Sentences with word «hyperphagia»

Revelli J.P, Smith D, Allen J, Jeter - Jones S, Shadoan M.K, Desai U, Schneider M, Sligtenhorst I, Kirkpatrick L, Platt K.A, Suwanichkul A, Savelieva K, Gerhardt B, Mitchell J, Syrewicz J, Zambrowicz B, Hamman B.D, Vogel P, Powell D.R. Profound obesity secondary to hyperphagia in mice lacking kinase suppressor of ras 2.

Prolonged hyperphagia with high - fat feeding contributes to exacerbated weight gain in rats with adult - onset obesity.

«This suggests that these elevations in peripheral endocannabinoid signaling are critical in driving hyperphagia associated with a western diet,» said Argueta, the first author of the research paper.

Prader - Willi syndrome (PWS) is a rare genetic disease characterized by hyperphagia — a chronic feeling of hunger that, coupled with a metabolism that utilizes drastically fewer calories than normal, often leads to excessive eating and obesity in patients with the disease.

Diet - induced obesity in female mice leads to offspring hyperphagia, adiposity, hypertension, and insulin resistance: a novel murine model of developmental programming

Although hyperphagia contributes to the obesity, homozygotes gain excess weight and deposit excess fat even when restricted to a diet sufficient for normal weight maintenance in lean mice.

If you give into the cravings, you could develop something call hyperphagia, a condition where the hormones in charge of regulating hunger don't shut - off and you enter a state of constant feeling of hunger which again makes you experience food cravings and not being able to resist the temptation of overeating.

Answer: Yes, she could've been hyperthyroid if she was always hungry = hyperphagia is what we call that.

Discussion: Previous studies showed an increase in orexigenic neurotransmitters production in IF, inducing hunger and hyperphagia in the ad libitum feeding days.

They found that when compared to mice fed a standard low - fat / low - sugar diet, mice fed a western diet for 60 days rapidly gained body weight and became obese, and displayed «hyperphagia,» that is, they consumed significantly more calories, and consumed significantly larger meals at a much higher rate of intake (calories per minute).

Decreased desire to eat is termed anorexia, while polyphagia (or «hyperphagia») is increased eating.

Mice homozygous for the obese spontaneous mutation, Lepob (commonly referred to as ob or ob / ob), exhibit obesity, hyperphagia, transient hyperglycemia, glucose intolerance, and elevated plasma insulin.

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