Sentences with phrase «exposure during fetal development»

Newbold said because decades can pass between exposure during fetal development or early childhood and the manifestation of the disease in adult life, it can be difficult to nail down a link.

The study is the first to link asthma to hexachlorobenzene exposure during fetal development, and builds on two earlier studies that linked the respiratory disease to polychlorinated biphenyls.

Not exact matches

Exposure begins during fetal development.

In animal models, exposure to cigarette smoke or nicotine during fetal development alters the expression of the nicotinic acetylcholine receptor in areas of the brainstem important for autonomic function, 28 alters the neuronal excitability of neurons in the nucleus tractus solitarius (a brainstem region important for sensory integration), 29 and alters fetal autonomic activity and medullary neurotransmitter receptors.30 In human infants, there are strong associations between nicotinic acetylcholine receptor and serotonin receptors in the brainstem during development.31 Prenatal exposure to tobacco smoke attenuates recovery from hypoxia in preterm infants, 32 decreases heart rate variability in preterm33 and term34 infants, and abolishes the normal relationship between heart rate and gestational age at birth.33 Moreover, infants of smoking mothers exhibit impaired arousal patterns to trigeminal stimulation in proportion to urinary cotinine levels.35 It is important to note also that prenatal exposure to tobacco smoke alters the normal programming of cardiovascular reflexes such that there is a greater - than - expected increase in blood pressure and heart rate in response to breathing 4 % carbon dioxide or a 60 ° head - up tilt.36 These changes in autonomic function, arousal, and cardiovascular reflexes might all increase an infant's vulnerability to SIDS.

But exposure to the chemicals blocked cell division, which could lead to neurological disabilities in babies if their cells are exposed at particular times during fetal development.

«Policy makers need to heed data such as those presented by Morales et al as limiting exposure to traffic - related pollution during fetal development and early postnatal life is one way that the burden of respiratory disease can be decreased.»

«This study shows that the combination of physical and psychosocial stressors during fetal development magnifies the effect of each exposure,» says lead author Frederica Perera, DrPH, PhD, director of the Center.

«Exposure of pregnant women to certain phenols may disrupt growth of boys during fetal development and first years of life.»

This link was only evident in blood samples taken at a time point reflecting exposure during early pregnancy when the fetal nervous system undergoes rapid development, not at birth.

[8] Increased exposure to environmental estrogens and other environmental pollutants during fetal development, as well as during the reproductive years, is suggested as a major reason for the tremendous rise in disorders of development and function of the male sexual system.

The World Health Organization and United Nations this year concluded: «Exposure to E.D.C.s during fetal development and puberty plays a role in the increased incidences of reproductive diseases, endocrine - related cancers, behavioral and learning problems, including A.D.H.D., infections, asthma, and perhaps obesity and diabetes in humans.»

«High exposure to estrogenic compounds during fetal and early childhood development through soy - based food products could disrupt the function of the natural steroid hormones and contribute to the high incidence of seizures associated with many childhood, neurological disorders including autism and FXS (Fragile X Syndrome).

Exposure to chemicals at low doses during early fetal development can cause brain injury to the fetus.

There are critical periods during fetal development for plant seeds just like for animals when exposure to something adverse has bad results — before or after the critical point, no worry.