Sentences with phrase «high glucose concentrations»

Using a blood test to diagnose diabetes isn't always straightforward, because healthy cats under stress (as they may experience in a veterinary clinic) often have high glucose concentrations in their blood (called stress hyperglycemia).

«It's well known in IVF clinics that if you have high glucose concentrations in cell culture, it favours survival of male blastocysts,» says Mathews.

Even without an inductor, this mutated fungus always produces the desired enzymes and, unlike other strains of fungus, does not stop doing so once a high glucose concentration has been reached.

Not exact matches

Besides glucose from complex carbs, essential fatty acids from whole foods like avocados are the «good» fats that are absolutely essential from your brain to run at its optimum level, helping with concentration, memory and overall production of that happy serotonin we all want high levels of.

In the case of heart disease, Reaven says that high blood concentrations of insulin and glucose can damage the endothelium that lines coronary arteries and set the stage for the formation of plaques.

This brought about an improvement in blood sugar, whilst at the same time reducing so - called sugar spikes, that is phases with particularly high blood glucose concentrations.

Acute insulin stimulation increased glucose transport by 21 % in control cultures precultured at 1 pmol / l insulin, but at higher insulin concentrations we could not detect an effect of insulin on glucose uptake.

In accordance with other studies, we found an increased glucose uptake under acute insulin stimulation in cultures established from control subjects precultured under basal physiological insulin concentrations, but when precultured at a higher insulin concentration, we could not see an effect on acute insulin stimulation indicative of induced insulin resistance (10).

In contrast, type 2 diabetic cultures precultured at very high insulin concentrations expressed an increased basal glucose uptake and a reduced insulin stimulated uptake.

They had basal glucose concentrations (fasting blood glucose), similar to control animals, but higher concentrations at 30, 45, and 60 min post glucose injection (P < 0.001) and also at 120 min (P < 0.005).

After ingestion of the high - protein diet, the mean peak glucose concentration was 10.9 ± 0.7 mmol / L (196 ± 12 mg / dL) after the breakfast meal, 7.9 ± 0.6 mmol / L (142 ± 10 mg / dL) after the lunch meal, 9.2 ± 0.8 mmol / L (165 ± 14 mg / dL) after the dinner meal, and 8.6 ± 0.3 mmol / L (154 ± 6 mg / dL) after snack 2.

Thus, the glucose concentration was consistently lower after the high - protein diet, particularly in the evening.

Results: With the fasting glucose concentration used as a baseline from which to determine the area under the curve, the high - protein diet resulted in a 40 % decrease in the mean 24 - h integrated glucose area response.

Both GI - tables show that foods with high GI raise glucose concentration at a rapid rate.

The mean 24 - h integrated glucose area responses, with the fasting glucose concentration as baseline, were 34.1 ± 7.2 mmol · h / L (614 ± 130 mg · h / dL) and 21.0 ± 4.2 mmol · h / L (378 ± 75 mg · h / dL) after the control and high - protein diets, respectively.

Glycation is a factor of glucose concentration exposure and time, with more AGEs forming upon longer exposure to higher concentrations of glucose.33 It follows that in a body that is hyperinsulinemic, and a brain that is insulin - resistant, the peripheral hyperinsulinism will inhibit the clearance of soluble Aβ by IDE, thereby causing it to remain in the extracellular space for an extended amount of time, and the functional «hyperglycemia» in the brain will provide an elevated level of glucose — the perfect storm for glycation of Aβ and its aggregation into insoluble plaques.

High glucose and insulin concentrations in the bloodstream are not desirable because they promote fat storage and rebound hypoglycemia.

Now when I say sugar, what I am referring to is a high concentration of fructose + glucose, such as soft drinks, candies, sweets, baked goods, breakfast cereals (yes, I know, I said cereal!

The study found that ingestion of the very high molecular weight carbohydrate resulted in faster and greater increase in blood glucose and serum insulin concentration compared to the low molecular weight carb and the placebo.

In contrast, higher postprandial insulin concentrations and increased fat oxidation with breakfast skipping suggest the development of metabolic inflexibility in response to prolonged fasting that may in the long term lead to low - grade inflammation and impaired glucose homeostasis.

The addition of sacha inchi oil slowed down glucose production in 16 (38.1 %) participants that showed a higher concentration of triglycerides before meals (fasting hypertriglyceridemia), whereas an improved expression of sirtuin - 1 (SIRT1), a gene responsible for regulating energy within the cells and coordinating cellular functions, was registered four hours after ingesting the meal enriched with sacha inchi oil, and linked to lowered levels of blood sugar measured at the same time.

Mean (± SE) glucose concentrations during the test period after the high - glycemic - index (HGI; •), low - glycemic - index (LGI; ▪), and HGI with lactulose (HGI - Lac; ▴) breakfasts.

You need to have a high plasma glucose concentration and spiked serum insulin levels to drive maximum cellular creatine delivery.

Consuming a high glycemic food within 45 minutes after exercise elevates plasma glucose concentrations and facilitates muscle glycogen replenishment.

To induce nutritional ketosis, you have to low blood glucose levels and a higher concentration of ketone bodies.

This is because of the higher concentration of glucose (sugar) found in some fruits, which could give you problems the same way that sweet foods do.

Daily plasma glucose concentrations were 10 percent lower with the high - fiber diet than with the ADA diet (values for the area under the curve, 3743 ± 944 vs. 3365 ± 1003 mg ․ hour per deciliter [207.8 ± 52.4 vs. 186.8 ± 55.7 mmol ․ hour per liter]; P = 0.02), and plasma insulin concentrations were 12 percent lower (values for the area under the curve, 1107 ± 650 vs. 971 ± 491 μU ․ hour per milliliter [6642 ± 3900 vs. 5826 ± 2946 pmol ․ hour per liter]; P = 0.05)(Figure 1).

The lower glucose AUC observed in this study is in agreement with previous research by Holmstrup et al. [120], who reported lower 12 - hour glucose concentrations as a result of consuming three high - carbohydrate meals compared to the equivalent distributed over the course of six meals.

The mean plasma glucose concentration was lower (by 13 mg per deciliter [0.7 mmol per liter], or 8.9 percent) when patients completed the high - fiber diet than when they completed the ADA diet (P = 0.04), and mean daily urinary glucose excretion was 1.3 g lower (P = 0.008).

During the sixth week of the high - fiber diet, as compared with the sixth week of the ADA diet, mean daily preprandial plasma glucose concentrations were 13 mg per deciliter (0.7 mmol per liter) lower (95 percent confidence interval, 1 to 24 mg per deciliter [0.1 to 1.3 mmol per liter]; P = 0.04) and mean daily urinary glucose excretion was 1.3 g lower (median difference, 0.23 g; 95 percent confidence interval, 0.03 to 1.83; P = 0.008).

Glucose enters the brain across a concentration gradient, hyperglycemia in the blood leads to toxicity to neurons, so hyperglycemia is potentially toxic; high insulin is not if it is physiological.

The high - fiber diet also lowered the area under the curve for 24 - hour plasma glucose and insulin concentrations, which were measured every two hours, by 10 percent (P = 0.02) and 12 percent (P = 0.05), respectively.

Mean (± SE) 24 - Hour Profile of Plasma Glucose Concentrations (Panel A) and Insulin Concentrations (Panel B) during the Last Day of the American Diabetes Association (ADA) Diet and the Last Day of the High - Fiber Diet in 13 Patients with Type 2 Diabetes Mellitus.

The magnesium loss appears to be secondary to higher concentrations of glucose in the kidney that increase urine output [2].

Among pregnancies for which screening occurred at Mount Sinai Hospital (n = 44 110), 859 (1.9 %) fulfilled the diagnostic criterion of a very high 1 - hour glucose concentration of 10.3 mmol / L or more for the 50 - g glucose challenge test.

Nevertheless, it is established that repeated consumption of high glycemic index, mixed meals results in higher mean 24 h blood glucose and insulin concentrations when compared with low glycemic index, mixed meals of identical caloric content (75,76).

Eating high GI meals before exercise may result in plasma glucose concentrations peaking before the onset of exercise and then hypoglycaemia occurring within the first 30 minutes of the exercise period.

If the blood glucose concentration is consistently higher than normal, it may indicate that your dog's pancreas is not secreting (enough) insulin.

This disorder, which is the result of a relative or absolute insulin deficiency or of peripheral cell insensitivity to insulin, is characterized by high blood glucose concentrations such that the renal threshold is exceeded.

A diagnosis of diabetes is confirmed if glucose is present in the urine and its also at a persistently high concentration in the blood.

The diagnosis is considered as confirmed when glucose is found at consistently high concentrations in blood and in urine.

A diagnosis of diabetes only becomes definite when glucose is found in the urine and at a persistently high concentration in the blood.

Cats fed the high - carbohydrate diet had significantly higher mean and peak (23 — 32 %) glucose concentrations and tended to have higher insulin concentrations than cats fed either the high - protein or the high - fat diet (54).

Diets high in protein are associated with lower postprandial glucose and insulin concentrations than diets high in either fat or carbohydrate in normal cats

Research also suggests that diabetic cats may be predisposed to developing higher postprandial (after a meal) blood glucose concentrations after eating a high - carbohydrate meal.

This high urine glucose concentration can actually pull excessive amounts of water into the urine, resulting in increased urine volume, increased urinary water loss, a propensity for dehydration, and a compensatory increase in thirst.

In Type I diabetes, glucose concentrations are high because of a decrease in the production of insulin.

In either case, glucose is not delivered effectively to the cells, and instead remains in high concentrations in the bloodstream.

A high concentration of glucose in the urine and blood will show up as well as high levels of electrolyte imbalances and liver enzymes.