Sentences with phrase «how tumors»

Recent evidence provides essential clues into how these tumors grow and progress, generating new ideas for treatment approaches.

; Eating Green to Prevent Cancer; How Tumors Use Meat to Grow; and Avoiding Dairy to Prevent Parkinson's.

; How Tumors Use Meat to Grow; Mushrooms for Breast Cancer Prevention; Foods That May Block Cancer Formation; Flax Seeds for Prostate Cancer; Flax and Breast Cancer Survival; and Treating Breast Pain with Flax Seeds.

For more context, check out my associated blog posts: Poultry and Penis Cancer; Poultry Paunch: Meat & Weight Gain; Treating an Enlarged Prostate With Diet; Eating Green to Prevent Cancer; How To Reduce Dietary Antibiotic Intake; and How Tumors Use Meat to Grow.

; The Anti-Wrinkle Diet; Increasing Muscle Strength with Fenugreek; How Tumors Use Meat to Grow; Mushrooms for Breast Cancer Prevention; Prevent Breast Cancer by Any Greens Necessary; Foods That May Block Cancer Formation; and Breast Cancer & Alcohol: How Much Is Safe?.

; How Tumors Use Meat to Grow; 98 % of American Diets Potassium Deficient; Best Treatment for Constipation; Breast Cancer & Alcohol: How Much Is Safe?

This is the final installment of Dr. Greger's video series that started with Cancer as an Autoimmune Disease, the mystery for which may have been solved with How Tumors Use Meat to Grow: Xeno - Autoantibodies.

See The Inflammatory Meat Molecule Neu5Gc for background and Friday's video - of - the - day How Tumors Use Meat to Grow: Xeno - Autoantibodies for the role it might play in cancer growth.

In my post How Tumors Use Meat to Grow I talked about the role Neu5Gc may play in stimulating breast cancer growth, but what about inflammation in our joints?

For more context, also see my associated blog posts: Inflammation, Diet, and «Vitamin S»; The Most Anti-Inflammatory Mushroom; How To Boost Serotonin Naturally; Treating Crohn's Disease With Diet; Eating Green to Prevent Cancer; and How Tumors Use Meat to Grow.

When Al moved to the MD Anderson hospital in 1969, he was no longer distracted by the folderol of L.A. Within two years he proposed a novel genetic theory, based on a complex mathematical model, to explain how tumors of the eye arose in retinoblastoma.

A series of experiments further elucidated just how tumors exploit the interaction between PD - 1 and PD - L1 to survive.

Funding Agency CPRIT - First Time Tenure Track Award Title Understanding Functional Heterogeneity in Sarcomas Status Active Active Period 2016 Role Principal Investigator Grant Detail Relapse is the major problem in the clinic and understanding how tumors change or evolve at relapse is the focus of this grant.

Two devices, developed independently, can gauge how tumors will respond to various drugs.

Patients were often put through grueling — and expensive — treatment protocols without knowing how their tumors would react.

In the May 11, 2015 issue of Nature, researchers from the University of Chicago show how these tumors shield themselves from T cells — the immune system's front - line anti-cancer weapon — by producing high levels of beta - catenin, an intracellular messenger.

These research collaborations have the power to answer questions critical to improving cancer outcomes, such as how tumors develop drug resistance, and what treatments are most effective against particular genomic traits.

Comparing proteins within the microvesicles with the originating tumors showed that the vesicles also accurately reveal the protein content of the tumors, providing still more information about how tumors might respond to specific therapies, the researchers report.

«Low oxygen, high risk: How tumors adapt to become more aggressive.»

The idea was immediately controversial but Hendrix and others have since pieced together a picture of how tumors build their own blood vessels and how they can affect prognosis and treatment.

Knowing that they have been present in human relatives since prehistoric times could shed light on how the tumors co-opt an ancient molecular pathway in our cells to grow.

The data may help scientists determine how tumors evade detection by the immune system.

This is important for the basic understanding of how these tumors are formed and can contribute to the development of more efficient and specific glioma therapies.

Stanford neuroscientists review how tumors exploit neuronal signals February 13 in Trends in Cancer.

«This should greatly improve our understanding of the genomics of bladder cancer, how these tumors respond to drugs, and how they develop drug resistance.

The study — Genomic profiles of low - grade murine gliomas evolve during progression to glioblastoma — published April 7, shows how these tumors continue to rapidly evolve, becoming ever more genetically diverse, as they become malignant and progress.

When the researchers took biopsies of the patients» tumors during the trial, they confirmed that their hypothesis about how the tumors would respond was correct.

Sharma had developed a new method for studying how tumors with different characteristics respond to different immunotherapies: She would treat patients before their growths were surgically removed, then analyze the tissue in her lab.

«Overall, survival for patients with recurrent rhabdomyosarcoma is just 17 percent, and until now nothing was known about how tumors evolve in response to therapy,» said corresponding author Michael Dyer, Ph.D., a member of the St. Jude Department of Developmental Neurobiology and a Howard Hughes Medical Institute Investigator.

The research explains how tumors evolve and cause cancer resistance to drugs designed to match the patient's unique genomic makeup.

Now that we know more about how cancer originates and how tumors re-grow after treatment, we can design new therapies to prevent this.

Although there have been great advances made in the treatment of leukemias and other cancers, little is known about how Glioblastomas are formed and how these tumors infiltrate the brain tissue.

When Markovic told his wife, a cardiologist, about his discovery of the striking similarities between how tumors and placentas control the immune system, he describes her as being wholly unsurprised.

«We're trying to build models that describe how tumors grow and respond to therapy,» said Yankeelov, director of the Center for Computational Oncology at The University of Texas at Austin (UT Austin) and director of Cancer Imaging Research in the LIVESTRONG Cancer Institutes of the Dell Medical School.

«But if you have a model that can recapitulate how tumors grow and respond to therapy, then it becomes a classic engineering optimization problem.

They are trying to reproduce those results in a community setting and extend their models by adding new factors that describe how the tumor evolves.

Scientists have uncovered how tumor cells in aggressive uterine cancer can switch disguises and spread so quickly to other parts of the body.

The 2016 People's Choice Award went to Emmanuelle Alaluf of the Free University of Brussels, for her video using ballet and other dance styles to show how tumor cells evade the immune system.

A Yale Cancer Center research team conducted a study to determine how those tumor cells manage to grow outside the lungs.

«This helps us distinguish malignant from healthy tissue and tells us how the tumor is responding to chemotherapy earlier than other imaging techniques can.»

Their finding — that the presence of high levels of NF - κB in lung cancer tumors can act as a suppressor — provides new insight into how tumor pathways regulate the anti-tumor response.

«If we want to understand the role of estrogen, we now have to look at how the tumor and the microenvironment communicate with one another,» den Boon says.

«We know very little about how gene mutations in tumor cells can change how a tumor might respond or not to certain chemotherapy drugs.

Here, we will explore how the tumor microenvironment promotes oncogenic progression, while protecting the tumor from therapeutic intervention.

Researchers developed models to examine the influence of driver mutations — mutations that promote cancer development — on the initiation and development of gliomas, and how tumor genomic profiles evolve as the cancer progresses.

Of particular interest are how tumor - specific metabolic changes promote oncogenic progression and how these changes can be exploited to develop more effective treatment options.

Meanwhile, researchers from the University of Rome found that texture analysis provides useful «imaging biomarkers» that indicate how the tumor is responding to chemotherapy and radiotherapy.

With the three - year grant, Vanapalli and his collaborators Boyd Butler in the Department of Biological Sciences and Everardo Cobos at the Texas Tech University Health Sciences Center, will build microfluidic devices that mimic blood flow to study how tumor cells move inside capillaries, how they squeeze through tight spaces, whether they are subject to fragmentation and how they become stuck.

For the first time, researchers have been able to grow, in a lab, both normal and primary cancerous prostate cells from a patient, and then implant a million of the cancer cells into a mouse to track how the tumor progresses.

Richard Moriggl and his co-workers have now published in the journal Leukemia how the tumor promoter STAT5 integrates metabolic signals that contribute to oncogenic transformation.