High negative reactivity could diminish capacity to attend social cues, leading to misinterpretation and incorrect processing of social information (e.g. Hostile attribution bias; [101]-RRB-, with the risk
for psychopathology such as externalizing disorders [102].
Not exact matches
To regard the mentally diseased merely as «cases»
for psychopathology is a convenient way of escaping their message about the fragile condition of the mind as
such.
The delicate balance between the human microbiome and the development of
psychopathologies is particularly interesting given the ease with which the microbiome can be altered by external factors,
such as diet, 23 exposure to antimicrobials24, 25 or disrupted sleep patterns.26
For example, a link between antibiotic exposure and altered brain function is well evidenced by the psychiatric side - effects of antibiotics, which range from anxiety and panic to major depression, psychosis and delirium.1 A recent large population study reported that treatment with a single antibiotic course was associated with an increased risk for depression and anxiety, rising with multiple exposures.27 Bercik et al. 28 showed that oral administration of non-absorbable antimicrobials transiently altered the composition of the gut microbiota in adult mice and increased exploratory behaviour and hippocampal expression of brain - derived neurotrophic factor (BDNF), while intraperitoneal administration had no effect on behavio
For example, a link between antibiotic exposure and altered brain function is well evidenced by the psychiatric side - effects of antibiotics, which range from anxiety and panic to major depression, psychosis and delirium.1 A recent large population study reported that treatment with a single antibiotic course was associated with an increased risk
for depression and anxiety, rising with multiple exposures.27 Bercik et al. 28 showed that oral administration of non-absorbable antimicrobials transiently altered the composition of the gut microbiota in adult mice and increased exploratory behaviour and hippocampal expression of brain - derived neurotrophic factor (BDNF), while intraperitoneal administration had no effect on behavio
for depression and anxiety, rising with multiple exposures.27 Bercik et al. 28 showed that oral administration of non-absorbable antimicrobials transiently altered the composition of the gut microbiota in adult mice and increased exploratory behaviour and hippocampal expression of brain - derived neurotrophic factor (BDNF), while intraperitoneal administration had no effect on behaviour.
This greater risk of
psychopathology is also likely to extend to other phenotypes associated with the opioid and serotonin systems
for which there is less available data,
such as antisocial behavior and substance abuse.
Interpersonal psychotherapy (or perhaps another specialty therapy
such as CBT) should be recommended as the treatment of choice
for that subset of individuals with BED (30 % of the sample in this study) with low self - esteem and a high level of specific eating disorder
psychopathology.
Adolescence is characterized by major biological, psychological and social challenges and opportunities, where interaction between the individual and environment is intense, and developmental pathways are set in motion or become established.2 — 4 Furthermore, adolescent
psychopathology can have important consequences
for education, relationships and socioeconomic achievement in later life.5 — 7 These characteristics of adolescence do not only set high demands
for cohort studies aiming to capture the most salient aspects of developmental pathways, they also ensure a great gain in empirical knowledge and an invaluable source of information
for public health policy from
such studies.
Policy - makers and clinicians should work together to make services,
such as screenings
for pregnant women and mothers, readily available.6 Programs aimed at reducing disruptions to family functioning are one avenue
for decreasing children's risk
for psychopathology.
Rather fewer meet the diagnostic criteria
for research, which
for the oppositional defiant type of conduct disorder seen in younger children require at least four specific behaviours to be present.7 The early onset pattern — typically beginning at the age of 2 or 3 years — is associated with comorbid
psychopathology such as hyperactivity and emotional problems, language disorders, neuropsychological deficits
such as poor attention and lower IQ, high heritability, 8 and lifelong antisocial behaviour.9 In contrast, teenage onset antisocial behaviour is not associated with other disorders or neuropsychological deficits, is more environmentally determined than inherited, and tends not to persist into adulthood.9
Further, despite accumulated findings on parental socialization, and early childhood research that shows that teachers are engaging in emotion socialization behaviours, we know little about how teachers (or,
for that matter, peers or siblings) socialize children's EC.34 Research is also needed to discern possible indirect contributors to EC,
such as parental
psychopathology, divorce, poverty and child care quality.
There is important overlap between knowledge of individuals and knowledge of groups,
such as the role of individual
psychopathology in treatment,
for example.
The examination of subclinical depressive symptoms is important given that
such symptoms are associated with an increased risk
for future
psychopathology (e.g., depressive, anxious, and behavioral disorders) as well as a wide range of negative outcomes including academic deficiencies, interpersonal difficulties with peers and family, and impaired cognitive functioning (Avenevoli et al. 2008; Kessler and Walters 1998; Reinherz et al. 1993).
The second hypothesis
for this study was whether neonatal reactivity (rated from the NNNS and Cry scales) would predict later maternal ratings of infant temperament, and whether parenting stress and
psychopathology would moderate
such relations.
Of all the childhood
psychopathologies, antisocial and aggressive behaviour problems
such as Oppositional Defiant Disorder (ODD) and Conduct Disorder (CD) account
for the greatest cost to psychological, psychiatric and social services [1].
It would be advantageous
for the purposes of prevention of borderline personality disorder to recognize that many aspects of these childhood or adolescent
psychopathologies are actually trait - like,
such as impulsivity, affective instability, or hyper - aggression [38].
For example, various parental
psychopathology symptoms
such as depressed mood, anxiety, and antisocial traits have been related to children's internalizing problems
such as withdrawn behavior and externalizing problems
such as aggression (Breaux et al. 2013; Cummings et al. 2005; Papp et al. 2005).
For this research, we examined the role of maternal characteristics
such as maternal substance abuse history and maternal
psychopathology as potential moderators of longitudinal relations between neonatal behavioral characteristics and parenting stress.
A score placing the individual at or above 90th percentile is indicative of high risk
for the existence of
psychopathology such as major depression.
Anxiety disorders are among the most common mental disorders during childhood and adolescence, with a prevalence of 3 — 5 % in school - age children (6 — 12 years) and 10 — 19 % in adolescents (13 — 18 years); 1, 2 and the prevalence of anxiety disorders in this population tends to increase over time.3 Anxiety is the most common psychological symptom reported by children and adolescents; however, presentation varies with age as younger patients often report undifferentiated anxiety symptoms,
for example, muscle tension, headache, stomachache or angry outbursts.4 According to the standard diagnostic systems, there are various types of anxiety disorders,
for example, generalised anxiety disorder (GAD), social phobias (SOP), social anxiety disorder (SAD), panic disorder (PD), overanxious disorder, separation anxiety, post-traumatic stress disorder (PTSD), obsessive - compulsive disorder (OCD).5 Anxiety disorders in children and adolescents often occur with a number of comorbidities,
such as autism spectrum disorders, 6 depressive disorders, 7 conduct disorder, 8 substance abuse9 or suicide - related behaviour.10 Youths with anxiety disorders experience serious impairment in social functioning (eg, poor school achievement; relational problems with family members and peers).11, 12 Childhood and adolescent anxiety disorders can persist despite treatment, 1 and they are associated with later adult
psychopathology.13, 14
Positive forms of parenting are beneficial
for cognitive and social development, while negative parenting
such as punishment and low warmth are associated with disruptive behavior in children and increased risk
for psychopathology [11, 15].
This is surprising given the increased rates of mental disorders including anxiety, depression, and substance abuse in the parents of anxious children [22, 23], and evidence that parental
psychopathology places children at increased risk
for parent behaviour - related stressors,
such as interparental conflict [24].
For example, parent
psychopathology may increase the likelihood of parent - dependent chronic adversities
such as interpersonal conflict or vice versa.