In a breed that has about 40 %
hip dysplastic dogs according to OFA, it is very tempting to ignore tests results in favor of breeding.
Not exact matches
In a
dysplastic dog, the
hip and femur don't fit together correctly.
Any breed or mix can be
dysplastic; however, there are almost no statistics regarding the incidence of
hip dysplasia in non-purebreds as these
dogs are seldom x-rayed.
If you look at the
hip dysplasia statistics of the Orthopedic Foundation of America,
hip X-rays of 4000 Flat - Coats were evaluated and only 4 % were
dysplastic, which would be a good rate for this size
dog — except that the true rate is higher because most of the obviously bad X-rays were not sent in for official evaluation.
Dogs with
hips scored as borderline or
dysplastic are not eligible to receive OFA breeding status.
The Orthopedic Foundation of America evaluated the
hip X-rays of 12,500 Ridgebacks and found 5 %
dysplastic, which is a good rate for this size
dog.
Many
dog breeds are ailed by
hip dysplasia, but only about 5 % of Rhodesian Ridgebacks (taken from a sample of 8,300
dogs) were found to be
dysplastic.
The Orthopedic Foundation of America evaluated the
hip X-rays of 1100 American Eskimo
Dogs and found 9 %
dysplastic.
In the years of 2011 - 2015, 6,221 bullmastiff
dogs were tested for
hip dysplasia, and 25.4 % of them were found to be
dysplastic.
Dr. Donald Patterson, chairman of Medical Genetics at University of PA School of Veterinary Medicine, states that some
dogs with radiographically normal
hips but a large number of hidden dysplasia - producing genes, if mated together, will produce at least some
dysplastic offspring.
According to current research, the best way to prevent
hip dysplasia is to screen
dogs and remove them from the breeding population if they have radiographic evidence of
dysplastic hips.
This is still somewhat controversial even among reputable breeders, because
dogs with excellent
hips can produce
dysplastic puppies, and
dysplastic parents can produce puppies with excellent
hips.
Most
dysplastic dogs are born with normal
hips but due to genetic and possibly other factors, the soft tissues that surround the joint start to develop abnormally as the puppy grows.
GENETIC DISEASE CONTROL (GDC) This is an open registry that evaluates for
hip dysplasia and makes available all data obtained, including that on
dysplastic dogs.
Dogs with
hips scored as borderline or
dysplastic are not eligible to receive OFA breeding numbers.
The OFA evaluated the
hip X-rays of 21,000 Bernese Mountain
Dogs and found 16 %
dysplastic.
While the scientific community is actually divided on the actual clinical benefits of providing
dogs with joint health supplements such as glucosamine, chondroitin sulfate, and methyl sulfonyl methane or MSM, many
dog owners are nonetheless providing these supplements to their
dogs with
dysplastic hip joints.
Scientists have yet to uncover the specific gene in
dogs that code for the canine
hip dysplastic trait.
Reducing the body weight of your
dog can do to help in its
dysplastic hips.
Any large breed of
dogs has a higher chance of developing
dysplastic hips or elbows, but a good breeder will have had OFA (Orthopedic Foundation for Animals) certification done for both the sire and dam before considering breeding.
Even though symptoms may not be as severe in these
dogs, they still have
dysplastic hips and carry the genes that contribute to the disease.
In both situations, the
dogs have
dysplastic hips and should not be considered for breeding.
No amount of x-rays can prevent bone and joint problems or change a
dog's genetic predisposition for luxating patellas (knees), and
dysplastic hips or elbows.
Hip dysplasia is not something a
dog gets; it either is
dysplastic or it is not.
And breeding two
dogs with less - than - perfect
hips (e.g., mild with mild) can produce some
dysplastic dogs, but also still a majority (about 70 %) with acceptable
hip scores.
Many
dysplastic dogs are overweight, so a reducing diet is in order if the
dog's weight is causing undue pressure on his
hips.
A
dog can be
dysplastic in one or both
hips, can have a shallow socket and a normal ball, a malformed ball and normal socket, a shallow socket and malformed ball, a misaligned joint, loose ligaments, or a combination of these structural problems complicated by environmental factors such as rate of growth, level of nutrition, and exercise.
The 40
dogs described as «normal» at 2 years of age all had some «minimal or mild degenerative changes» by 9 years of age, and those 22
dogs diagnosed as
dysplastic (lax joints in the
hip - extended view) at 2 years had the same mild or minimal changes.
Because of this, my usual suggestion is to treat
dysplastic dogs medically for as long as possible and then opt for a total
hip replacement.
Dogs with a DI of under 0.3 almost always have normal
hips, and those over 0.7 are almost always
dysplastic.
The statistics published by the OFFA show that breeding two
dogs with «Good»
hips together would produce 10 %
dysplastic offspring.
Breeding studies have shown breeding 2
dysplastic dogs results in 85 - 95 %
dysplastic puppies, breeding a normal
dog to a
dysplastic dog results in approximately half of the puppies affected and breeding two normal
dogs will result in about one third of the puppies having
hip dysplasia.
Conversely, if a
dog with tight sockets is radiographed without rotating the femurs sufficiently, the femoral neck may appear shortened and at a valgus angle, both of which may cause some less - experienced vets to give a
dysplastic diagnosis to a «normal» set of
hips.
The vast majority of
dogs that undergo this procedure need only one
hip replaced because the artificial joint picks up the slack for the
dysplastic one.