Bottom row: skeletal muscle with restored
dystrophin after application of the CRISPR / Cas9 platform.
Not exact matches
It's important to remember that we're not going
after the primary cause of the disease,
dystrophin deficiency.»
Skeletal muscle cells isolated using the ERBB3 and NGFR surface markers (right) restore human
dystrophin (green)
after transplantation significantly greater than previous methods (left).
After cultured rat heart cells were infected with the Coxsackie B virus, the amount of normal
dystrophin declined within 3 days.
Indeed,
after injecting microdystrophin (a «shortened» version of the
dystrophin...
The therapy delivered directly to a leg muscle restored
dystrophin and increased muscle strength in the adult mouse,
after which the researchers injected a combination of CRISPR and AAV, the most popular virus today for delivering genes, into the bloodstream to reach every muscle.