The researcher below brings up the point that the children from the well - fed families were eating a lot of peanut butter with
high aflatoxin amounts (he says could have initiated the cancer).
On the point about extremely
high aflatoxin to promote cancer, is it not a greater testament to the plant based diet that it was able to stop the cancer growth?
In countries with
high aflatoxin exposure, like China and parts of Africa, fewer than 1 in 1000 people develop liver cancer.
Not exact matches
I'm always skeptical because I know peanuts can have a
high mold /
aflatoxin content, and most companies just balance out the peanuts with
high mold into enough non-mold peanuts so they can get below the «legal» limit....
«Every time we measure
aflatoxin exposure in humans, in children and adults [in Africa], there's always very
high positive rates,» says Yun Yun Gong, a food toxicologist at Queen's University Belfast.
Heat and humidity — prime contamination conditions — run rampant, and so the risk of
aflatoxin developing remains
high.
For protein, the children depended largely on peanuts, which in the tropics often contain relatively
high amounts of
aflatoxin, a carcinogen produced by mold growth.
Crops like corn and groundnuts are more susceptible to
aflatoxin - producing mold when their immune systems are weakened by conditions like
high temperature and drought stress.
The problem is, in developing countries that limit is much harder to enforce, meaning that
aflatoxin levels could soar as
high as 100,000 ppb.
The National Cancer Institute has stated that exposure to
aflatoxins can cause an increased risk of liver cancer, and in my practice, I've found a diet
high in
aflatoxins contribute to
higher levels of inflammation.
I mean it's known to be
higher in
aflatoxin, it's a legume as well.
Pistachios are at
high risk of contamination by
aflatoxin and may be bleached or fumigated during processing; choose organic pistachios and avoid those that are dyed, bleached, or show signs of decay
This almost sounds like an admission that
high - protein diets promote detoxification of
aflatoxin.
We investigated this question by giving two groups of rats either a
high -
aflatoxin dose or a low -
aflatoxin dose, along with a standard baseline diet.
Then, during the promotion phase, we fed a low - protein diet to the
high -
aflatoxin dose groups and a
high - protein diet to the low -
aflatoxin dose group.
High doses of
aflatoxin, by contrast, would cause cancer in the monkeys gorging out on protein and just kill the others.
In these studies, a chronic low dose of
aflatoxin produced precancerous lesions in monkeys fed 5 % casein but not in those fed 20 % casein; a chronic medium dose saved the low - protein monkeys from precancerous lesions by — yup, you guessed it — killing them, while the
high - protein monkeys suffered neither plight; and a chronic
high dose was finally able to give the
high - protein monkeys pre-cancerous lesions.
Effect of
High and Low Dietary Protein on the Dosing and Postdosing Periods of
Aflatoxin B1 - induced Hepatic Preneoplastic Lesion Development in the Rat.
Animals starting with the most cancer initiation (
high -
aflatoxin dose) developed substantially less foci when fed the 5 % protein diet.
Large enough doses of
aflatoxin are a liver carcinogen in
high doses (it's actually what T. Colin Campbell used to induce liver cancer in mice during his China Study crusade to indict animal protein).
Aflatoxins are potent carcinogens in grains and are present in
high quantities in highly processed foods such as crackers, cookies, chips, and cereals.
Indeed, the researchers weren't pulling our legs: This study really did show that a low - protein diet was both more «cancer promoting» and more deadly than a
high - protein diet when the dose of
aflatoxin was lower.
Rats exposed to
aflatoxin while on a 5 % protein diet were far more likely to develop neoplasms than rats exposed to
aflatoxin on a
higher protein diet.
Monkeys on low protein diet [with 0.16 ppm
aflatoxin] surviving for 90 weeks or more show foci of preneoplastic lesions, whereas those on
high protein diet reveal no such alterations at the corresponding time interval.
The hepatic injury again is more accentuated in the low protein group as compared with the
high protein group [with 0.5 ppm
aflatoxin].
The rats on the low - casein diet were more vulnerable to acute
aflatoxin toxicity — they were more likely to die right away from
aflatoxin poisoning than the
high - casein rats.
So, eating a 100 gram portion of liver from this calf would give you 4.6 mg
aflatoxin, which is pretty
high.
1983 May; 43 (5): 2150 - 4 Effect of
high and low dietary protein on the dosing and postdosing periods of
aflatoxin B1 - induced hepatic preneoplastic lesion development in the rat)
Campbell and K. D. Mainigi fed rats
high - and low - protein diets with or without
aflatoxin.
One exception I think was the use in one study of 5ppm
aflatoxin for
high - casein and 2.5 ppm
aflatoxin for low - casein rather than just using 2.5 ppm for both groups so the low - casein rats wouldn't die.
Aflatoxin proved lethal to Fisher 344 rats fed low - protein diets when rats fed
high - protein diets were immune to the same doses (4).
* A note about pepper: Black pepper tends to be especially
high in mold toxins, particularly
aflatoxin and ochratoxin A.
More clues for understanding the casein - cancer research come from another Indian study — this one published in the late 1980s, and examining the effects of protein in
aflatoxin - exposed monkeys instead of rats.14 As with Campbell's experiments, the monkeys were fed diets containing either 5 percent or 20 percent casein, but with one important difference: instead of being slammed with an astronomically (and unrealistically)
high dose of
aflatoxin, the monkeys were exposed to lower, daily doses — mimicking a real - world situation where
aflatoxin is consumed frequently in small amounts from contaminated foods.
When the
aflatoxin dose is more moderate, animals eating a low - protein diet develop cancer while their
higher - protein counterparts remain in mighty fine health.
For instance, it would be unethical to dose a group of volunteers with
high - cholesterol foods to see if they had more heart attacks, or to feed them
high doses of the fungal poison
aflatoxin to determine whether they suffered more cases of liver cancer.
Grains that are used in pet foods are considered to be feed grade (instead of human grade), and have
higher allowable levels of toxins (mold - based toxins like
aflatoxin and vomitoxin).
Also, peanut butter has a
higher risk of toxins such as
aflatoxin.
Plant ingredients, often contaminated with
aflatoxin and other toxic molds, can be deficient in iron, zinc, selenium, magnesium, vitamin A and C, and lysine, among other essential nutrients and
high in phytoestrogens, endocrine disrupting agrichemicals, dioxins, and PCBs, the latter being a serious problem because of bioaccumulation in animal - derived food ingredients.
Peanuts can be quite
high in
aflatoxins.
High calcium levels, high vitamin D, low vitamin A, dietary protein levels and even aflatoxin ingestion can all slowly work together to damage your bird's kidn
High calcium levels,
high vitamin D, low vitamin A, dietary protein levels and even aflatoxin ingestion can all slowly work together to damage your bird's kidn
high vitamin D, low vitamin A, dietary protein levels and even
aflatoxin ingestion can all slowly work together to damage your bird's kidneys.
Maize, groundnuts (peanuts), pistachios, brazils, chillies, black pepper, dried fruit and figs are all known to be
high risk foods for
aflatoxin contamination, but the toxins have also been detected in many other commodities.