Dr. Kanda emphasized that there is currently no proof that gadolinium is responsible
for hyperintensity on brain MRI.
Not exact matches
Authors examined the association between SVD and AD pathology by looking at magnetic resonance imaging (MRI)- based microbleeds (MB), white matter
hyperintensities (WMH) and lacunes (which are measures
for SVD) along with certain protein levels in cerebrospinal fluid (CSF) which reflect AD pathophysiology in patients with AD, VaD and healthy control patients.
«The
hyperintensity of DN and GP on unenhanced T1WI may be due to gadolinium deposition in the brain independent of renal function, and the deposition may remain in the brain
for a long time,» Dr. Kanda suggested.
«Our results demonstrate that aortic arch pulse wave velocity is a highly significant independent predictor of subsequent white matter
hyperintensity volume and provides a distinct contribution — along with systolic blood pressure, hypertension treatment, congestive heart failure and age — in predicting risk
for cerebrovascular disease,» Dr. King said.