Quantitative Imaging of Gut Microbiota Spatial Organization, Oct. 2015 — Sonnenburg published findings, elimination of microbiota - accessible carbohydrates (MACs) from the diet results in thinner mucus, increased proximity of microbes to the epithelium, and heightened
expression of inflammatory markers.
Prior update Nov 19, 2016 added Sonnenburg links (elimination of microbiota - accessible carbohydrates (MACs) from the diet results in thinner mucus, increased proximity of microbes to the epithelium, and heightened
expression of inflammatory markers.)
[Updated Nov 19, 2016] In reply to your comments, you are correct, I have mentioned another lab, the Sonnenburg Lab, and their analysis that elimination of microbiota - accessible carbohydrates (MACs) from the diet results in thinner mucus, increased proximity of microbes to the epithelium, and heightened
expression of inflammatory markers.
Selective NAc IKKβ inhibition reversed the upregulated
expression of inflammatory markers, prevented anxiodepressive behavior and blunted compulsive sucrose - seeking in mice fed the saturated HFD.
Additionally, the researchers said they found evidence that preconception alcohol exposure increased
the expression of some inflammatory markers in pancreatic tissue.
Prior update Nov 19, 2016 added Sonnenburg links (elimination of microbiota - accessible carbohydrates (MACs) from the diet results in thinner mucus, increased proximity of microbes to the epithelium, and heightened
expression of inflammatory markers.)
Not exact matches
We are examining the effect
of a maximal exercise stress test on gene
expression, metabolism,
inflammatory markers, and cytokine levels in ME / CFS patients and healthy controls.
Current projects are investigating: 1) the
expression of inflammatory genes during ischemia and reperfusion
of kidneys during urology transplantation and in mouse models; 2) the
expression of inflammatory genes and proteins in urine as
markers indicating the presence
of rejection in renal allografts; and 3) the role
of adhesion molecules and chemokines in directing leukocyte infiltration into organ allografts.
Increased
expression of Cd11b - a
marker of macrophages [38] seen with L - arginine admininistration in WNIN / Gr - Ob rats, might be due to increased influx
of inflammatory cells in the exocrine fraction via adhesion molecules [17].
Herein, we report alterations in arterial gene and protein
expression in the canine MPS I model system, the identification
of a potential
marker for MPS I cardiovascular disease, and data supporting the hypothesis that a GAG - induced
inflammatory process is responsible for the pathogenesis
of MPS I cardiovascular disease.