Sentences with phrase «for oxygen metabolism»

This broad - spectrum antimicrobial agent suffocates fungus, virus and bacteria by destroying enzymes needed for oxygen metabolism.

Not exact matches

They also are rich in iron, which helps transport oxygen from the lungs to the rest of the body, and is integral for energy production and metabolism.

Lentils are also a good source of vegetarian iron, an essential mineral needed for oxygen transportation in the blood and for proper metabolism.

Whole grains are also rich in essential nutrients, including B vitamins to help regulate metabolism; folate to help form red blood cells; iron, which carries oxygen in the blood; magnesium for building bones and releasing energy from muscles; and selenium for a healthy immune system.

HIF - 1 senses reduced oxygen levels and triggers many changes in cellular function, including a changed metabolism and sending signals for the formation of new blood vessels.

Hypoxia - inducible factor 1α (HIF - 1α), an oxygen sensor that controls the expression of various genes for glucose metabolism under conditions of low oxygen levels, is the most well documented substrate of the von Hippel - Lindau (VHL) enzyme.

This approach allowed them to create a metabolic index for each species, which sets clearly defined limits for oxygen - breathing sea life: «Marine animals like eelpouts, rock crabs and Atlantic cod can only survive in environments with enough oxygen for them to increase their metabolism to between two and five times their resting metabolic rate if need be.

«The enzyme makes sure that the muscles can use a more effective oxygen - based metabolism for as long as possible and then promotes a very quick transition to anaerobic metabolism.»

Thus, chlorination can have important ecological functions related to the activity of microorganisms, such as dealing with dangerous oxygen radicals that form during metabolism, or being part of the chemical battle that the microorganisms are engaged in, in the competition for resources in the ground.

For example, when there is no oxygen (a likely scenario in underground disposal vaults) to help these bacteria «breath» and break down the ISA, these simple single - cell microorganisms are able to switch their metabolism to breath using other chemicals in the water, such as nitrate or iron.

Crocodiles use a waste product of metabolism — the bicarbonate ions formed when carbon dioxide dissolves in water — as the trigger for haemoglobin to unload the oxygen it carries.

This efficient system of oxygen allocation, combined with the reptile's very slow metabolism, explains how it is able to survive underwater for so long.

The team, led by Professor Greg Cook, found that in such conditions the bacterium is able to quickly switch its cellular metabolism from a primarily oxygen - based one over to one that uses fermentation for energy production instead.

Life as we know it requires three primary ingredients: liquid water; a source of energy for metabolism; and the right chemical ingredients, primarily carbon, hydrogen, nitrogen, oxygen, phosphorus and sulfur.

When their waters get warmer, their metabolism accelerates and they need more oxygen to sustain their body functions,» said William Cheung, co-author of the study, associate professor at the Institute for the Ocean and Fisheries and director of science for the Nippon Foundation - UBC Nereus Program.

Aerobic metabolism utilizes oxygen and creates far more energy for hungry muscles, but it's slow.

Hemodynamic changes after visual stimulation and breath holding provide evidence for an uncoupling of cerebral blood flow and volume from oxygen metabolism.

Regardless of when the cyanobacteria appeared, it is widely accepted that they comprised the predominant form of life on early earth for some two billion years, and were responsible for the creation of earth's atmospheric oxygen, consuming CO2 and releasing O2 by photosynthetic metabolism.

HIIT has also been shown to boost metabolism for up to 36 hours post-workout with the body's excess post-exercise oxygen consumption (EPOC) working hard to return the body to homeostasis (read: the norm).

Iron is useful for participating in lots of different chemical reactions in the body, as part of normal metabolism, but it's also important for caring oxygen to our tissues and oxygen is what we need in a process called oxidative phosphorylation, which gives us energy.

Starting way back in 1931 (when Otto Warburg was awarded his Nobel Prize for research on the link between cancer and adequate oxygen to the cells), there has been a growing body of evidence indicating the link between immune deficiency and reduced oxygen metabolism.

More technically speaking, following a bout of high intensity exercise, you'll create a heighted degree of excess post-exercise oxygen consumption (EPOC), which revs up your metabolism for many hours afterwards.

More than one hundred vitamin B6 - dependent enzymes have been identified, mostly involved in amino acid metabolism: for oxygen transport via hemoglobin synthesis; in blood sugar regulation via conversion of stored carbohydrate to energy; in the development of the myelin sheath surrounding nerve cells; in the conversion of alphalinoleic acid to the essential long - chain fatty acid DHA; 28 and in the synthesis of neurotransmitters, phospholipids and sphingolipids, the vitamin niacin from tryptophan, and other vital metabolites.5 In addition to its role in enzyme reactions, B6 appears to moderate the action of some steroid hormones such as the glucocorticoid hormones, which in turn influence the metabolism of protein, carbohydrate and lipids.5, 9 B6 also is a potent antioxidant, rivaling carotenoids and vitamin E in its ability to quench reactive oxidants in the body.29

Athletes and hard exercisers may have low body B12 status, due to increased metabolism and demand for this vitamin to repair damaged blood cells and injured muscle tissue, and to carry more oxygen around in the blood while exercising.

And we also need triiron for thyroid activation from T4 and T3, and we need it for just generally carrying oxygen to ourselves, which is really important for cellular metabolism.

In fact, protein is vital for practically every process that occurs within the body such as metabolism, digestion and the transportation of nutrients and oxygen in the blood.

For instance, if you're sitting still reading this article, you're probably burning mostly fats through the oxygen - requiring, slow - energy - producing, aerobic metabolism.

Putting a thick, heavy cream on your face at night literally suffocates your skin, blocking the flow of oxygen that is vital for the metabolism and elimination of toxins from your pores.

We want a solution that not only burns a great number of calories during the training session, but also increases our metabolism for many hours AFTER the workout (EPOC, Excess Post-Exercise Oxygen Consumption).

They are great for the emotional health, releasing endorphins that make us happy and they are also great for our physical health, driving oxygen to all the cells throughout the body, building muscle, boosting metabolism and helping the body to function better on many different levels.

Fat oxidation on the other hand occurs in the oxygen CO2 exchange ratio of 1:0.7 so not only do you produce less CO2 as a by product which allows for slower breathing, but you also aren't dealing with the ever increasingly acidic environment created by glucose metabolism.

It should come as no surprise that if chronic under - eating can lower the amount of oxygen we use, and hence lower metabolism, then eating more food for such individuals could increase metabolism.

If exercising at a low intensity (or below 50 percent of max heart rate), you have enough stored fat to fuel activity for hours or even days as long as there is sufficient oxygen to allow fat metabolism to occur.

The role of your metabolism is to take the oxygen we breathe and the food we eat and process it to make energy, the fuel for life.

The problem is that at heart rates higher than MAF, energy needs outpace both (1) the breakdown and use of fats for energy and (2) the intake and transport of oxygen, meaning that the body has no choice but to engage anaerobic channels — not of a particular muscle to fuel that particular muscle, but of the muscles across the body as a whole, in order to feed the metabolism's additional energy needs.

These two conventional nutrients play a critical role in our body's regulation of oxygen metabolism, and they are widely known for their role in helping lower our risk of oxidative stress.

After a full - body resistance - based alternation - style workout session, you'll spike your metabolism for 2 - 3 days as your body re-uptakes the lactic acid from your bloodstream, restores oxygen to the muscle tissue you've worked, and repairs the micro-tears that took place in your muscles.

These exercises are sure to get the heart rate up, and keep your metabolism rocking for the next 12 - 24 hours after exercise due to the increased oxygen demand that is called EPOC (Exercise Post Oxygen Consumpoxygen demand that is called EPOC (Exercise Post Oxygen ConsumpOxygen Consumption).

Oxygen is an essential element for life, and every cell in your body requires it for cellular metabolism.

For example, KBs were recently reported to act as neuroprotective agents by raising ATP levels and reducing the production of reactive oxygen species in neurological tissues, 80 together with increased mitochondrial biogenesis, which may help to enhance the regulation of synaptic function.80 Moreover, the increased synthesis of polyunsaturated fatty acids stimulated by a KD may have a role in the regulation of neuronal membrane excitability: it has been demonstrated, for example, that polyunsaturated fatty acids modulate the excitability of neurons by blocking voltage-gated sodium channels.81 Another possibility is that by reducing glucose metabolism, ketogenic diets may activate anticonvulsant mechanisms, as has been reported in a rat model.82 In addition, caloric restriction per se has been suggested to exert neuroprotective effects, including improved mitochondrial function, decreased oxidative stress and apoptosis, and inhibition of proinflammatory mediators, such as the cytokines tumour necrosis factor - α and interleukins.83 Although promising data have been collected (see below), at the present time the real clinical benefits of ketogenic diets in most neurological diseases remain largely speculative and uncertain, with the significant exception of its use in the treatment of convulsion diseasFor example, KBs were recently reported to act as neuroprotective agents by raising ATP levels and reducing the production of reactive oxygen species in neurological tissues, 80 together with increased mitochondrial biogenesis, which may help to enhance the regulation of synaptic function.80 Moreover, the increased synthesis of polyunsaturated fatty acids stimulated by a KD may have a role in the regulation of neuronal membrane excitability: it has been demonstrated, for example, that polyunsaturated fatty acids modulate the excitability of neurons by blocking voltage-gated sodium channels.81 Another possibility is that by reducing glucose metabolism, ketogenic diets may activate anticonvulsant mechanisms, as has been reported in a rat model.82 In addition, caloric restriction per se has been suggested to exert neuroprotective effects, including improved mitochondrial function, decreased oxidative stress and apoptosis, and inhibition of proinflammatory mediators, such as the cytokines tumour necrosis factor - α and interleukins.83 Although promising data have been collected (see below), at the present time the real clinical benefits of ketogenic diets in most neurological diseases remain largely speculative and uncertain, with the significant exception of its use in the treatment of convulsion diseasfor example, that polyunsaturated fatty acids modulate the excitability of neurons by blocking voltage-gated sodium channels.81 Another possibility is that by reducing glucose metabolism, ketogenic diets may activate anticonvulsant mechanisms, as has been reported in a rat model.82 In addition, caloric restriction per se has been suggested to exert neuroprotective effects, including improved mitochondrial function, decreased oxidative stress and apoptosis, and inhibition of proinflammatory mediators, such as the cytokines tumour necrosis factor - α and interleukins.83 Although promising data have been collected (see below), at the present time the real clinical benefits of ketogenic diets in most neurological diseases remain largely speculative and uncertain, with the significant exception of its use in the treatment of convulsion diseases.

This provides for the high metabolism of the corpus luteum, which consumes 2 to 6 times more oxygen per unit weight than the liver, kidney, or even the heart.

Iron is an integral component of hemoglobin, which transports oxygen from the lungs to all body cells, and is also part of key enzyme systems for energy production and metabolism.

ACT - activated clotting time (bleeding disorders) ACTH - adrenocorticotropic hormone (adrenal gland function) Ag - antigen test for proteins specific to a disease causing organism or virus Alb - albumin (liver, kidney and intestinal disorders) Alk - Phos, ALP alkaline phosphatase (liver and adrenal disorders) Allergy Testing intradermal or blood antibody test for allergen hypersensitivity ALT - alanine aminotransferase (liver disorder) Amyl - amylase enzyme — non specific (pancreatitis) ANA - antinuclear antibody (systemic lupus erythematosus) Anaplasmosis Anaplasma spp. (tick - borne rickettsial disease) APTT - activated partial thromboplastin time (blood clotting ability) AST - aspartate aminotransferase (muscle and liver disorders) Band band cell — type of white blood cell Baso basophil — type of white blood cell Bile Acids digestive acids produced in the liver and stored in the gall bladder (liver function) Bili bilirubin (bile pigment responsible for jaundice from liver disease or RBC destruction) BP - blood pressure measurement BUN - blood urea nitrogen (kidney and liver function) Bx biopsy C & S aerobic / anaerobic bacterial culture and antibiotic sensitivity test (infection, drug selection) Ca +2 calcium ion — unbound calcium (parathyroid gland function) CBC - complete blood count (all circulating cells) Chol cholesterol (liver, thyroid disorders) CK, CPK creatine [phospho] kinase (muscle disease, heart disease) Cl - chloride ion — unbound chloride (hydration, blood pH) CO2 - carbon dioxide (blood pH) Contrast Radiograph x-ray image using injected radiopaque contrast media Cortisol hormone produced by the adrenal glands (adrenal gland function) Coomb's anti- red blood cell antibody test (immune - mediated hemolytic anemia) Crea creatinine (kidney function) CRT - capillary refill time (blood pressure, tissue perfusion) DTM - dermatophyte test medium (ringworm — dermatophytosis) EEG - electroencephalogram (brain function, epilepsy) Ehrlichia Ehrlichia spp. (tick - borne rickettsial disease) EKG, ECG - electrok [c] ardiogram (electrical heart activity, heart arryhthmia) Eos eosinophil — type of white blood cell Fecal, flotation, direct intestinal parasite exam FeLV Feline Leukemia Virus test FIA Feline Infectious Anemia: aka Feline Hemotrophic Mycoplasma, Haemobartonella felis test FIV Feline Immunodeficiency Virus test Fluorescein Stain fluorescein stain uptake of cornea (corneal ulceration) fT4, fT4ed, freeT4ed thyroxine hormone unbound by protein measured by equilibrium dialysis (thyroid function) GGT gamma - glutamyltranferase (liver disorders) Glob globulin (liver, immune system) Glu blood or urine glucose (diabetes mellitus) Gran granulocytes — subgroup of white blood cells Hb, Hgb hemoglobin — iron rich protein bound to red blood cells that carries oxygen (anemia, red cell mass) HCO3 - bicarbonate ion (blood pH) HCT, PCV, MHCT hematocrit, packed - cell volume, microhematocrit (hemoconcentration, dehydration, anemia) K + potassium ion — unbound potassium (kidney disorders, adrenal gland disorders) Lipa lipase enzyme — non specific (pancreatitis) LYME Borrelia spp. (tick - borne rickettsial disease) Lymph lymphocyte — type of white blood cell MCHC mean corpuscular hemoglobin concentration (anemia, iron deficiency) MCV mean corpuscular volume — average red cell size (anemia, iron deficiency) Mg +2 magnesium ion — unbound magnesium (diabetes, parathyroid function, malnutrition) MHCT, HCT, PCV microhematocrit, hematocrit, packed - cell volume (hemoconcentration, dehydration, anemia) MIC minimum inhibitory concentration — part of the C&S that determines antimicrobial selection Mono monocyte — type of white blood cell MRI magnetic resonance imaging (advanced tissue imaging) Na + sodium ion — unbound sodium (dehydration, adrenal gland disease) nRBC nucleated red blood cell — immature red blood cell (bone marrow damage, lead toxicity) PCV, HCT, MHCT packed - cell volume, hematocrit, microhematocrit (hemoconcentration, dehydration, anemia) PE physical examination pH urine pH (urinary tract infection, urolithiasis) Phos phosphorus (kidney disorders, ketoacidosis, parathyroid function) PLI pancreatic lipase immunoreactivity (pancreatitis) PLT platelet — cells involved in clotting (bleeding disorders) PT prothrombin time (bleeding disorders) PTH parathyroid hormone, parathormone (parathyroid function) Radiograph x-ray image RBC red blood cell count (anemia) REL Rocky Mountain Spotted Fever / Ehrlichia / Lyme combination test Retic reticulocyte — immature red blood cell (regenerative vs. non-regenerative anemia) RMSF Rocky Mountain Spotted Fever SAP serum alkaline phosphatase (liver disorders) Schirmer Tear Test tear production test (keratoconjunctivitis sicca — dry eye,) Seg segmented neutrophil — type of white blood cell USG Urine specific gravity (urine concentration, kidney function) spec cPL specific canine pancreatic lipase (pancreatitis)-- replaces the PLI test spec fPL specific feline pancreatic lipase (pancreatitis)-- replaces the PLI test T4 thyroxine hormone — total (thyroid gland function) TLI trypsin - like immunoreactivity (exocrine pancreatic insufficiency) TP total protein (hydration, liver disorders) TPR temperature / pulse / respirations (physical exam vital signs) Trig triglycerides (fat metabolism, liver disorders) TSH thyroid stimulating hormone (thyroid gland function) UA urinalysis (kidney function, urinary tract infection, diabetes) Urine Cortisol - Crea Ratio urine cortisol - creatine ratio (screening test for adrenal gland disease) Urine Protein - Crea Ratio urine protein - creatinine ratio (kidney disorders) VWF VonWillebrands factor (bleeding disorder) WBC white blood cell count (infection, inflammation, bone marrow suppression)

This formula helps optimize oxygen metabolism for increased endurance.