Sentences with phrase «impaired glucose»

Sense of Humor and General Life Satisfaction in Association with the Biological Effects of Resistance Training in People with Impaired Glucose Tolerance

Impaired glucose tolerance in adolescent offspring of diabetic mothers.

Association of napping and night - time sleep with impaired glucose regulation, insulin resistance and glycated haemoglobin in Chinese middle - aged adults with no diabetes: a cross-sectional study

Provided nutritional counseling for people with diabetes, gestational diabetes, impaired glucose tolerance, impaired fasting glucose and insulin resistance

Individuals with Type 2 Diabetes Mellitus, Pre-Diabetes (Impaired Glucose Tolerance / Impaired Fasting Glucose), and Hypertension.

Individuals with Type 2 Diabetes Mellitus, Pre-Diabetes (Impaired Glucose Tolerance / Impaired Fasting Glucose), and Hyper... Read more

In individuals where this balance can not be maintained, impaired glucose tolerance and overt diabetes ensues.

It is interesting to speculate whether the underlying low insulin sensitivity in our lean cats, which increased their risk of impaired glucose tolerance with obesity, was genetically determined as it is in people.

In fact, having an insulin sensitivity index below the reference - range median resulted in a threefold increased risk of developing impaired glucose tolerance with weight gain.

Despite the evidence that obesity causes impaired glucose tolerance, it appears that very few dogs develop overt diabetes as a consequence of obesity - induced insulin resistance.

Fasting hyperinsulinemia in lean cats was the greatest single risk factor for the development of impaired glucose tolerance with obesity.

In dogs, obesity causes insulin resistance (114 — 116), which leads to hyperinsulinemia and impaired glucose tolerance (117,118).

Studies have shown that almost half of all healthy cats will develop impaired glucose tolerance — a risk factor for diabetes — when they increase their body weight by around 40 percent.

Clear evidence that high - dose (300 mg) niacinamide causes oxidative stress and impaired glucose tolerance.

The study, Fish oil ameliorates trimethylamine N - oxide - exacerbated glucose intolerance in high - fat diet - fed mice, found that in mice, dietary FO ameliorated TMAO - induced impaired glucose tolerance, insulin signal transduction in peripheral tissue, and adipose tissue inflammation in HFD - fed mice.

Relative to active adults who do not participate in large amounts of sedentary behavior, these «active couch potatoes» exhibit impaired glucose metabolism and increased blood pressure despite their regular exercise habits [6, 7].

I was also starting to see some signs of impaired glucose control.I wasn't feeling good and I was in need of a kind of reset of sorts.

But, what is less known is those that have impaired glucose metabolism — which falls below the diagnostic threshold for diabetes — have an increased risk of cancer as well.

Using a more accurate conversion, it would be 118; still very unhealthy and considered impaired glucose tolerant.

However, what my experience has been with my VLC (very low carb) and higher fat diet, is that fasting and post prandial glucose become further improved, into completely healthy, non-diabetic, non impaired glucose tolerant ranges.

Impaired glucose tolerance: Without sleep, the central nervous system becomes more active, inhibiting the pancreas from producing adequate insulin, the hormone the body needs to digest, glucose.

Postprandial hormonal responses to different types of complex carbohydrate in individuals with impaired glucose tolerance

Studies have shown that by 30 years of age, an incredible 25 - 30 percent of obese PCOD women will have either an impaired glucose tolerance or Type 2 diabetes.

Testing should be considered for all individuals who are overweight or obese (BMI 25 or greater) plus the additional risks: first degree relative with diabetes mellitus or Polycystic Ovarian Disease, history of gestational diabetes, thyroid disorder, HDL level < 35 mg / dl and / or Triglyceride level > 250 mg / dl, hypertensive, sedentary, impaired glucose tolerance (IGT) or impaired fasting glucose (IFG) on previous testing, women who delivered macrosomic baby (e.g. weighing > 9 lbs), and other clinical conditions such as acanthosis nigricans, severe obesity, polycystic ovary syndrome, dyslipidemia.

1) Fasting biochemical screen and lipid profile 2) 2 - hour oral glucose tolerance test (GTT) in patients with risk factors (obesity, family history, history of gestational diabetes) and may indicate impaired glucose tolerance (insulin resistance) in 15 - 30 % of women with PCOS.

in 2016, genetically identical mice that consumed a high - fat diet were more likely to produce obese offspring with impaired glucose tolerance, an early sign of type 2 diabetes.

In rats moderate maternal dietary protein restriction is known to alter phenotypes in the offspring, which manifests as hypertension, dyslipidemia, and impaired glucose metabolism.

They found that post-menopausal women with PCOS had greater CRP levels, impaired glucose metabolism and insulin resistance than women without PCOS; levels worsened with age.

RESEARCH DESIGN AND METHODS Subjects (n = 54) were given a high - fat meal (75 g fat, 5 g carbohydrate, 6 g protein) after an overnight fast (nonobese control [NOC]: age 39.9 ± 11.8 years [mean ± SD], BMI 24.9 ± 3.2 kg / m2, n = 9; obese: age 43.8 ± 9.5 years, BMI 33.3 ± 2.5 kg / m2, n = 15; impaired glucose tolerance [IGT]: age 41.7 ± 11.3 years, BMI 32.0 ± 4.5 kg / m2, n = 12; type 2 diabetes: age 45.4 ± 10.1 years, BMI 30.3 ± 4.5 kg / m2, n = 18].

Even in the absence of health conditions such as type 2 diabetes and impaired glucose tolerance, higher blood glucose levels appear to negatively affect brain health and overall health.

Diets based on foods that can reduce postprandial blood glucose excursions [ie, fiber - rich foods with a low glycemic index (GI)-RSB- are receiving increasing attention regarding their ability to reduce the risk of diseases related to impaired glucose metabolism (1).

In addition, researchers have correlated obstructive sleep apnea — a disorder in which breathing difficulties during sleep lead to frequent arousals — with the development of impaired glucose control similar to that which occurs in diabetes.

Plus, eating quickly has been associated with health risks, such as insulin resistance and impaired glucose tolerance.

Some people experience problems processing glucose, which is known as impaired glucose tolerance (IGT) and leads to blood sugar spikes after heavy meals, whereas type 2 diabetes patients have a chronic condition where insulin simply can not be generated in adequate amounts, creating an imbalance.

This result is an impaired glucose tolerance, decreasing immunity and increasing susceptibility to illness.

In contrast, higher postprandial insulin concentrations and increased fat oxidation with breakfast skipping suggest the development of metabolic inflexibility in response to prolonged fasting that may in the long term lead to low - grade inflammation and impaired glucose homeostasis.

Impaired glucose tolerance, decreased insulin sensitivity and low - grade inflammation are also all associated with impaired sleep.

For those of us who are lean vegans with impaired glucose metabolism or prediabetes, I wonder if other factors are responsible.

In today's conversation, Amy reveals that impaired glucose metabolism and insulin signaling are at the root of the disease.

A recently published paper entitled «Molecular pathophysiology of impaired glucose metabolism, mitochondrial dysfunction, and oxidative DNA damage in Alzheimer's disease brain» by Abolhassani et al (1) suggests no.

It is well documented that impaired glucose metabolism or mitochondrial dysfunction is one of the major pathological changes observed in various neurodegenerative diseases including Alzheimer's disease (AD), Parkinson's disease (PD) or Huntington's disease (HD), thus suggesting that regulation of glucose metabolism and maintenance of mitochondrial homeostasis are critical for brain function.»

Some researchers believe the connection between impaired glucose metabolism, insulin signaling and AD is so strong that they refer to AD as «type 3 diabetes.»

[A] deficit of GnT - 4a glycosyltransferase expression in beta cells... produced signs of metabolic disease, including hyperglycemia, impaired glucose tolerance, hyperinsulinemia, hepatic steatosis and diminished insulin action in muscle and adipose tissues.

The major symptom of chromium deficiency is impaired glucose tolerance.

One result of these changes is insulin resistance and impaired glucose tolerance.

The chronically increased insulin demand could sooner or later lead to pancreatic ß cell failure and, consequently, impaired glucose tolerance as well as increased insulin resistance, which is a diabetes predictor.

WNIN Mutant Obese Rats Develop Acute Pancreatitis With the Enhanced Inflammatory Milieu WNIN Mutant Obese Rats Develop Acute Pancreatitis With the Enhanced Inflammatory Milieu WNIN / Gr - Ob rats demonstrate features of metabolic syndrome that include obesity, insulin resistance, impaired glucose tolerance, and hyperinsulinemia.

In agreement, transgenerational transmission of impaired glucose tolerance also through the paternal lineage has been previously reported [34].

WNIN / Gr - Ob rats demonstrate features of metabolic syndrome that include obesity, insulin resistance, impaired glucose tolerance, and hyperinsulinemia.

Studies in diet - induced obese (DIO) mice with IRAB - A reduced fed blood glucose and insulinemia yet impaired glucose tolerance and led to protracted insulinemia during a meal challenge.