Not exact matches
Even before the company's novel gene - profiling tool was market - ready, even before they knew how to outflank a long - established competitor (Affymetrix), Stuelpnagel was looking far into the fut
Even before the company's novel
gene - profiling tool was market - ready,
even before they knew how to outflank a long - established competitor (Affymetrix), Stuelpnagel was looking far into the fut
even before they
knew how to outflank a long - established competitor (Affymetrix), Stuelpnagel was looking far into the future.
For example, we see the
genes for air - based olfaction (smelling) in whales that
no longer
even have olfactory organs.
So in the cited reference,
even though we start with 4 alleles, we
know that for many, many
genes, 3/4 would be lost, forever, after a few generations.
The DNA of many ancestors is in our blood and
genes have * memes *, which may explain why we sometimes get that deja vu feeling
even though we
know we've never been here before.
If Chad and others argue that naturalistic evolution must be dismissed because we don't
know exactly what happened with
gene mutation and transmission frequencies during particular periods of rapid change, then how can we accept a replacement argument in which we don't
even know what happens at all?
@ED The only thing that is assumed to be at least more or less constant in evolutionary theory is the mutation rate of individual
genes, and
even that, since mutations are
known, eg, to increase under higher radiation, is only true «on average, over the long run».
And
even though she isn't a sweet tooth (I guess it's not the dominant
gene in my family), I
know she can't resist a couple of desserts:
No, not
even Gene Hackman.
«If we
know which
genes are damaged in a breast cancer patient's immune system, prevention or
even therapy can be tailored by giving vaccines or perhaps antiviral drugs to reduce the chances of recurrence,» said Friedenson.
Using what we
know about human
genes, for example, could help us extrapolate details like Neanderthal hair and eye color, their genetic diseases, and possibly
even their language capabilities.
Zimmer: We do hardly
know anything about
genes that have to do with intelligence and what -LSB-'s] surprising is that this ignorance is taking place
even when we have really sophisticated tools.
We don't
know enough,» Esvelt says, adding, «I'm probably the foremost scientific critic of
gene drives
even though I'm a leader in the field.»
«Little is
known even about what
genes distinguish one neuron from another,
even though they might perform quite different functions,» Kandel says.
Though this is true for some traits, we now
know that many traits are controlled by tens or
even hundreds of
genes spread throughout our genomes.
«We don't
know if studying the fish will directly help us,» said Rohner, «but evolution has tried a lot of
gene variants over millions of years and I think that's smarter than anything we can come up with,
even with machine learning.
We're still struggling with the smallest bacterial cell, in which we don't
know what
even one - fifth of the
genes do.
«
Even when the
genes driving cancer are
known, clinicians don't have an efficient way to choose among the hundreds of possible drug therapies,» said study leader Kai Wang, PhD, associate professor of biomedical informatics and director of clinical informatics at the Institute for Genomic Medicine at CUMC.
It's long been
known that bacteria can swap DNA through a process called conjugation, which allows helpful
genes to spread quickly between individuals and
even between species.
We do not
know even the basic facts about it, such as how many
genes it contains — although the guesses are converging on a figure of 38,000 — let alone what each
gene is and how
genes interact with one another.
But oddly, the secretion still took place
even when they drastically mutated the
gene — a resilience not found in any
known secretion signal.
«We do not
even know the traits we will need in the future and which of the present breeds possess the requisite
genes,» he says.
Knowing the origin of each cell and which
genes control their normal function are the foundations for scientists to decipher the disease process and eventually to find out how to guide the cells to self - repair or
even to build up a brand new organ using amended cells from the patients.»
Her message: Not
even the experts can distinguish a 1 from a 4 if they don't
know the
genes.
«We don't
even know how
gene expression changes on a day - to - day basis!»
For instance, in the human genome we still see the remnants of the large olfactory receptor
gene family that gave our evolutionary ancestors a keen sense of smell,
even though humans
no longer rely on them.
Even some
known cancer
genes for lung adenocarcinoma, one of the most intensively studied cancers, haven't popped out in cancer genome surveys.
When mice were
no longer babies — at five weeks — the scientists were not able to regenerate their limbs,
even if the
gene was stimulated.
Compounding this risk, the liver cells that now contain the DNA for ZFNs will keep making the nucleases for perhaps years,
even though they are
no longer needed to guide the new
gene to its spot in the genome.
Segal: «We can not change our
genes, but we now
know that we can affect — and
even reshape — the composition of the different kinds of bacteria we host in our bodies.
«Lots of people working around
gene drives hadn't been in touch with each other and some didn't
even know what the CBD was,» Coche says.
Sharma and She note the reality that not all patients with the high - risk
genes even get the disease, although they still don't
know why.
CRISPR / Cas systems are
known as promising «
gene scissors» in the genome editing of plants, animals, and microorganisms by targeting specific regions in their DNA - and perhaps they can
even be used to correct genetic defects.
Yet
even knowing exactly which mutated
gene causes a disease — as in Huntington's and cystic fibrosis — doesn't necessarily mean it is either preventable or curable.
Almost one - third of the
genes in the minimal genome are secret ingredients that do something important,
even though scientists don't
know what that something is.
«With 20,000
genes, how is it
even possible to understand what you must
know in researching disease?
What is
known from global studies is that exposure to biologically active compounds,
even at ng / L concentrations, can impact endocrine systems in nature and manifest as negative impacts like altered
gene regulation, the presence of mixed reproductive tissues (intersex) and skewed sex ratios.
But she
knew if she succeeded, CRISPR would be «a profound discovery» — and maybe
even a powerful
gene therapy technique.
This is because the precise combination of
genes contributing to the trait can not be predicted either (or, perhaps,
even known).
«Our study demonstrates that
even well - studied bacteria — the ones that we
know cause disease or their close relatives — have many
genes associated with antibiotic resistance that have not been characterized before,» said senior author Gautam Dantas, PhD, associate professor of pathology and immunology.
«
Even though we identified 81 percent of the
genes that were already
known to be cell - cycle regulated, we found only about a 65 percent overlap between cell - cycle
genes identified by our method and the previous method.
That genetic litmus test has been applied since before people
even knew the chemical DNA is what makes up
genes.
But did you
know that we are often healthy
even when we have two faulty copies of the same
gene?
And for a vegan bodybuilder who must unfortunatelly play tetris with the food sources that he choses in order to give to his body the right ammounts of aminos, restricting SPI and soy foods so much does not make his goal any easier.There are sometimes that you need a meal thats complete with aminos and soy provides that meal with the additional benefits of lacking the saturated fats trans cholesterol and other endothelium inflammatory factors.I'm not saying that someone should go all the way to 200gr of SPI everyday or consuming a kilo of soy everyday but some servings of soy now and then
even every day or the use of SPI which helps in positive nitrogen balance does not put you in the cancer risk team, thats just OVERexaggeration.Exercise, exposure to sunlight, vegan diet or for those who can not something as close to vegan diet, fruits and vegetables which contains lots of antioxidants and phtochemicals, NO STRESS which is the global killer, healthy social relationships, keeping your cortisol and adrenaline levels down (except the necessary times), good sleep and melatonin function, clean air, no radiation, away from procceced foods and additives like msg etc and many more that i can not
even remember is the key to longevity.As long as your immune system is functioning well and your natural killer cells TP53
gene and many other cancer inhibitors are good and well, no cancer will ever show his face to you.With that logic we shouldn't eat ANY ammount of protein and we should go straight to be breatharians living only with little water and sunlight exposure cause you like it or not the raise of IGF1 is inevitable i
know that raise the IGF1 sky high MAYBE is not the best thing but we are not talking about external hormones and things like this.Stabby raccoon also has a point.And
even if you still worry about the consumption of soy... http://www.ncbi.nlm.nih.gov/pubmed/21711174.
Even though genetics (the last sub-group) is reported to accounted for 35 - 50 % of happiness, we now
know that «our
genes are not our destiny» and we can actually switch on good
genes and switch off bad
genes when we change our diet and environment.
Of course, we
know that
genes alone are not responsible for cancer because we share many of the same
genes as our hunter — gatherer ancestors and
even just the same
genes as our ancestors several generations ago, and yet the rate of cancer keeps going up.
Early - life stressors are
known to induce changes in
gene expression that set the stage for disease in later life.30 Thus, unfortunately, exposure to mercury in either parent,
even prior to conception, can affect the child's own genetic expression.
Last month of January I found out about stocktwits.com and been reading about «
GENE» also
known as Genetic Technologies and saw it going up more on the trending page then 100 % sometimes
even 150 + + % a day and so I sold (which was stupid)
It is well
known that purebred dog breeds are associated with differing susceptibility to specific malignancies, suggesting that selected breeds of dog are inheriting «at risk» alleles for very few
genes, perhaps
even a single
gene, with a profound effect.
Anyone who has studied genetics,
even at an introductory level,
knows that certain dominant
genes may actually be dormant for a generation.
You do not
even want to
know what we are doing by allowing the USDA to unleash a genetically engineered rabies virus live virus chimera into the environment without not one
gene impact study to validate the methodology.