This conclusion is supported by previous work (1, 12 - 14) showing that various low - quality proteins are better able to
enhance tumor development when they are supplemented with the amino acid in greatest deficit.
The researchers observed that
enhanced tumor development was associated with an altered intestinal microbiota, characterized by an increased pro-inflammatory potential.
Not exact matches
TGFβ inhibits
tumor initiation and progression by inducing cell cycle arrest and apoptosis; however epithelial tumorigenesis may escape this common antitumor mechanism by inducing aberrations in TGFβ signaling resulting in
enhanced development and progression of human carcinomas.