Recent theoretical work suggests that bullying might arise out of
early cognitive deficits — including language problems, imperfect causal understanding, and poor inhibitory control — that lead to decreased competence with peers, which over time develops into bullying.14, 15 A small number of studies provide circumstantial evidence that such a hypothesis might have merit7: 1 study found a link between poor early cognitive stimulation and (broadly defined) inappropriate school behavior, 16 and another found cognitive stimulation at age 3 years to be protective against symptoms of attention - deficit disorder at age 7 years.17 A study of Greek children found that academic self - efficacy and deficits in social cognition were related to bullying behavior.18 A large US national survey found that those who perceive themselves as having average or below - average academic achievement (as opposed to very good achievement) are 50 % to 80 % more likely to be bullies.8 Yet these studies are based on cross-sectional surveys, with the variables all measured at a single point in time.
The early intraneuronal pathology was accompanied by a significant elevation of soluble Aβ42 peptides that paralleled the presence and progression of
early cognitive deficits, several months prior to amyloid plaque deposition.
While many contend that
the earliest cognitive deficits are caused by damage to the striatum — a structure deep in the brain known to be severely affected in HD — recent evidence suggests that this claim may paint an incomplete picture of the widespread changes occurring in the brains of HD patients during the very early stages of the disease.
Not exact matches
Understanding
deficit patterns very
early, particularly speech and language delays,
cognitive - intellectual
deficits, sensory - motor impairments and a rough estimate of the «stage of psychological development or trauma» will help plot out the most appropriate treatment interventions.
Adversity
early on — malnutrition or neglect of an infant's physical and emotional needs, for example — can leave
cognitive deficits that persist for life.
The results, which could have implications for the
early diagnosis of autism and other
cognitive deficits, were published in the online
early edition of the Proceedings of the National Academy of Sciences on November 6, 2017.
«Neuroscientists identify source of
early brain activity: Brain cells that support
early structural development also transmit sensory information; discovery could enable
early diagnosis of autism and other
cognitive deficits.»
The results of this study provide measures associated with normal
cognitive aging in Border collies, and thus could be used as a basis for breed comparisons as well as
early recognition and treatment of certain
cognitive deficits.
The clinical symptoms and
cognitive and functional
deficits of schizophrenia typically begin to emerge during late adolescence and
early adulthood.
This finding is of great significance considering that
cognitive deficits paralleled the
early accumulation of soluble Aβ peptides and further progressed across age and with the advancement of the soluble Aβ42 pathology.
We therefore suggest that self - to - self assessments that reflect a
cognitive decline from an individual - adjusted baseline may be required to detect
early (and yet subtle) memory
deficits in pre-symptomatic human AD.
The
earliest cognitive impairment manifests at 4 months as a
deficit in long - term retention and correlates with the accumulation of intraneuronal Abeta in the hippocampus and amygdala.
Cognitive deficits may be an
early sign of B12 deficiency, which is more frequent in vegans and vegetarians than omnivores (see also here).
These children are considered «unready» for school because of
early cognitive and / or non-
cognitive «
deficits».
For example, in one study, neglected children had a smaller corpus callosum relative to control and comparison groups.8 Compared to their non-maltreated peers, children in another study who experienced emotional neglect
early in life performed significantly worse on achievement testing during the first six years of schooling.9 Furthermore, although both abused and neglected children performed poorly academically, neglected children experienced greater academic
deficits relative to abused children.10 These
cognitive deficiencies also appear to be long lasting.
These effects not only determine physical health but also emotional and
cognitive development.3 — 5 Maternal separation and nonbonding have a significant impact on
early brain development, which places children at risk of emotional and
cognitive deficit.
Deficits in self - regulation across multiple domains of functioning, from the physiological to the
cognitive, are associated with
early behavior problems, and may place children at greater risk for the development of later antisocial behavior.
Early paternal depressive symptoms predicted many aspects of children's outcome 3 years later, including externalizing and internalizing problems, social skills
deficits, and lower
cognitive and academic functioning, and predicted changes in children's externalizing, internalizing, and social problems across the preschool years.