Sentences with phrase «gondii infection»

One problem: the island where the researchers found the greatest seroprevalence of T. gondii infection among the birds, Molokai, just so happens to be home to perhaps the most dramatic increase in their numbers in recent years.

«These findings may give further clues about how T. gondii infection can possibly [alter] the risk of specific psychiatric disorders.»

Toxoplasma gondii (I) While it's true that cats are the definitive host of Toxoplasma gondii, it's important to note that «wild game can be a source of T. gondii infection in humans, cats, and other carnivores.

The findings reveal an association between Toxoplasma gondii infection in seniors subject to a «test battery for measuring memory functions» [1] and certain of those memory functions.

In a second recent study, A.L. Sutterland from the Department of Psychiatry at the Academic Medical Centre in Amsterdam and colleagues analyzed the findings of 50 published studies to confirm that T. gondii infection is associated with mental disorders.

The low frequency of infected meat suggests that cats potentially were a cause for much T. gondii infection in the United States.»

Results indicated no increased risk of aberrant mental conditions associated with T. gondii infection.

Studies showing correlative links between Toxoplasma gondii infection and potential increases in schizophrenia, suicide attempts and poor impulse control have often been conducted on small clinical or convenience samples, rather than large populations.

Approximately 28 % of the population tested positive for T. gondii infection.

Some previous studies have suggested a link of some sort between childhood Toxoplasma gondii infection and schizophrenia later in life.

Maccari ME, Scarselli A, Di Cesare S, Floris M, Angius A, Deodati A, Chiriaco M, Cambiaso P, Corrente S, Colafati GS, Utz PJ, Angelini F, Fierabracci A, Aiuti A, Carsetti R, Rosenberg JM, Cappa M, Rossi P, Bacchetta R, Cancrini C. Severe toxoplasma gondii infection in a member of a NFKB2 - deficient family with T and B cell dysfunction.

Taken together, our studies suggest that permanent interruption of mouse innate aversion to feline urine is a general trait of T. gondii infection that occurs within the first three weeks, independent of parasite persistence and ongoing brain inflammation.

They were more likely to show signs T. gondii infection and had higher cholesterol, although it is unclear if the latter has the same consequences in otters as it does in humans.

In fact, Grigg and his colleagues found that «T. gondii infections peaked in 2007 then declined relative to S. neurona.»

Not exact matches

The biggest risk is toxoplasmosis which is an infection you can get from a microscopic parasite called Toxoplasma gondii.

The study adds to a growing body of evidence suggesting that toxoplasmosis — usually a mild or nonsymptomatic infection from a protozoan parasite called Toxoplasma gondii — may somehow alter people's brain chemistry to cause long - term behavior problems.

Using mouse models Dr Grainger and his team looked at how and where monocytes are programs in response to toxoplasmosis, an infection caused by a common parasite called Toxoplasma gondii.

This defect is linked to infection by the Toxoplasma gondii parasite.

While earlier studies showed that mice lose their fear of bobcat urine for a few weeks after infection, Ingram showed that the three most common strains of Toxoplasma gondii make mice less fearful of cats for at least four months.

The researchers sought to explore the link between maternal infection and risk for autism, focusing on five pathogens known collectively as ToRCH agents — Toxoplasma gondii, rubella virus, cytomegalovirus, and herpes simplex viruses type 1 and 2 — to which exposure during pregnancy can lead to miscarriage and birth defects.

Toxoplasma gondii is the most common parasitic infection worldwide.

Combined with previously published studies, our data indicate that infection with all three major North American T. gondii clonal lineages results in loss of innate, hard - wired aversion to feline predator urine in mice.

We assayed blood serum for antibodies specific for T. gondii using an enzyme - linked immunosorbent assay (ELISA) and confirmed that both attenuated Type I and Type III parasites had established infection in our mice (Figure 2C).

To begin exploring these questions, we evaluated the effects of infection with two previously uninvestigated isolates from the three major North American clonal lineages of T. gondii, Type III and an attenuated strain of Type I. Using an hour - long open field activity assay optimized for this purpose, we measured mouse aversion toward predator and non-predator urines.

This suggests that T. gondii - mediated interruption of mouse innate aversion toward cat urine may occur during early acute infection in a permanent manner, not requiring persistence of parasite cysts or continuing brain inflammation.

It is possible that T. gondii causes a permanent change in the brain during acute infection, thereby not requiring continued parasite presence and an ongoing immune response.

Lafferty started out believing from review of previous published literature that «infectious disease was preventing the recovery of sea otters, and the most likely source of infection of Toxoplasma gondii was terrestrial runoff containing oocysts defecated by cats,» but when he and colleagues tagged 135 California sea otters in 2009 and followed them for four years, they found that «Counter to expectations, sea otters from unpopulated stretches of coastline,» around Big Sur, are less healthy and more exposed to parasites than city - associated otters,» from the Monterey Bay area, who have more exposure to fecal matter from cats.

As the alleged evidence that feral cats are a major threat to wild bird populations evaporates, ailurophobes have amplified claims that free - roaming cats are to blame for outbreaks of the parasitic protozoan infection Toxoplasma gondii that were first found to afflict California sea otters in 2002,» causing about 17 % of identified deaths, and have killed at least eight Hawaiian monk seals in 2015 alone.

This infection is caused by the Toxoplasma gondii parasite.

Toxoplasma gondii is present as a chronic infection in more than 60 million people.

Melissa Miller, Wildlife Pathologist, California Department of Fish and Game Miller was one of 14 co-authors to link the Type X strain of T. gondii — responsible for nearly three - quarters of sea otter infections, according to one study [1]-- to wild felids (e.g., mountain lions and bobcats) rather than domestic cats.

While cats are the definitive host for T. gondii, research has shown that contact with cats is not a risk factor for infection and even vets are no more likely to be infected with the parasite than non-vets.

Toxoplasmosis infection is caused by a parasite called toxoplasma gondii.

In other words, there's no evidence that infection with t. gondii causes behavioral illness in humans.

«In addition to live prey, eviscerated tissues (gut piles) from hunted deer and black bears would be a source of infection for wild cats... Prevalence of T. gondii in wild game and venison in the USA is very high and hunters need to be aware of the risk of transmission of infection to humans and, more importantly, spread of infection in the environment.

Outdoor cats have an increased likelihood of infection with T. gondii.

But in those with weaker immune systems, infection with T. gondii can cause an illness called toxoplasmosis, which can result in miscarriages, fetal development disorders, weeks of flu - like illness, blindness and even death.

One of the world's most common parasitic infections, Toxoplasma gondii infects approximately 1 out of 3 humans.

The study's lead author explained: «We can't say with certainty that T. gondii caused the women to try to kill themselves, but we did find a predictive association between the infection and suicide attempts later in life that warrants additional studies.»

She gave birth prematurely due to toxoplasmosis infection — the moose had browsed an area contaminated by «community» cats and its tissues were infected with Toxoplasma gondii oocysts.

At the very least, the low infection rates found in feral cats — combined with the much higher rates in bobcats and pumas — raise serious questions about domestic cats» role in environmental contamination of T. gondii.

Although the authors noted that their study did not provide evidence for great risk associated with T. gondii in cats, they advised that the risk should still be considered, as the infection in humans can cause significant health problems, and cats who are not otherwise transmitting the infection can begin shedding the virus in times of stress.