Not exact matches
For this study, Dr. Lou and his colleagues focused on triple - negative breast cancer, which is difficult to treat, because it
does not exhibit receptors for
estrogen, progesterone or the HER - 2 / neu gene, which are targets for many current breast cancer
treatments.
When a woman naturally reaches menopause — most often in her 50s — or
does so prematurely, for reasons such as a hysterectomy or cancer
treatment, levels of
estrogen in her body begin to plummet.
Triple - negative cancers are so called because they
do not express receptors for the hormones
estrogen and progesterone, nor for HER2 (human epidermal growth factor 2), and hence patients with these cancers are not candidates for
treatment with modern hormonal therapies or the highly effective HER2 - targeted drug Herceptin (trastuzumab).
Some women with
estrogen receptor - positive breast cancer
do extremely well on such anti-hormone
treatment.
«We don't know exactly how
estrogens are reduced,» Servos says of the water
treatment plant.
However, almost half of tumors that express the
estrogen receptor,
do either not respond well to the
treatment or develop resistance with time.
Because of this, this kind of breast cancer
does not respond to
treatments such as tamoxifen, which targets the
estrogen receptor, and trastuzumab, which disrupts the HER2 receptor.
While we occasionally
do see elevated
estrogen levels prior to starting
treatment for Low T, is is most often a problem that arises because of higher testosterone levels.
That
does not make
estrogen the right choice for
treatment.
Are there any
treatments of hormonal acne that
do not involve taking in
estrogen?
I
do not recommend this
treatment due to the fact that
estrogen replacement therapy, be it conventional or «natural» (50 % natural and 50 % synthetic hormones), is shown to increase risks of uterine cancer, breast cancer, stroke, heart attacks, blood clots, and even mental decline.
That progesterone may be used alone for severe VMS and unlike
estrogen does not appear to cause a withdrawal rebound increases VMS
treatment options.»
As more research is being
done on the impact of synthetic hormones such as Provera, synthetic progesterone, and Premarin, synthetic
estrogen, it is becoming increasingly evident that they are not viable means of
treatment.
WebMD [2] explains that the specific type of
treatment often also depends on some factors related to the woman who is experiencing these symptoms.When it comes to a pharmaceutical approach, a woman who still has a uterus will often received combination HRT (Hormone Replacement Therapy), while a woman who
does not have a uterus anymore (those who had undergone a hysterectomy) may only be prescribed a dose of
estrogen.