The Illinois investigators suggested that if the turnover of vitamin E in adipose tissue was
higher than the turnover of PUFAs, then the accumulation of PUFA in adipose tissue would eventually cause a vitamin E
deficit, especially if that extra supply of vitamin E were at some point cut off: if, for example, someone were to switch from a vegetable oil back to an animal fat, leaving their adipose tissue stuffed to the brim with PUFA but suddenly taking in much less vitamin E in their
daily diet.