Myc is a proto - oncogene or master cancer gene that spurs tumor
growth in a variety of cancers including breast, lung, colon, brain, skin, leukemia, prostate, pancreas, stomach and bladder.
Not exact matches
«Next steps are to further explore this possibility
in human trials
in order to assess if it will help patients, but these two drugs make sense from a
variety of studies and we find that they act together through multiple mechanisms to control
cancer growth in the laboratory.»
miR - 184 is known to suppress tumor development by regulating a
variety of genes involved
in cancer growth, while SND1 has been shown to play a significant role
in the development
of breast, colon, prostate and liver
cancers.
Scientists have known for years that collagen remodeling plays an important role
in a wide
variety of biological processes ranging from wound healing to
cancer growth.
The disease retards
growth and causes bone defects
in children and makes them susceptible to a
variety of cancers.
However, studies have also shown that Minocycline is capable
of inducing modest
cancer cell death and
growth inhibition
in a
variety of cancer types.
In a previous study, Lonard and co-senior study author Bert O'Malley of Baylor College of Medicine screened a large number of compounds to identify SRC - inhibiting molecules that kill a wide variety of cancer cells and inhibit tumor growth in animal model
In a previous study, Lonard and co-senior study author Bert O'Malley
of Baylor College
of Medicine screened a large number
of compounds to identify SRC - inhibiting molecules that kill a wide
variety of cancer cells and inhibit tumor
growth in animal model
in animal models.
Important features
of XMRV biology include (1) tropism for a
variety of cell lines, including prostate
cancer DU145 and LNCaP cells [27], [43], [48], and human neural cell types [57], (2) adaptations that promote
growth in prostate epithelium and human - derived prostate
cancer cell lines including an androgen response element
in the promoter region [58] and downregulation
of APOBEC3G [59], and (3) cellular effects with potential oncogenic properties including increased tumor aggressiveness mediated by downregulation
of p27 [60] and differential regulation
of several microRNAs [61].