Targeted disruption of nuclear
factor erythroid - derived 2 - like 1 in osteoblasts reduces bone size and bone formation in mice.
Not exact matches
Sustained activation of nuclear
erythroid 2 - related
factor 2 / antioxidant response element signaling promotes reductive stress in the human mutant protein aggregation cardiomyopathy in mice.
Its presence in all erythropoietin - independent
erythroid colonies demonstrates a link with growth
factor hypersensitivity, a key biological feature of these disorders.