These findings are consistent with an estrogenic hormonal proliferative
effect on breast tissue (6 — 10) that is reflected in the amount of dense tissue and breast density (11).
(55) A 2007 animal study with primates treated with either bioidentical progesterone or a progestin (Provera ™) showed that the progestin drug (Provera ™) worsens breast cancer biomarkers, while the bioidentical progesterone provides a beneficial
effect on breast tissue and reduces cancer markers.
For severe cases, doctors sometimes prescribe Danazol, a steroid derivative that decreases levels of the reproductive hormones FSH and LH, or tamoxifen, a breast - cancer drug that helps relieve breast pain by blocking estrogen receptors, thus preventing estrogens
effect on breast tissue.
Not exact matches
It may also have an
effect on the growth of the milk - making
tissue of the
breasts.
The authors believe theirs is the first study to show that mouse mammary gland
tissues are sensitive to a mixture of 23 commonly used UOG chemicals, with dose - specific
effects on tissue morphology, cell proliferation and induction of intraductal hyperplasias, an overgrowth of cells considered a marker for future
breast cancer risk.
Injections of iPSC - EPCs did not however have significant
effect on tumor growth or
on overall survival, but transducing cells with a baculovirus expressing CD40L, a member of the TNF gene family which can induce apoptosis [6, 7], and injection into the
breast cancer lung metastasis, increased levels of pro-apoptotic cytokines in lung
tissues, indicating the induction of apoptosis by CD40L carried by the EPCs (See figure).
Their results showed that by treating the
breast tissue directly results in a much more potent drug concentration where it is needed and with far fewer adverse
effects on tissue outside the
breast.
The cancer promoting
effects of estrogen
on breast tissue are restricted by lignan compounds which bind to estrogen receptors.
Turmeric is one of those few natural substances that work by directly blocking estrogen receptors
on the cellular level, hence having the
effect of reducing the sensitivity of your
breast tissue to estrogen.
You're body is used to all these endogenous artificial testosterone, you know, your testosterone to estrogen ratio is getting messed up and so you do have to use what's called post cycle therapy when you're
on pro-hormones and we won't get into the post cycle therapy as much
on this podcast «cause I know we're kinda pushing for time but the problem is that if you don't cycle pro hormones, it can be tough
on your liver, it can be tough
on your own endogenous production so it's something that you do wan na make sure that you do, that you understand how to cycle properly and I have to be careful of course, giving out recommendations like that
on this show just because so many people who are listening to this are competing in event like triathlons and marathons and thing of that nature where they're gonna be drug tested and stuff like this would be a big no - no anyways, you know, or they're going after more natural means and let's face it, prohormones can be kinda damaging to your body and the reason for that is because a lot of these side
effects: acne and hair loss,
breast tissue enlargement, or you know, what we affectionately call bitch tits in dudes, prostate swelling, you know, a lot of these hormonal imbalances that get created from dumping exogenous sources of hormones into your body and creating like a hormone milieu that can be a real real issue from a health standpoint.
We combine that with some of the like the possible pro-cancer
effects and the
effects on the prostate,
on acne,
on your own endogenous production of hormones which is why you get the testicular shrinkage and you know, the high amounts of testosterone getting converted to estrogen and the
breast tissue formation, you know I'm not really convinced that pro hormones are worth it in my opinion.
... The estrogenic compounds of the root are not the type that have an
effect on the uterus or the
breast tissue, but rather they are perceived by the hypothalamus of the brain as an estrogen metabolite and tend to lessen the pituitary surges of luteinizing hormone (LH) in the absence of ovarian estrogens.