured later than peers.

The first objective of the present study was therefore to investigate whether being isolated from cliques from age 11 to 13 years predicted an increase in depressive symptoms at age 14 years, while controlling for other problems in the peer relations domain.

Results indicated that maternal positive and negative parenting significantly concurrently predicted adolescent depressive symptoms at all three waves, whereas TaqIA polymorphism had no main effect on depressive symptoms.

Smoking cessation at the end of treatment and at 6 months after the start of treatment: measured by self - report and / or biochemical verification (expired carbon monoxide (CO) level); reduction in nicotine dependence at the end of treatment and at 6 months (measured by change in expired CO level and self - reported number of cigarettes smoked, and other biochemical measurements such as serum cotinine levels); change in positive, negative and depressive symptoms at the end of treatment measured using validated tools; adverse events.

Recently, it was reported that only academic self - efficacy, and not social self - efficacy, predicted depressive symptoms at 6 to 8 months follow - up in adolescents from fifth to eight grade, when controlling for shared variance of academic and social self - efficacy (Scott and Dearing 2012).

However, higher levels of emotional self - efficacy lead to lower levels of depressive symptoms at 2 years follow - up in middle adolescence (Bandura et al. 2003).

Depressive symptoms at the beginning of treatment were negatively associated with general life satisfaction (GLS) in all models.

Similarly, in a study of Interpersonal Psychotherapy approaches to preventing youth depression, Young and colleagues found that teens who participated in a skills - based intervention targeting interpersonal role disputes, role transitions and interpersonal deficits reported fewer depressive symptoms at six - months follow - up than teens who were assigned to a school counseling control group.

Guided by the Behavioral Vaccine Theory of prevention, this study uses a no - control group design to examine intervention variables that predict favorable changes in depressive symptoms at the six - to - eight week follow - up in at - risk adolescents who participated in a primary care, Internet - based prevention program.

Children in CFF - CBT had more improvement in parent - reported mania scores, lower parent - rated depression scores and a steeper response curve for depressive symptoms at post-treatment and 6 - months (effect sizes of 0.48 — 0.69).

Children whose parents had higher subthreshold depressive symptoms at baseline showed greater improvements in depressive symptoms in CFF - CBT than in TAU (d = 0.57).

Results also indicate younger grandmothers experienced higher levels of depressive symptoms at baseline and lower levels of mental health quality of life when compared with older grandmother.

The purpose of this study was to examine the effects of the Strong African American Families (SAAF) on a subset of 167 families in which the primary caregivers demonstrated elevated levels of depressive symptoms at pretest as indicated by a score of 16 or higher on the Center for Epidemiologic Studies — Depression scale (CES — D).

Twice as many of the intervention group maintained a reduction in depressive symptoms at six month follow - up.

Group without type 2 diabetes at baseline: 3.2 years; group without elevated depressive symptoms at baseline: 3.1 years.