Sentences with phrase «glucose than your cells»

Insulin - sensitive cells that are exposed to insulin and sodium arsenic appear to take in less glucose than cells exposed only to insulin.

Not exact matches

Rather than being used by muscles for energy, the glucose is redirected to fat cells.

Glucose is the fuel for neurons, and the cells consume more glucose when they are active than when they are aGlucose is the fuel for neurons, and the cells consume more glucose when they are active than when they are aglucose when they are active than when they are at rest.

Rather than just providing fuel, exercise - induced autophagy appears to help cells fine - tune their glucose metabolism.

The hope is that 3BP specifically kills certain cancer cells — while leaving normal cells alone — because they rely more on glucose metabolism than on an alternative pathway called oxidative phosphorylation.

The metabolism of bone cells determines how much sugar they use; if the bone cells consume more sugar than normal, this can lower the glucose level in the blood.

In order to generate enough energy, the bone cells in our mice therefore take up much more glucose than normal.»

Healthy cells use most of their glucose «fuel» to produce energy, rather than for building components of new cells, such as fats and DNA.

«We found that colorectal cancer cells survived under the condition of glucose depletion, and their resistance to such conditions depended on genomic alterations rather than on KRAS mutation alone.

The researchers found that the Type I diabetic mice that received the modified insulin and blood cell system were able to significantly reduce blood glucose levels for more than two days.

Cancer cells tend to use glucose to make more cell «building blocks» than energy, and this is thought to help them to divide and grow.

When the researchers turned off SIRT1 genes in cultured cells, the cells secreted less than half as much glucose as unaltered cells.

Follow - up work in cell cultures by King's lab showed that this defensive role for PKC - delta is triggered by high levels of lipids rather than glucose.

«Rather than having damaged mitochondria, somehow these cells, when their glucose metabolism is activated, stimulate themselves to make new mitochondria, so the mitochondria actually work better,» King says.

Weir notes that blood glucose levels run higher in mice than in humans, so that in this way the transplanted cells were in an environment similar to that of an actual transplant.

The researchers went on to demonstrate that SHP - 1 is reduced in mouse vascular smooth muscle cells primarily by the high levels of lipids in the blood associated with diabetes and related conditions, rather than the high levels of glucose also present in those conditions.

When developing heart cells are exposed to high levels of glucose, the researchers found, the cells generate more building blocks of DNA than usual, which leads the cells to continue reproducing rather than mature.

Human heart cells grown from stem cells show less - robust muscle fibers (green) in the presence of high levels of glucose (left) than when glucose levels were lower (right).

Type 2 diabetic cells exposed to 1 μmol / l insulin expressed a basal glucose transport activity similar to corresponding control cultures and significantly greater than the basal glucose transport activity seen under the other insulin conditions (P < 0.05).

In cells with high glucose levels, the pentose phosphate pathway made more nucleotides than usual.

Studies have shown that high - intensity cardio sessions that last more than 30 minutes are the most effective at increasing the uptake of glucose into cells.

In this way, insulin sensitivity is defined by how much insulin is needed to store blood glucose within the cells of the body — healthy people need a much smaller amount of insulin to store a certain amount of glucose than insulin resistant individuals, and the latter have higher levels of both blood glucose and insulin.

«If we're consuming carbohydrates at a faster rate than our bodies are utilizing them for energy, that extra glucose gets stored in the fat cells of the liver, which decreases its ability to break down excess estrogen and allowing it to hang around in our systems longer than it should.

Cancer cells have more glucose receptors than healthy cells so they take in more sugar than healthy cells.

So, if I understand correctly, what you're saying is that when your body feels as though it's constantly stressed out, whether from exercise or some other stressor, what can happen is that it switches on pathways to develop insulin resistance so that, rather than putting food stuff into, say, muscle storage or liver storage, you might actually create new fat cells or put glucose, you know, that has been converted into triglycerides, et cetera, into fat cells so that your body has storage to rely upon in times of need even though you're not necessarily in a time of need.

Isoleucine can be seen as the BCAA which mediates glucose uptake (into a cell) and breakdown (into energy) to a larger degree than other amino acids and may serve a role as a hypoglycemic (in diabetics) or as a performance enhancer (if taken preworkout in a carbohydrate replete state).

And when these cells are exposed to both glucose and fructose, they secrete more insulin than they do when exposed to glucose alone.

As a result, one could argue that things would run the opposite way than Adele proposes: reducing dietary glucose, which generally does not reduce blood glucose levels, will not affect cancer metabolism, but will limit availability of glucose to normal cells for structural use.

If glucose can not be stored or transported into cell than you get hyperglycemia and the resulting hyperinsulinemia creates hypertriglyceridemia.

On high - carb diets, cells / tissues utilize more glucose than in the evolutionarily favored state.

Insulin also plays a key role in fat storage: when insulin levels rise, our cells are forced to burn glucose rather than fat.

So, even though a cancer cell has far more glucose than a normal cell, less of this glucose gets inside its mitochondria than in a normal cell.

Many cells function better on ketones than glucose, because ketones take less effort to get into the mitochondria (which are our furnaces) than glucose does.

Cancer cells have more glucose receptors than normal cells and thus are attracted to honey.

Cancer cells have more glucose receptors than normal cells and 15 times more glucose than normal cells, though the microbes intercept most of the glucose.

Isoleucine is also known to increase glucose uptake in the cell and works to break it down into energy at a larger degree than any other amino acid.

As glucose enters the cells faster than it can be used for energy, it accumulates inside the cell.

Cancer cells consume 15 times more glucose than a normal cell.

Glucose oscillations, more than constant high glucose, induce p53 activation and a metabolic memory in human endotheliaGlucose oscillations, more than constant high glucose, induce p53 activation and a metabolic memory in human endotheliaglucose, induce p53 activation and a metabolic memory in human endothelial cells

The liver releases the glucose and cancer cells are likely to pick up this glucose because cancer cells consume about 15 times more glucose than normal cells.

Chlorophyll, the pigment that makes plants green, acts as a blood cleanser, antioxidant, anti-inflammatory and detoxifying pigment that heals cell walls, oxygenates the blood, absorbs carcinogens and provides a fuel source cleaner than glucose.

Rather than just building our muscles and fat stores or forcing glucose into the cells, insulin also works to keep fat and muscle in storage.

Glucose is replenished much faster than glycogen, but as it circulates the blood, it must be moved into the muscle cells through an insulin mediated process in order to be used.

Cancer cells love glucose and actually consume 10 to 20 times more glucose than normal healthy cells.

«Cancer cells behave differently than normal cells in terms of glucose metabolism,» said Wisner.