Not exact matches
A 2016 study observed the influence of the monk fruit component, mogrol, on
fat accumulation and found that it inhibited the
differentiation of pre-adipocytes (pre-
fat cells) into mature adipocytes (
fat cells)(11).
Using human
fat - derived stem
cells from young (aged 24 - 36 years), middle - aged (aged 48 - 55 years) and elderly (aged 60 - 81) participants, the team used ECIS to collect complex measurements during the growth and
differentiation stages the hASC exhibited as they converted to bone
cells.
PPARγ was initially linked to adipocyte (
fat cell)
differentiation, but we now know that it also regulates genes responsible for lipid uptake, accumulation, and storage of lipids in those
cells.
Unlike highly specialized skin -
cell fibroblasts, these
cells in the
fat have a relatively wide portfolio of
differentiation options — becoming
fat, bone or muscle as needed.
If brown
fat is to be increased, the signals / messages for
cell differentiation need to be switched from turning into muscle to turning into brown
fat.
Testosterone may even have a direct effect on
fat cells via «inhibited adipogenic
differentiation «[1].
When you gain body
fat, it's a combination of growth in
fat cell size and the formation of new adipocytes (aka
fat cells) through a process called adipogenesis, or adipocyte
differentiation [13].
A 2016 study observed the influence of the monk fruit component, mogrol, on
fat accumulation and found that it inhibited the
differentiation of pre-adipocytes (pre-
fat cells) into mature adipocytes (
fat cells)(11).