These toxins manipulate
host cell processes to create optimal growth conditions for the bacteria in hiding.
Not exact matches
That is, they depend on the
host cell for the raw materials and energy necessary for nucleic acid synthesis, protein synthesis,
processing and transport, and all other biochemical activities that allow the virus to multiply and spread.
Combing the genetic data from a transmission study in ferrets, a team led by Thomas Friedrich, a professor of pathobiological sciences at the University of Wisconsin - Madison School of Veterinary Medicine, found that during transmission, when one animal is infected by another through sneezing or coughing, the
process of natural selection acts strongly on hemagglutinin, the structure the virus uses to attach to and infect
host cells.
The new study shows that the synthetic compound is capable of inhibiting the activities of several DNA -
processing enzymes, including the «integrase» used by the Human Immunodeficiency Virus (HIV) to insert its genome into that of its
host cell.
Viruses can't survive without a
host, and the most - studied viruses linked to disease are lytic in nature: They get inside a
cell and make copies of themselves, destroying the
cell in the
process.
In particular, they have examined the ways in which viruses hijack
cell proteins to help regulate their exit and spread from the
host cell through the budding
process.
They seem most lifelike only when they invade and co-opt the machinery of living
cells in order to make more of themselves, often killing their
hosts in the
process.
That viral DNA is then directly spliced into the
host cell's DNA and passed along with the
cell's natural replication
process.
Once the viral RNA is transcribed, HCV initiates a
process known as IRES - mediated translation, which allows the viral RNA to be translated into proteins by bypassing certain protein translation checkpoints that would normally be required by the
host cell to start protein translation.
This
process enables the virus to take advantage of the
host cell's protein translation machinery for its own purposes.
The prM cleavage
process is important for new viral particles to be able to infect a
host cell.
Researchers from Melbourne's Walter and Eliza Hall Institute have discovered how a common parasite hijacks
host cells and stockpiles food so it can lie dormant for decades, possibly changing its
host's behavior or personality in the
process.
Melbourne researchers have discovered how a common parasite hijacks
host cells and stockpiles food so it can lie dormant for decades, possibly changing its
host's behaviour or personality in the
process.
Their findings — that nanotubes and vesicles are an important part of the communications
process — show that the extracellular vesicles contribute to the complexity of African trypanosomiasis through the transfer of virulence factors between parasites and inadvertent interaction with
host cells, which has a profound effect on disease, the study notes.
Sepsis, Drs. Tracey and Deutschman wrote, «is not a stereotypical, steadily progressive immunological
process that follows infection or injury; that innate and adaptive immune responses do not inevitably damage
host cells; and that the long - held notion that sepsis is a distinct immunological disorder is incorrect.
Animal cloning uses a
process known as somatic -
cell nuclear transfer, where the nucleus from an adult (donor)
cell is transferred into an egg (
host)
cell lacking a nucleus.
This resulted in a staggering 50,000 virus -
host protein interactions, approximately 500 of which appeared to be involved in viral infection.By removing the interacting
host proteins from the
cell one at a time, the researchers were able to determine what their functional contribution was in the infection
process: whether the
host proteins were hijacked by the virus and used to spread infection, or whether they were part of a defense mechanism against the virus.
This must be accomplished by using a
process known as germ - line transmission, where the primordial germ
cells (PGCs) of an early embryo are edited, grown in cultures, and then reproductively transmitted through surrogate
host parents to generate live, engineered birds.
Providing rigidity to the
cell, actin filaments are involved in a
host of
processes including muscle contraction,
cell mobility and
cell division.
LA JOLLA, CA — When the body is under attack from pathogens, the immune system marshals a diverse collection of immune
cells to work together in a tightly orchestrated
process and defend the
host against the intruders.
By playing with the protein, they showed that the virus could be switched on or off separate from the rhythms of its
host immune
cell, indicating that the two
processes were independent of one another.
A
host of endocytic
processes has been described at the plasma membrane of eukaryotic
cells.
This multi-stage
process requires tumour
cells to survive in the circulation, extravasate at distant sites, then proliferate; it involves contributions from both the tumour
cell and tumour microenvironment («
host», which includes stromal
cells and the immune system).
While erythrocyte invasion is a rapid
process, the brief extracellular exposure of merozoites outside of their intra-erythrocytic niche places them in direct contact with
host antibodies, which contribute to naturally acquired immunity to malaria [8, 9]; therefore, merozoite
cell surface and secreted proteins have long been considered attractive targets for rational vaccine development.