Sentences with phrase «hyperglycemia in»

It's proven safe and effective for the reduction of hyperglycemia (high blood glucose) and clinical signs associated with hyperglycemia in cats with diabetes.

Vetsulin (porcine insulin zinc suspension) is a prescription medication used to control hyperglycemia in dogs and cats with diabetes mellitus.

Vetsulin is Purified porcine (pork) insulin Zinc Suspension that is used to control hyperglycemia in dogs and cats with diabetes mellitus.

This is safer and we don't see the long term complications seen with mild persistent hyperglycemia in dogs [5].

Used to control hyperglycemia in dogs and cats with diabetes mellitus This medication will cause the blood sugar to go down What dogs / cats should not take this medication?

Humulin also helps to control hyperglycemia in dogs with diabetes.

Hyperglycemia in Cats The term hyperglycemia refers to higher than normal levels of glucose in the blood.

Hyperglycemia in dogs occurs when blood sugar (glucose) levels are higher than 130 mg / dl, although symptoms and signs appear only when a critical level of 180 - 220 mg / dl is reached.

I should add — I was referring to hyperglycemia in diabetics.

Glucose enters the brain across a concentration gradient, hyperglycemia in the blood leads to toxicity to neurons, so hyperglycemia is potentially toxic; high insulin is not if it is physiological.

Dietary cyanidin 3 - O - β - D - glucoside - rich purple corn color prevents obesity and ameliorates hyperglycemia in mice (2013):

Dietary cyanidin 3 - O - beta - D - glucoside - rich purple corn color prevents obesity and ameliorates hyperglycemia in mice.

In this study we demonstrate that anthocyanins from blueberry have the potency to alleviate symptoms of hyperglycemia in diabetic C57b1 / 6J mice.

Specific bioactive compounds in ginger and apple alleviate hyperglycemia in mice with high fat diet - induced obesity via Nrf2 mediated pathway.

One study found that vitamin K2 supplementation was capable of preventing hyperglycemia in diabetic rats.5 Human studies of vitamin K demonstrate that both K1 and K2 are effective in reducing the effects of type II diabetes.

Persistent hyperglycemia in diabetes mellitus results in an accumulation of a series of reactive α - dicarbonyl compounds (α - DCs, e.g. glyoxal / GO, methylglyoxal / MGO, 3 - deoxyglucosone / 3DG) and α - DC - derived metabolites, called advanced glycation end products (AGEs).

Published in the journal Scientific Reports, the study investigated how a single bout of intense exercise could reduce olanzapine - induced hyperglycemia in male mice.

Dietary cyanidin 3 - O - β - D - glucoside - rich purple corn color prevents obesity and ameliorates hyperglycemia in mice (2013):

Not exact matches

Children who are diagnosed with this disorder are unable to produce insulin, which leads to hyperglycemia or high levels of glucose in the bloodstream.

A regulated pulse of insulin secretion resulted in a transient correction of serum glucose concentrations in a mouse model of hyperglycemia.

Rapamycin can make patients susceptible to some opportunistic infections and has been linked to hyperglycemia, which can cause type 2 diabetes, and high levels of triglycerides in the blood, which can contribute to heart disease.

Worrisome is that already in the state of prediabetes hyperglycemia is associated with increased risk of type 2 diabetes, cardiovascular disease, dementia and cancer.

Previous research has shown that hyperglycemia (high blood sugar) after CABG and other cardiac surgery is associated with increased morbidity and mortality; however, more recent studies have shown that liberal maintenance of blood glucose levels (< 180 mg / dL) after CABG surgery can be safer and more advantageous in both diabetic and non-diabetic patients.

The new study in the American Journal of Clinical Nutrition reports that hundreds of people who were gestated during a horrific famine that afflicted China between 1959 and 1961 had significantly elevated odds of both hyperglycemia and type 2 diabetes.

In contrast, in mice with normal immune systems, emulsifiers induced low - grade or mild intestinal inflammation and metabolic syndrome, characterized by increased levels of food consumption, obesity, hyperglycemia and insulin resistancIn contrast, in mice with normal immune systems, emulsifiers induced low - grade or mild intestinal inflammation and metabolic syndrome, characterized by increased levels of food consumption, obesity, hyperglycemia and insulin resistancin mice with normal immune systems, emulsifiers induced low - grade or mild intestinal inflammation and metabolic syndrome, characterized by increased levels of food consumption, obesity, hyperglycemia and insulin resistance.

The increased risk of hyperglycemia associated with prenatal exposure to famine is also passed down to the next generation, according to a new study of hundreds of families affected by widespread starvation in mid-20th Century China.

Even worse, a miscalculation or lapse in regimen can cause blood sugar levels to rise too high (hyperglycemia), potentially leading to heart disease, blindness and other long - term complications, or to plummet too low (hypoglycemia), which in the worst cases can result in coma or even death.

However, researchers found that aerobic exercise - induced improvements in glycemic control were reduced by ambient hyperglycemia, particularly in participants with T2DM.

Exercise - induced improvements in glycemic control are dependent on the pre-training glycemic level, and although moderate - intensity aerobic exercise can improve glycemic control, individuals with ambient hyperglycemia (high blood glucose) are more likely to be nonresponders, according to a research letter by Thomas P. J. Solomon, Ph.D. of the Centre of Inflammation and Metabolism, Copenhagen, Denmark, and colleagues.

Hyperglycemia is a potentially harmful environment for a growing fetus and can contribute to obesity and diabetes later in the child's life.

In our studies, we observed hyperglycemia and impaired glucose tolerance in the Tg - hIAPP mice treated with IAPP aggregates, which occurred concomitant to a significant loss of β cells (> 60 %In our studies, we observed hyperglycemia and impaired glucose tolerance in the Tg - hIAPP mice treated with IAPP aggregates, which occurred concomitant to a significant loss of β cells (> 60 %in the Tg - hIAPP mice treated with IAPP aggregates, which occurred concomitant to a significant loss of β cells (> 60 %).

For that purpose, we used a streptozotocin (STZ) model of diabetes, which is widely used in the field to produce destruction of β cells and hyperglycemia (Wilson and Leiter, 1990).

Osteocalcin - deficient mice exhibit insulin resistance and hyperglycemia (27), and administration of undercarboxylated osteocalcin protects against the metabolic consequences of diet - induced obesity in WT mice (30, 60).

Her research team found that cellular oxidative stress (arising because of reactive oxygen species) increases in mice exposed to THS, damaging proteins, fats and DNA, and leading to hyperglycemia (excess glucose in the blood stream) and insulinemia (excess insulin in the blood)-- a condition also called insulin resistance.

Our findings demonstrate that induction of IAPP misfolding and aggregation by administration of preformed IAPP - aggregated seeds leads to the development of marked T2D - like pathology, including severe hyperglycemia, impaired glucose homeostasis, loss of β cells, and changes in islets morphology.

Because PD - L1 / programmed cell death (PD - 1) deficiency accelerates development of diabetes in mouse models, Nasr et al. hypothesized that a defect in the PD - L1 / PD - 1 pathway may underpin the hyperglycemia observed in the nonobese diabetic (NOD) mouse model.

Further analysis revealed that the mice in Tg / Tg group, which developed severe hyperglycemia, lost > 75 % of their β cells compared with WT mice of the same strain and similar age.

In fact, by 20 wk of age > 70 % of the animals in this group developed hyperglycemia, defined as a blood glucose concentration > 250 mg / dL (Fig. 4 BIn fact, by 20 wk of age > 70 % of the animals in this group developed hyperglycemia, defined as a blood glucose concentration > 250 mg / dL (Fig. 4 Bin this group developed hyperglycemia, defined as a blood glucose concentration > 250 mg / dL (Fig. 4 B).

(B) Inoculation with diabetic pancreas homogenate produced severe hyperglycemia (> 250 mg / dL) in a portion of the animals.

Type 2 diabetes (T2D) mellitus is characterized by hyperglycemia, insulin resistance, defective insulin secretion, loss of β cell function and mass, and accumulation of amyloid in the islets of Langerhans (Stumvoll et al., 2005).

In homozygosis, male Tg mice spontaneously develop IAPP amyloid deposits, which are associated with selective β cell death, impaired insulin secretion and hyperglycemia (Janson et al., 1996).

This is in agreement with the situation in vivo, where defects in GS activity are compensated for by hyperinsulinemia and hyperglycemia until glycogen synthesis becomes «normal.»

We then tested whether known risk factors for T2D, including obesity and hyperglycemia, affect DNA methylation of the 15,627 CpG sites differentially methylated in case - control cohort 2, already in nondiabetic subjects.

Ikemoto S, Takahashi M, Tsunoda N, Maruyama K, Itakura H and Ezaki O. High - fat diet - induced hyperglycemia and obesity in mice: differential effects of dietary oils.

Plenty of conditions occurring in pregnancy, such as over - or undernutrition, hyperglycemia, and acute stress situations, for example, are known to influence the phenotype of the progeny via epigenetic effects without affecting the genetic coding directly (140).

Thus, increased MYCL does not appear to be an early marker in the pathogenesis of diabetes but a consequence of chronic hyperglycemia (228).

In diabetic non ‐ obese diabetic (NOD) mice, adipose - derived MSCs (ASCs) have been shown to decrease hyperglycemia and insulitis through attenuation of the Th1 immune response and expansion of T regulatory lymphocytes [3].

«We were interested in investigating how GLP - 1 could protect against the effects of hyperglycemia on renal function.»

Through assessment of hASCs potential role in protecting against STZ ‐ induced hyperglycemia and loss of β cell mass, the authors have uncovered a novel role for the matrix metalloproteinase inhibitor TIMP ‐ 1 in promoting β cell survival.

Josin researchers made two major findings: They identified the mechanisms by which GLP - 1 can induce protective actions on the glomerular (renal) endothelial cells by inhibiting the signaling pathway of Ang II and its pro-inflammatory effect; and demonstrated a dual signaling mechanism by which hyperglycemia, via PKCβ activation, can increase Ang II action and inhibit GLP - 1's protective effects by reducing the expression of GLP - 1 receptors in the glomerular endothelial cells.