Sentences with phrase «elegans also»

«Dying C. elegans also undergo what we term a «belly punch» phenomenon where death contraction in the head drives the pharynx backwards into the intestine, and the impact triggers cell death,» added Professor Gems.

As with humans, he says, the oocytes of C. elegans also show an increase in chromosome abnormalities with aging.

Not exact matches

The entire genome of the tiny nematode (Caenorhabditis elegans) also has been sequenced as a ta - ngen = tial study to the human genome project.

Many of these future studies will also employ C. elegans.

Working in the laboratory of Heather A. Hundley, corresponding author on the paper and an assistant professor of biochemistry and molecular biology in the IU School of Medicine's Medical Sciences Program at Bloomington, Washburn and undergraduate Medical Sciences program student Emily Wheeler collaborated with the team from UCSD to show that the region of ADR - 1 protein that binds to target mRNAs in C. elegans is also required for regulating editing.

We also lack information about C. elegans synapses.

Behaviour similar to that of a PS has also been observed in fission yeast (S. pombe) and the roundworm (C. elegans), albeit with no molecular explanation.

Their most surprising discovery was that serotonin isn't the sole driver of this weight - loss pathway, but works in concert with another neurotransmitter, octopamine — the C. elegans version of adrenaline (also called epinephrine) in mammals.

Mutations that make millimeter - long transparent worms known as Caenorhabditis elegans live longer also extend the proportion of their lives the worms spend being frail, Heidi Tissenbaum of the University of Massachusetts Medical School in Worcester and colleagues reported last year in the Proceedings of the National Academy of Sciences.

The study also solves a technical issue that has stymied C. elegans scientists for decades.

Species that live closer to shore, like the elegant cuttlefish (Sepia elegans), have also seen a steady rise in numbers, the researchers report today in Current Biology.

This mechanism was also found in another species, a worm called Caenorhabditis elegans, suggesting it is relevant to a variety of animals.

We also observe this process during development in C. elegans, and use measures of P granule growth rate, size distribution, and mechanical properties to test possible physical models that could underlie this behavior.

In a screen of nearly 200,000 compounds, Tardiff and collaborators identified one chemical entity that not only reversed alpha - synuclein toxicity in yeast cells, but also partially rescued neurons in the model nematode C. elegans and in rat neurons.

They next turned to a couple of model organisms with more cells — nematodes, also known as C. elegans, and fruit flies, or Drosophila.

Our work will not only address fundamental questions about the mechanisms of aging, but also provide a platform to greatly accelerate C. elegans aging research.

Dr. Falk is also PI of an NIH, pharma, and philanthropic funded translational research laboratory group at CHOP that investigates the causes and global metabolic consequences of mitochondrial disease, as well as targeted therapies, in C. elegans, zebrafish, mouse, and human tissue models of genetic - based respiratory chain dysfunction, and directs multiple clinical treatment trials in mitochondrial disease patients.

With similar precision, Professor Bargmann has also identified many of the intracellular signalling pathways in C. elegans that relay information from cell surface odorant receptors (G protein - coupled receptors) to the interior of each sensory neuron.