This work identifies novel steps in disease progression that could potentially be targeted by drugs to
halt cognitive decline.
Not exact matches
UT Southwestern Medical Center researchers have found that
halting production of new neurons in the brain following traumatic brain injury can help reduce resulting epileptic seizures,
cognitive decline, and impaired memory.
The operationalization of mild
cognitive impairment (MCI) led to targeting earlier symptomatic cases of the illness and treatment strategies based less on pathology and more on a chance to
halt or slow
decline than there would be earlier in the disease.1 With the development of amyloid imaging, MCI due to AD diagnosis was refined, 2 and early - stage AD was extended further to include preclinical AD, 3 wherein a positive amyloid positron emission tomography (PET) scan or diagnostic low levels of cerebrospinal fluid β - amyloid (Aβ) indicated the presence of pathology in people who were cognitively normal.
This way you can easily stack the deck in your favor by
halting or reversing the
decline in
cognitive function that leads to such memory lapses as forgetting common words or the inability to recall the names of familiar faces.