But more importantly this old study (
by mitochondrial membrande phospholipid fatty
acids composition remodeling towards a low perodizable index PI and double bond unsaturation index DBI
by genetically reduced desaturase (essentially switching perodizable polyunsaturates with monounsaturates / saturates, rendering membranes lipid peroxidation resistant / blocking hydroperoxide formation
by DHA n - 3 and lowering mitochondrial DNA lesions formation
by membrane lipid peroxidation chain propagation), It's true though that these effects are due to transcriptional lipid genes regulation (so the genes are the reason of this dramatic effect and this is also a gene therapy, that
hit the key points) this old study showed
In contrast to the amino
acid divergence and the ratio of non-synonymous to synonymous substitutions per site that we examined in the present investigation, they analyzed the proportion of most diverged genes
by using the E value of the best
hit when searching annotated honeybee genes against the Genbank non-redundant protein database.