Not exact matches
Also, they have chosen a cheap populist theme, attacking
cell phone, bank and credit - card charges instead of hammering the Conservatives on their dangerous, right - wing deregulation agenda — even though people are still
dying as a result of Harper's «self - regulation» changes to food safety.
And we can forget opennes on abortion, stem
cell research and let people make their own decisions
as to how to
die.
The researcher told me it was the sound of a
dying cell — a high - frequency discharge
as the
cell's life ebbed away.
This is the most effective probiotic strain to take when trying to kill candida
as it actively punctures the yeast
cell walls and causes
die off.
Much like our webbed fingers and toes,
as your fetus matures the
cells between the tongue and the lower jaw mature and some
die off, leaving the tongue
as a separate muscle in the mouth.
Middle - aged people who eat protein - heavy diets are four times
as likely to
die of cancer
as those who eat only a little protein, according to the study, which was published in the journal
Cell Metabolism.
These symptoms arise because nerve
cells producing the neurotransmitter dopamine, located in an area of the brain known
as the substantia nigra, slowly
die.
Lowered density usually means that nerve -
cell bodies, known
as gray matter, or the material around them, have thinned out or
died.
«Amyloid is one of many substances that builds up in plaques
as a result of
dying cells and atrophy in the brain,» he says.
They found —
as they did in the 2016 study — that when these
cells expressed SOD1 mutants that predominantly form trimers, the
cells died much more quickly than control
cells containing normal SOD1.
The main reason why people
die of cancer is that the cancer
cells spread to form daughter tumours, or metastases, in vital organs, such
as the lungs and liver.
Also known
as the «Guardian of the Genome,» p53 fights cancer by causing damaged
cells to
die or by halting the growth of mutant
cells before they become cancerous and spread to the rest of the body.
As it is normal for skin
cells to
die and flake off, a small amount of flaking is normal and in fact quite common.
The animals were five times
as likely
as regular mice to go into shock and
die when exposed to bacterial
cells, the group reports in the November 15 issue of Genes and Development.
The act of reprogramming
cells to make them
as capable
as ones from embryos apparently can result in aberrant
cells that age and
die abnormally, suggesting there is a long way to go to prove such
cells are really like embryonic stem
cells and can find use in therapies.
Using
cells from cadavers, doctors have been experimentally transplanting pancreatic islets into humans for decades, but
as many
as 60 percent of the transplanted islets
die immediately because they are cut off from their blood supply and are killed by an immune response due to direct injection into the bloodstream, and those that survive the transplant usually
die within several months.
Failure of an enzyme to break down DNA spilling into the bloodstream
as cells die may be a major driver of inflammation in lupus.
The tremors and other movement impairments of Parkinson's are triggered by the death of dopamine - producing
cells in the brain, so the investigators used flies that had been genetically engineered to have their dopamine
cells die off
as they age.
«Instead of cleaning up when neurons
die, the microglia assume they're under attack when they see too much
cell death and kill nerve
cells as collateral damage,» says Tanzi, who led the research team.
Now, scientists have a provocative new theory that might explain in part this universal human decline:
Dying cells secrete a protein that could trigger others to
die as well, accelerating the body's deterioration.
The control mice, with all genes intact, should have lost sight
as photo - receptors — the light - sensitive
cells in the retina —
died.
The retinal pigment epithelium (RPE) is a single layer of
cells that accomplishes multiple functions, such
as providing survival molecules that prevent photoreceptors from
dying.
Following cancer therapy, the dominant
cells may
die first, and other
cells that were originally not
as fit may find themselves better able to compete for necessary space and nutrients and continue to grow and take over the tumor.
This led to a gradual increase in Spo0A activity
as opposed to a spike, and such
cells were several times more likely to fail or
die during sporulation.
Researchers used the new therapy to inhibit a pathway in the cancer
cells known
as mTOR signaling — putting the brakes on this pathway, combined with the current standard therapy, caused more of the cancer
cells to
die.
Because retinal
cells do not divide or
die out but remain constant throughout our life, the deposited gene would last
as long
as the eye itself.
As a result, Katlyn's immune
cells were
dying.
More recently, however, researchers have suggested that macrophages — specialized immune
cells that reside in the dermis — are attracted to the wound inflicted by the tattoo needle and gobble up the tattoo pigment just
as they would normally engulf an invading pathogen or piece of a
dying cell.
Nearly all of the cancer
cells died as a result, but a residual population of tumor
cells survived and became dormant.
Known
as the phosphatidylserine receptor, or PSR - 1, the molecule can locate and clear out apoptotic
cells that are pre-programmed to
die as well
as necrotic
cells that have been injured and are causing inflammation, said CU - Boulder Professor Ding Xue, who led one study and co-authored the other.
Necrosis is virtually the opposite: The
cell isn't trying to
die, but factors such
as blood loss or oxygen deprivation make it swell and explode.
One of them was used to reproduce acquired aplastic anemia: due to various types of damage, some of the stem
cells die, and those that remain need to divide more frequently in order to maintain the production of blood
cells;
as a result of so many divisions, the telomeres shorten and the disease appears.
Optic nerve hypoplasia is closely related to optic nerve atrophy, in which the optic nerve develops normally initially, but later degenerates
as its
cells die off.
Cardiovascular disease in these young patients develops
as vulnerable
cells lining the interior of major arteries (vessels that carry blood away from the heart) accumulate the toxic protein and
die.
The therapy employs a virus to insert a gene for a common ion channel into normally blind
cells of the retina that survive after the light - responsive rod and cone photoreceptor
cells die as a result of diseases such
as retinitis pigmentosa.
This finding is somewhat counterintuitive with regard to
cell growth since EGFR activation functions
as a positive growth signal, the researchers note; however, that fact
cells die when EGFR is inhibited is not counterintuitive, but shows the
cells are now addicted to the EGFR signal.
Researchers from Massachusetts Eye and Ear have, for the first time, identified rapidly proliferating
cells (known
as «neural crest - derived progenitor
cells») in the corneal endothelium of specimens from normal corneas and from corneas with Fuchs» Endothelial Corneal Dystrophy (FECD), a condition in which the
cells responsible for keeping the cornea clear
die prematurely — often leading to blindness.
«
Dying cells in fruit fly alert neighboring
cells to protect themselves:
As a result, neighbors become harder to kill.»
The consequences,
as described in a paper published in
Cell this June: The melanocytes that originally went on to rejuvenate instead only matured and
died.
Single -
celled organisms however can not utilise this strategy,
as they only comprise a single
cell — hence upon significant damage, they usually
die.
We replace our entire bodies
as our
cells die off and regenerate through the decades.
Much like thermostats that adjust the temperature in a room to make sure it's not too hot or too cold, kinases make sure that the right number of new
cells are created
as old ones
die.
Previous attempts to maintain cultures of the so - called nephron progenitor
cells often failed,
as the
cells died or gradually lost their developmental potential rather than staying in a more medically useful precursor state.
With the passage of time, patches of retinal pigment epithelial
cells may
die, resulting in bare spots known
as geographic atrophy.
Most of the
cells died, but
as expected, some found a way to survive and multiply.
In other neurodegenerative diseases, such
as Parkinson's — which primarily affects a person's motor abilities — scientists know that nerve
cells begin to
die long before symptoms appear.
An asymmetric
cell division in non-stem
cells can instead result in two daughter
cells with very different fates, such
as one large
cell that reinitiates
cell division prematurely and a much smaller
cell that either grows very slowly or
dies.
The drug, known
as HRX9, works by preventing the cancer
cells from avoiding apoptosis — the natural process by which unhealthy and damaged
cells close themselves down and
die.
Specifically, the
dying cells produced seven times
as much of the protein
as normal brain
cells did.
Earlier research had shown that
cells surrounding an HIV - infected
cell die as a result of apoptosis, or programmed
cell suicide, and scientists knew that Nef had a hand in starting that process.