Not exact matches
Glycation leads to deleterious chemical complexes called AGEs (advanced glycation end products) that result from common but undesirable reactions between blood sugars, such as glucose, and proteins in many parts of our bodies, including the lenses of
Glycation leads to deleterious chemical complexes called AGEs (advanced
glycation end products) that result from common but undesirable reactions between blood sugars, such as glucose, and proteins in many parts of our bodies, including the lenses of
glycation end products) that result
from common but undesirable reactions between blood sugars, such as glucose, and proteins in many parts of our bodies, including the lenses of our eyes.
so 80's, people drop dead
from oxidised cholesterol, mediated by too much omega 6 plant oil in the diet, and
from weak artery walls caused by
glycation and calcification, google vitamin K2
Diabetics, for example, may suffer
from poor bones, not because of low mineral density but because their collagen is damaged by the advanced
glycation end products (AGEs) created when blood sugar levels are chronically high.
This has several positive effects, here's a few: (a) because blood sugar levels are low, there won't be any further damage to cells
from Glycation (high blood sugar kind of «fries» a cell, like a grilled cheese sandwich — literally.)
When faced with these daily stressors the PUFAs would be a detriment to all aspects of defending the brain / body
from said stressors as they disrupt many vital hormonal signalling pathways, damage lipoproteins, make us more sensitive to sunlight, accelerate
glycation, and less resistant to social stress.
In fact, these are the same toxins found in French fries, the ones that make them really really bad for us (aside
from excess calories and rancid unsaturated oils), known as Advanced
Glycation End products (AGEs).
From the ratio of prevalence of the acyclic to cyclic form of monosaccharide results the relative reactivity for AGE production, because only the acyclic forms of sugar participate in the
glycation process.»
Dietary advanced
glycation endproducts
from browned meats (and sadly, also toast) accelerate the Maillard reaction in vivo by depleting our AGE receptor (PMID: 22908267), while numerous phytochemicals appear to interrupt
glycation (see Odjakova et al..
Most effects of diabetes seem to result
from acceleration of
glycation, and anti-diabetic pharmaceuticals are one of the most promising «gerosuppressants».
Extra padding in the abdominal region prevents the body
from properly utilizing insulin; as a result glucose begins to build up in the blood, which causes
glycation - damage to internal organs and blood vessels.
Complications
from diabetes are stimulated by the accumulation of advanced
glycation end - products (AGEs), inflammation, and oxidative stress.
Some proponents also suggest that benfotiamine can shield the body
from the harmful effects of advanced
glycation end products (AGEs).
I think that was Dr. McCauley said, any of the advanced
glycation end - products that you get
from the charring aspect that you just eat a whole bunch of vegetables and ad some of those herbs on there like you mentioned.
... and many studies have been found that show that, at least for a short period of time, damage
from glycation and other adverse molecular events occur with far lower blood glucose.
Many of the complications of diabetes come
from advanced
glycation endproducts (AGE), which occur when glucose cross links with proteins in the body, a process closely associated with the formation of free radicals.
In addition to Methionine promoting VEGF with Methionine coming primarily
from meat, meat is also the primary dietary source of advanced
glycation end products which have also been shown to facilitate VEGF.