Sentences with phrase «glucose by rat»

In addition, the researchers observed that adiponectin regulated the production of glucose by rat liver cells — suggesting that the hormone helps suppress the release of sugar stores.

Not exact matches

Research published in the Journal of Agricultural and Food Chemistry found that buckwheat extract lowered blood glucose levels by 12 to 19 percent when added to the diets of diabetic rats.

The most promising chemical — sulforaphane, a naturally occurring compound found in cruciferous vegetables — tamped down glucose production by liver cells growing in culture, and shifted liver gene expression away from a diseased state in diabetic rats.

To find these patterns, researchers used to expose rats to an odor for 45 minutes — an unnaturally long time — then kill them and look for changes in the uptake by the olfactory bulb of a labeled form of glucose, which indicates neuronal activity.

To assess the effects of preconception alcohol use, Sarkar, with doctoral candidate Ali Al - Yasari, MS, and their colleagues, conducted a study, funded by the National Institutes of Health, in rats, whose basic processes of glucose function are similar to those in humans, Sarkar said.

min, in 4 AL, 18 AL, and 18 CR, respectively (P < 0.01 between all groups), and in 18 CR rats infused with insulin at similar rates as in the 18 AL (1.4 mU / kg / min) hepatic glucose production was decreased by 32 % (P < 0.

Hafizur RM, Kabir N, and Chishti S. Asparagus officinalis extract controls blood glucose by improving insulin secretion and β - cell function in streptozotocin - induced type 2 diabetic rats.

In rat studies, naringin also has the potential to retard as well as improve diabetic complications by improved glucose intolerance, plasma lipid concentrations, and liver mitochondrial dysfunction [6].

This paper, Effect of hypothalamic - pituitary - adrenal axis alterations on glucose and lipid metabolism in diabetic rats, reports that astragalus may improve HPA axis functioning and aid in the treatment of diabetes by lowering blood sugar and high levels of corticosterone (the rat version of cortisol).

Moreover, a decrease in the activity of plasma glutathione peroxidase observed in the glucose - fed rats was prevented by lipoic acid treatment.

Psychologists at Purdue University's Ingestive Behavior Research Center reported that relative to rats that ate yogurt sweetened with glucose (a simple sugar with 15 calories / teaspoon, the same as table sugar), rats given yogurt sweetened with zero - calorie saccharin later consumed more calories, gained more weight, put on more body fat, and didn't make up for it by cutting back later, all at levels of statistical significance.

For example, KBs were recently reported to act as neuroprotective agents by raising ATP levels and reducing the production of reactive oxygen species in neurological tissues, 80 together with increased mitochondrial biogenesis, which may help to enhance the regulation of synaptic function.80 Moreover, the increased synthesis of polyunsaturated fatty acids stimulated by a KD may have a role in the regulation of neuronal membrane excitability: it has been demonstrated, for example, that polyunsaturated fatty acids modulate the excitability of neurons by blocking voltage-gated sodium channels.81 Another possibility is that by reducing glucose metabolism, ketogenic diets may activate anticonvulsant mechanisms, as has been reported in a rat model.82 In addition, caloric restriction per se has been suggested to exert neuroprotective effects, including improved mitochondrial function, decreased oxidative stress and apoptosis, and inhibition of proinflammatory mediators, such as the cytokines tumour necrosis factor - α and interleukins.83 Although promising data have been collected (see below), at the present time the real clinical benefits of ketogenic diets in most neurological diseases remain largely speculative and uncertain, with the significant exception of its use in the treatment of convulsion diseases.