A three - step, hierarchical regression analysis was performed to predict change in generalized anxiety
from cognitive vulnerabilities, sub-dimensions of psychological well - being, and their interaction (as well as T1 generalized anxiety).
Not exact matches
Physical punishment is associated with a range of mental health problems in children, youth and adults, including depression, unhappiness, anxiety, feelings of hopelessness, use of drugs and alcohol, and general psychological maladjustment.26 — 29 These relationships may be mediated by disruptions in parent — child attachment resulting
from pain inflicted by a caregiver, 30,31 by increased levels of cortisol32 or by chemical disruption of the brain's mechanism for regulating stress.33 Researchers are also finding that physical punishment is linked to slower
cognitive development and adversely affects academic achievement.34 These findings come
from large longitudinal studies that control for a wide range of potential confounders.35 Intriguing results are now emerging
from neuroimaging studies, which suggest that physical punishment may reduce the volume of the brain's grey matter in areas associated with performance on the Wechsler Adult Intelligence Scale, third edition (WAIS - III).36 In addition, physical punishment can cause alterations in the dopaminergic regions associated with
vulnerability to the abuse of drugs and alcohol.37
These depression and anxiety symptoms add to those
cognitive vulnerabilities, but can be treated,» says senior author and NAMS Board of Trustees President - Elect Pauline M. Maki, PhD,
from the University of Illinois at Chicago.
HD comprises several
cognitive and affective symptoms, as well as uncontrolled movement (chorea), which have been hypothesized to arise
from a preferential
vulnerability of indirect pathway spiny projection neurons (iSPNs) preceding dysfunction of the direct pathway SPNs (dSPNs) in the striatum.
- Directly
from the paper: «Systemic inflammation, blood - brain barrier
vulnerability and
cognitive / non-
cognitive symptoms in Alzheimer disease: relevance to pathogenesis and therapy ``:
The findings
from these studies converge on the theme that attachment theory has considerable utility in potentially extending and refining current
cognitive vulnerability models through a consideration of interpersonal context and the
cognitive mechanisms by which negative interpersonal experiences may confer increased risk to later anxious and depressive symptoms.
To improve our understanding of the development of depressive symptoms, future research could test hypotheses in which factors
from different levels interact, i.e., cognitions, genetics, environment, affect, negative life experiences, as suggested by the
cognitive vulnerability - transactional stress model (Hankin and Abramson 2001).
CATCH - IT includes 14 web modules that teach strategies
from behavioral activation,
cognitive behavioral therapy, and interpersonal psychotherapy to strengthen protective factors and reduce
vulnerability to depressive disorder.
We examined all publications
from the ROOTS study up to July 2015, selected those examining adolescent mental health, and classified them as investigating (a) childhood risk factors for adolescent depression, (b) genetic and
cognitive vulnerability to depression in adolescence, (c) genetic markers, childhood adversities, and neuroendophenotypes, (d) morning cortisol and depression, (e) physical activity and depression symptoms, and (f) the underlying structure of mental health in adolescence.
The current study examined
cognitive vulnerability models in relationship to depressive symptoms
from a developmental perspective.
Moderated multiple regression analysis predicting T2 GAD symptoms
from the interaction between
cognitive vulnerability and positive relationships