This explains how many primitive societies could tolerate extremely high carbohydrate diets without
developing hyperinsulinemia or insulin resistance.
I remember Dr. Rosedale speaking of how there are genetic differences in how much insulin is released in response to carbs and that the people who have higher insulin responses to meals are prone to
developing hyperinsulinemia over time.
Not exact matches
WNIN Mutant Obese Rats
Develop Acute Pancreatitis With the Enhanced Inflammatory Milieu WNIN Mutant Obese Rats
Develop Acute Pancreatitis With the Enhanced Inflammatory Milieu WNIN / Gr - Ob rats demonstrate features of metabolic syndrome that include obesity, insulin resistance, impaired glucose tolerance, and
hyperinsulinemia.
That means they get hyperglycemia and
hyperinsulinemia after consumption of sugar and
develop insulin resistance after regular consumption.
Beyond genetics, a powerful risk factor for
developing AD is chronically elevated insulin, also called
hyperinsulinemia.
An extreme example of TOFI is lipodystrophy were people can not gain body fat and quickly
develop diabetes,
hyperinsulinemia, fatty liver, hypertriglyceridemia and heart disease because they aren't able to buffer their energy in their fat stores before it goes to their vital organs.