Sentences with phrase «elegans showing»

This is a picture of germ line cells of C. elegans showing PIWI and piRNAs (red dots) located outside of germ cell nuclei (blue circles) to scan for RNA passengers that move from nuclei to cytoplasm.

CK: We have studies in C. elegans showing that the daf - 2 gene functions exclusively in the adult to control aging.

Not exact matches

POISONED BY MOM A C. elegans nematode that inherits a gene for the antidote to a maternal toxin grows a normal feeding tube (shown first).

MOMMIE DEAREST Covertly poisoning offspring appears to be a common part of motherhood in the C. elegans nematode (shown) that stars in biology labs worldwide.

«In this study, we used the small roundworm C. elegans as a model to show that autophagy in the intestine is critical for lifespan extension,» said Malene Hansen, Ph.D., associate professor in SBP's Development, Aging, and Regeneration Program and senior author of the study.

These results showed that different genes control the life span of C. elegans and the length of reproductive time.

In a new study, Murphy, a molecular biologist at Princeton University, showed that long - lived bodily, or somatic, cells in Caenorhabditis elegans, a one - millimeter nematode commonly used as a model for aging studies in labs, activate genetic pathways completely separate from those found in long - lived egg, or oocyte, cells.

As with humans, he says, the oocytes of C. elegans also show an increase in chromosome abnormalities with aging.

Images of C. elegans embryos show inheritance and transmission of an epigenetic mark.

Working in the laboratory of Heather A. Hundley, corresponding author on the paper and an assistant professor of biochemistry and molecular biology in the IU School of Medicine's Medical Sciences Program at Bloomington, Washburn and undergraduate Medical Sciences program student Emily Wheeler collaborated with the team from UCSD to show that the region of ADR - 1 protein that binds to target mRNAs in C. elegans is also required for regulating editing.

Both studies relied on a popular lab organism known as C. elegans, a nearly microscopic nematode that is fast growing, translucent and has a sequenced genome showing that nearly half its genes are closely related to corresponding human genes.

Except for houseflies, animal species tested with CR so far, including primates, rats, mice, spiders, Drosophila, C. elegans and rotifers, have shown lifespan extension.

This shows that TRH is essential to normal growth in human beings as well as in C. elegans,» biochemist Isabel Beets explains.

The background image shows a picture of the nematode C. elegans with clumps of the Huntington's aggregates (bright green because they're tagged with green fluorescent protein) in the body wall muscle cells.

«Worms, mice, humans, and even fruit flies show similar effects of intoxication at similar alcohol concentrations,» he says, and human neurons contain a switch similar to that in C. elegans.

(b) An optical image showing a mid-L4 stage C. elegans trapped by multiple oscillating microbubbles.

The new study shows how a similar process of muscle contraction and relaxation occurs at death in C. elegans.

In old C. elegans specimens, those treated with dietary restrictions (right) showed more youthful gene splicing patterns than naturally - aged animals (left)(Credit: Caroline Heintz)

However, they found that when they, at the same time, knocked down proteins that are chromatin remodelers or part of chromatin complexes, they found that then reprogramming became feasible and they showed in particular that you can reprogram a cell in the germ line of C. elegans.

«C. elegans is a great tool to study aging in because the worms only live for about three weeks, yet during that time they can show clear signs of age,» says Caroline Heintz, first author of the study.

Next, the team used the process of dietary restriction, which has been shown to increase the lifespan of organisms like C. elegans, and observed that the worms» splicing patterns stayed youthful throughout the creatures» lives.

We have shown that the nematode C. elegans is a useful tool to study the cellular mechanisms involved in muscle degeneration.

In old C. elegans specimens, those treated with dietary restrictions (right) showed more youthful gene splicing patterns than naturally - aged animals (left)(Credit: Caroline Heintz)

Lithgow's recent work at the Buck Institute with the nematode C. elegans (a type of worm) has shown how iron accumulation can accelerate the aging process.

In a new study published Friday in the journal Science, a team of researchers has shown that such «epigenetic» memories can be passed down for 14 generations in a tiny nematode worm species called Caenorhabditis elegans (roundworms).

In a new study published Friday in the journal Science, a team of researchers has shown that such «epigenetic» memories can be passed down for 14 generations in a tiny nematode worm species called Caenorhabditis elegans (roundworm).

Terskikh et al. showed that their versatile timer could be used in vitro, in mammalian cells, in C. elegans, and in Xenopus embryos.

Caenorhabditis elegans, a worm with just 302 neurons, shows considerable sophistication in its behaviors, and its defined neuronal wiring and genetic accessibility make it an ideal subject in which to study these interactions.

As a postdoctoral fellow in Dr. Cori Bargmann's lab at the Rockefeller University, she showed that the nematode C. elegans produces a neuropeptide that is an evolutionary precursor of the mammalian peptides vasopressin and oxytocin, and mapped a neural circuit by which this molecule, nematocin, modulates mating behavior.

Studies on Caenorhabditis elegans, leucocytes diapedesis and cancer cell invasion have shown that BM transmigration is a conserved three - stage process.

The two components of Cas9 / gRNA have shown high DNA cleavage activity in cultured cells [6], [7], C. elegans [10], zebrafish [11] mice [12] and pigs [13].

Previously we showed that the innexin unc - 7 gene is essential for coordinated locomotion in C. elegans [9].

Reconstruction of the C. elegans nervous system by EM serial section showed that 92 of 104 motor neuron classes establish gap junctions [13].

Their results show the green color produced by mNeonGreen is significantly brighter than GFP in C. elegans, allowing visualization of low - expression genes that can't be seen using GFP.