So the even length SFAs can reflect excessive
dietary SFA or excessive carbohydrates.
Not exact matches
Over the years, data revealed that
dietary saturated fatty acids (
SFAs) are not associated with CAD and other adverse health effects or at worst are weakly associated in some analyses when other contributing factors may be overlooked.
In this cohort, substituting
dietary n - 6 LA in place of
SFA increased the risks of death from all causes, coronary heart disease, and cardiovascular disease.
These findings could have important implications for worldwide
dietary advice to substitute n - 6 LA, or PUFAs in general, for
SFA.
These unfavorable effects of n - 6 LA shown in the SDHS are consistent with two other randomized controlled trials, in which experimental
dietary conditions selectively increased n - 6 LA in the place of
SFAs by replacing animal fats and common margarines with corn oil.50 51 Together, these three trials provide a rare opportunity to evaluate the specific effects of increasing n - 6 LA without confounding from concurrent increases in n - 3 PUFAs.
My negative comments regarding the high fat low carb paleo diet are based on a review of
dietary guidelines around the globe in regards to
SFA.
Because of the lack of
dietary data on individual fatty acids in the Iranian food composition table, we were unable to compare
dietary intakes of
SFAs, TFAs, monounsaturated fatty acids, and PUFAs across quintiles of PHVOs and non-HVOs.
Perhaps the more important question is the role of
dietary fat, especially
SFA, based on apoA - II genotype.
Strong and consistent evidence indicates that
dietary n - 6 polyunsaturated fatty acid (PUFA) are associated with improved blood lipids related to cardiovascular disease (CVD), in particular when PUFA is a replacement for
dietary saturated fatty acid (
SFA) or trans fatty acid.
«Strong evidence indicates that
dietary saturated fatty acids (
SFA) are positively associated with intermediate markers and end - point health outcomes for two distinct metabolic pathways: 1) increased serum total cholesterol (TC) and LDL cholesterol (LDL - C) and increased risk of cardiovascular disease (CVD) and 2) increased markers of insulin resistance and increased risk of type 2 diabetes (T2D).
Depression was associated with higher GI quintiles; younger age; higher BMI; less physical activity; higher intakes of
SFAs, MUFAs, PUFAs, and trans fat; and lower intakes of fruit, vegetables,
dietary fiber, and Healthy Eating Index score.
MUFA has, however, been associated with higher HDL cholesterol concentrations, 15,30 ⇓ reflected in lower total: HDL cholesterol and LDL: HDL cholesterol ratios, as potentially important predictors of cardiovascular risk.21 Higher
SFA intakes in exchange for carbohydrate in the DELTA (
Dietary Effects on Lipoproteins and Thrombogenic Activity) study were associated with a lower Lp (a) level, 31 an effect associated in the present study with higher MUFA intakes.
Because of the inherently lean nature of wild animal tissues throughout most of the year (Figure 5) and the dominance of MUFAs and PUFAs, high
dietary levels of
SFAs on a year - round basis (Figure 6) could not have exerted adverse selective pressure on the hominin genome before the development of agriculture.