Following their recent discovery of a protein pathway, Moffitt Cancer Center researchers are one step closer to understanding
how lung cancer cells metastasize.
Not exact matches
A protein called YAP1 was previously shown to contribute to the growth of
lung cancer cells; however, it was unknown
how YAP1 controls
lung cancer growth and progression.
A Yale
Cancer Center research team conducted a study to determine
how those tumor
cells manage to grow outside the
lungs.
The study, called «Molecular Determinants of Drug - Specific Sensitivity for Epidermal Growth Factor Receptor (EGFR) Exon 19 and 20 Mutants in Non-Small
Cell Lung Cancer,» and published online in the journal Oncotarget, demonstrates how computer modeling of EGFR mutations found in lung cancer can elucidate their molecular mechanism of action and consequently optimize the selection of therapeutic agents to treat patie
Lung Cancer,» and published online in the journal Oncotarget, demonstrates how computer modeling of EGFR mutations found in lung cancer can elucidate their molecular mechanism of action and consequently optimize the selection of therapeutic agents to treat pat
Cancer,» and published online in the journal Oncotarget, demonstrates
how computer modeling of EGFR mutations found in
lung cancer can elucidate their molecular mechanism of action and consequently optimize the selection of therapeutic agents to treat patie
lung cancer can elucidate their molecular mechanism of action and consequently optimize the selection of therapeutic agents to treat pat
cancer can elucidate their molecular mechanism of action and consequently optimize the selection of therapeutic agents to treat patients.
Marcus and former graduate student Jessica Konen, PhD began by observing
how a mass of
lung cancer cells behaves when embedded in a 3 - D protein gel.
«For example, mouse mammary tumors shared a signaling pathway that is found in human
lung cancer and controls
how cells reproduce and move from one location to another.»
Professor Dive's exhibit was designed to introduce visitors to the concept of circulating tumour
cells (CTCs) and
how liquid biopsies could hold the key for better diagnosis and treatment of
lung cancer.
To better understand
how these normally helpful components of the immune system are put to nefarious tasks in
cancer cells, Verma and his colleagues developed a new method of inducing non-small-cell
lung cancer in mice.
Assuming that the findings of the review reflected a causal relation between smoking cessation and risk of all cause mortality, we further investigated the data by constructing life tables for a hypothetical group of 100 patients aged 65 years with early stage
lung cancer to estimate
how many deaths would be prevented by smoking cessation within the non-small
cell lung cancer and small
cell lung cancer populations during five years.
One study presented in the journal — from a group led by Patrick Singleton, PhD, assistant professor of medicine at the University of Chicago Medicine — shows
how opioids already present in the body can enhance the malignant tendencies of human
lung cancer cells transplanted into mice, even without the addition of morphine.
It's unbelievable story
how she was left to die with Stage IV, non-small
cell lung cancer.