Calcium supplements interact significantly with long - term diet while suppressing rectal
epithelial proliferation of adenoma patients
TERT promotes
epithelial proliferation through transcriptional control of a Myc - and Wnt - related developmental program.
Not exact matches
Transient Silencing of 14 -3-3sigma promotes
proliferation of p63 - positive progenitor cells isolated from human breastmilk in mammary
epithelial cell culture.
Secretion of the hormones estrogen and progesterone set the stage for dramatic changes that take place in the breast during pregnancy: a massive
proliferation of mammary
epithelial cells, and the formation of thousands of ductal structures, which support milk production and transport during lactation.
In cell culture, high expression levels of GPR161 induced
proliferation of mammary
epithelial cells, disrupted the acinar structures formed by these cells, and enhanced their invasive capacity.
Immunohistochemical stainings of Ki67 and pancytokeratin (Ki67 is stained brown, pancytokeratin is stained pink) indicate
proliferation of hepatocytes (arrowheads) and biliary
epithelial cells (arrows) in TAK1 / RIP3 - deficient mice.
«The inflammasome in turn activates the enzyme caspase - 1, which then produces two proteins (IL - 1β and IL - 18) that play important roles in the GI tract, including activation of immune cells, induction of antimicrobial peptides, and regulation of
epithelial cell
proliferation.»
Collectively, these results suggest that Nrk negatively regulates the estrogen - dependent
proliferation of mammary
epithelial cells, the disruption of which leads to breast tumors.
The absence of tumors in virgin Nrk mutant mice strongly suggests that the tumorigenesis is closely related to the
proliferation of mammary
epithelial cells during pregnancy.
In Nrk mutant mice, therefore, mammary tumorigenesis was proposed to be triggered by a combination of dysregulation of
proliferation in mammary
epithelial cells and excessive blood estrogen.
This suggests that Nrk is expressed in mammary
epithelial cells late in pregnancy in order to terminate their
proliferation.
During pregnancy, the elevated blood estrogen level induces the
proliferation of mammary
epithelial cells, leading to the development of the mammary gland in preparation for lactation.
However, the regulatory mechanisms of mammary
epithelial cell
proliferation during pregnancy have been unclear.
This distribution helps to maintain the health and rapid
proliferation rate of adult
epithelial stem cells using MimEX ™ Tissue Model Systems.
MicroRNA -7-5p regulates the
proliferation and migration of intestinal
epithelial cells by targeting trefoil factor 3 via inhibiting the phosphoinositide 3 - kinase / Akt signalling pathway.
miR - 34a suppresses
proliferation and induces apoptosis of human lens
epithelial cells by targeting E2F3.
MiR - 514a - 3p inhibits cell
proliferation and
epithelial - mesenchymal transition by targeting EGFR in clear cell renal cell carcinoma.
In a first set of experiments, postdoctoral researcher and co-lead author Max - Felix Häring found that insulin increased the
proliferation of mouse
epithelial tumor cells in culture, as expected.
Effects of microRNA - 211 on
proliferation and apoptosis of lens
epithelial cells by targeting SIRT1 gene in diabetic cataract mice.
ANXA1 affects cell
proliferation, invasion and
epithelial - mesenchymal transition of oral squamous cell carcinoma.
Jens - Christian Roeper (Eaton, MPG)-- «The influence of cell mechanics, cell - cell interactions and
proliferation on
epithelial packing» (2009)
Reeh KA, Cardenas KT, Bain VE, Liu Z, Laurent M, Manley NR, Richie, ER (2014) Ectopic TBX1 suppresses thymic
epithelial cell differentiation and
proliferation during thymus organogenesis.
Smyd3 function is required for the activation of oncogenic pathways, cell
proliferation and
epithelial to mesenchymal transition during tumorigenesis.
In particular, they are focused on studying the functional role and molecular mechanisms of cellular senescence in both of these events, and how a deregulation of normal
epithelial stem cell
proliferation is involved.
In human gastric tissue, however, gastric
epithelial cell
proliferation is significantly higher but apoptotic indices are lower among persons colonized with cag + compared to cag - strains or uninfected persons leading us to postulate that reduced rates of cell loss, when accompanied by hyperproliferation, may heighten retention of mutagenized cells, which could predispose towards cancer.
For most of the cancer cells, 45 — 100 nM RIPK2 inhibitor 1 inhibited > 70 % of the
proliferation of colon and blood cancer cells while preserving the
proliferation of normal Rat - 1 and ModeK intestinal
epithelial cells (Fig. 6A).
In addition to its structural role in
epithelial junctions, beta - catenin activates TCF -4-mediated transcription of genes required for cell
proliferation.
Intestinal permeability was assessed by Ussing chamber;
epithelial cell (EC) ultra-structure by electron microscopy; RNA expression of genes coding for junctional proteins by Q - real - time PCR; immune response by in - vitro antigen - specific T - cell
proliferation and cytokine analysis by cytometric bead array; intestinal microbiota by fluorescence in situ hybridization and analysis of systemic antibodies against intestinal microbiota by surface staining of live bacteria with serum followed by FACS analysis.
The group who consumed soy protein had significant increases in the
proliferation of
epithelial cells in the breasts.
Intestinal
epithelial cell
proliferation is dependent on the site of massive small bowel resection
Effects of dietary wheat - bran fiber on rectal
epithelial cell
proliferation in patients with resection for colorectal cancers.