It was by studying the cells of patients with an RNA
exosome mutation, which were contributed by six collaborating medical centers, that the investigators were able to understand how influenza A hijacks the RNA exosome inside a cell's nucleus for its own purposes.
Not exact matches
The role of rebiopsy and repeat analysis in the setting of post-treatment relapse, along with testing of blood samples for
mutations in circulating tumor cells, cell free tumor DNA, or
exosomes will be considered.
The study findings, published online today in Cell, also identifies a link between congenital defects in that machinery — the RNA
exosome — and the neurodegeneration that results in people who have that rare
mutation.