Cosmesis Skin Care Advanced Anti-Glycation Peptide Serum enhances collagen production, reduces wrinkles, hydrates skin, and overall slows the visible signs of aging by reducing harmful
glycation levels in the body.
Not exact matches
Sugar has no nutritive value and raises blood glucose
levels, stimulating the release of insulin as well as the formation of advanced
glycation end products (AGE's) which can damage skin collagen and lead to wrinkles, among many other issues.
Elevated blood sugar
levels also accelerate a natural process called
glycation, in which glucose attaches to proteins in the body and makes them rigid.
Glycation can be determined by a test for hemoglobin A1c, which is a marker of long - term blood sugar
levels and how the body processes sugar.20
Sebekova K, Krajcoviova - Kudlackova M, Schinzel R, Faist V, Klvanova J, Heidland A. Plasma
levels of advanced
glycation end products in healthy, long - term vegetarians and subjects on a western mixed diet.
Diabetics, for example, may suffer from poor bones, not because of low mineral density but because their collagen is damaged by the advanced
glycation end products (AGEs) created when blood sugar
levels are chronically high.
Overeating also contributes to higher blood sugar
levels and can promote the premature aging of your face — although it's unclear why, there's some evidence that high blood sugar
levels contribute to the formation of substances called AGES (advanced
glycation end products)-- these substances may prevent the efficient repair of collagen and even harm the heart and kidneys and promote disease.
This has several positive effects, here's a few: (a) because blood sugar
levels are low, there won't be any further damage to cells from
Glycation (high blood sugar kind of «fries» a cell, like a grilled cheese sandwich — literally.)
Diagnostic utility of serum or cerebrospinal fluid
levels of toxic advanced
glycation end - products (TAGE) in early detection of Alzheimer's disease.
Glycation is a factor of glucose concentration exposure and time, with more AGEs forming upon longer exposure to higher concentrations of glucose.33 It follows that in a body that is hyperinsulinemic, and a brain that is insulin - resistant, the peripheral hyperinsulinism will inhibit the clearance of soluble Aβ by IDE, thereby causing it to remain in the extracellular space for an extended amount of time, and the functional «hyperglycemia» in the brain will provide an elevated level of glucose — the perfect storm for glycation of Aβ and its aggregation into insoluble
Glycation is a factor of glucose concentration exposure and time, with more AGEs forming upon longer exposure to higher concentrations of glucose.33 It follows that in a body that is hyperinsulinemic, and a brain that is insulin - resistant, the peripheral hyperinsulinism will inhibit the clearance of soluble Aβ by IDE, thereby causing it to remain in the extracellular space for an extended amount of time, and the functional «hyperglycemia» in the brain will provide an elevated
level of glucose — the perfect storm for
glycation of Aβ and its aggregation into insoluble
glycation of Aβ and its aggregation into insoluble plaques.
• EXOGENOUS INSULIN: Although insulin has been shown to improve memory and cognition acutely, chronically high insulin
levels are known to impair brain function.42, 64 Exogenous insulin would serve to inhibit IDE more strongly, thereby preventing the clearance of Aβ, causing it to linger in the brain interstitial fluid even longer, where it is subject to
glycation and oxidation.
I wonder if this might be a result of lower
levels of taurine, a potent
glycation inhibitor.
A HgA1C test looks at the
level of
glycation (indicative of blood sugar
levels) in the red blood cell over a 90 - 120 day period.
And remember that yet another benefit of maintaining lower blood sugar
levels is that you help to SLOW the aging process, since high blood sugar
levels over time increase
glycation in your body, which ages you faster.
Maintaining lower and more stable blood sugar
levels also helps to prevent cravings and slow the aging process by reducing formation of advanced
glycation end products (AGEs) in your body.
Most importantly, high blood sugar
levels generate free radicals called advanced
glycation end products (AGEs), formed when proteins react with glucose and become useless.
It was reported recently that urine
levels of pentosidine (i.e., an advanced
glycation end product formed by glycosylation) is associated with the activity of rheumatoid arthritis.
Jaminet has admitted as much by stating below, «the number of
glycation reactions may be proportional to the concentration of glucose, and if
glycation products are health damaging toxins then toxicity may be proportional to glucose
levels..»
However, when blood sugar spikes send glucose to toxic
levels and all the other pathways are at their rate limit,
glycation is an important contributor in managing the immediate crisis of bringing down blood sugar.
This view makes sense as a matter of molecular chemistry: the number of
glycation reactions may be proportional to the concentration of glucose, and if
glycation products are health damaging toxins then toxicity may be proportional to glucose
levels.
«the number of
glycation reactions may be proportional to the concentration of glucose, and if
glycation products are health damaging toxins then toxicity may be proportional to glucose
levels..»
As a bonus, olive leaf extract protects against the damage of high blood sugar — against advanced
glycation end products (AGEs), free radicals which form when elevated blood glucose
levels react with proteins and run wild.